A few days ago, at Director Zhao Tianji’s online workstation, there was a patient’s message: “I’m 60 years old, and I’ve been hearing a neuromagnosis for over a year, and I can’t get my feet out of the way right now. What is the probability of a paraplegic aneurysm being removed?”
Director Zhao Tien Ji-ji explained that, according to the patient ‘ s current examination information, there are very low rates of convulsion at present from the age of the patient, the size of the tumour, and over 90 per cent of the risk of dysentery at the time of the neuroma operation.
Listening to neuromagnosis first and foremost to avoid increasing the difficulty of the surgery, while the development of medical imaging, micro-neurological surgery and cranial neural monitoring techniques has led to an increased focus on the preservation of the neurological function and the maintenance of the quality of life after surgery, in particular the protection of the facial nerve, in addition to increasing the rate of detoxification, as an important factor in the choice of treatment by cranial surgeons and most patients.
The majority of early signs of hearing neuromagnosis are characterized by an acoustic acoustic acoustics and a decrease in hearing, which tends to draw less attention to patients when the symptoms are lighter; as the tumours grow, the hearing can decline and even develop to deafness. The early onset of hearing impairment and dizziness due to the tumour pressure on the front nervous system of the main department, in particular the tumour in which the subject of the tumour is located within the osteopathic hearing, can affect hearing and the frontal nervous function.
Listening to neuromas are benign tumours, and if discovered, the procedure should be completed as soon as possible in order to be fully cured.
What are the clinical symptoms of hearing a nervous tumor?
Listening to neurological tumours that are flat, slow, early symptoms — osteoporosis, facial palsy, etc. — makes them very susceptible to error or omission. The early manifestations of the hearing of a nervous tumor are mainly one-sided, persistent, stubborn ear ringing, and the effects of the treatment are not visible, and the prolonged hearing decline is an irritation caused by the tumour pressure on the nerves, which is difficult to discern. As the tumour increases, the symptoms gradually increase, there is an abnormal and numbness of the side, a slow or disappearing reflection of the cornea, reduced limbs, numbness of the limbs, reduced feelings, etc.
These tests “clip out” hearing neuromas.
1. Acoustic aneurysms, typical of the history of the disease, have the clinical manifestation of these incremental increases. The common symptoms of hearing aneurystic patients are hearing changes, trident nerve pains, headaches and pre-court abnormalities.
2. Neural ear examinations are frequently performed in the ear because of the early hearing and deafness of patients. Hearing and front-court neurofunctional examinations are commonly used.
(1) Hearing examination: There are four methods of hearing examination that distinguish hearing impairments from hearing impairment tests from the conductor system, snails or hearing nerve, type I is a normal or mid-heart disease; type II is hearing loss of ear cochlear; and type III and IV is the threshold test for hearing neuropsychiatric decomposition. If the tone recedes more than 30 dB for hearing neuropsychological disorders, the success of the short-enhanced sensitive trials is 60% to 100% for ear snails, the cysts for which there is an addition to the double-heart interchangeal volume balance, and the cysts for which there is no addition.
(2) Pre-court neurofunctional examination: hearing a neuromagnosis occurs mostly in the front-of-hearing section, and early use of cold and hot water tests almost all detects pre-pathological neurofunctional impairment, complete disappearance of reactions or partial disappearance. It’s a common method of diagnosing neuromas. However, as fibres from the former court core are on the shallower side when they cross through the brain bridge to the opposite side, and are vulnerable to the tumours of the small brain horns of the large bridge, approximately 10 per cent of the body side of the front court function can be impaired.
3. Video inspection
(1) Skull X-rays: the skeletal skeletal tablets see the expansion of the inner ear, bone erosion or bone absorption.
(2) CT and MRI scans: CT is expressed as tumour equivalent or low density, with few high density images. The tumours are mostly circular or irregular and are located in the inner-heart mouth zone, with a multi-acoustic expansion, with a clear multiplier effect. The MRIT1 weighted image displays a slightly lower or equal signal, and the T2 weighted image displays a high signal. The fourth brain chamber is pressured into deformation, as are brain stem and small brain deformation. The tumour substance of the injection is significantly reinforced and the cystic zone is not reinforced.
