What’s an IBD? Inflammatory Bowel Disase, IBD is a group of chronic intestinal inflammation diseases for which there is no clear cause, which mainly includes two types: Crohn’s Disase, CD and Ulcerative Colitis, UC. They can lead to inflammation and ulcer in the intestinal mucous membranes, in the form of repeated abdominal pain, diarrhoea and digestive disorders. 1. Inflammatory enteropathy (UC) is mainly confined to mucous membranes in the colon and rectum. The pathology ranges extend from rectal to rectum and are distributed continuously. Common symptoms include haemorrhage, diarrhoea and lower left abdominal pain. 2. Cronn Disease (CD) can stretch through the whole digestive tract, from mouth to anal, but most commonly at the end of the intestine and colon. The inflammation distribution is ejected and can stretch to all levels of the intestine wall. Complex symptoms such as abdominal pain, intestinal infarction and fistula may occur. The exact cause of IBD ‘ s causes of inflammatory intestinal diseases is not yet fully established and is now considered to be the result of a combination of genetic, immunological and environmental factors. 1. Genetic factors IBD have a certain degree of family concentration and certain genetic variations may increase the risk of disease. The intestinal immune system reacts excessively to normal strains or harmless antigens, causing inflammation to spiral out of control. 3. Environmental factors. Diet: High fat and sugar diets may be associated with IBD morbidity. Infection: Certain viruses or bacterial infections may be the cause. Smoking: This is closely related to the increased risk of Cron disease, but is protective of ulcer colonitis. Environmental changes in intestinal microorganisms may trigger or exacerbate inflammatory reactions.
Symptoms of inflammatory enteropathy 1. Symptoms of digestive tract abdominal pain: usually in the lower abdomen or the lower right abdominal. Diarrhoea: Repeated outbreaks, which can be accompanied by blood or slime (ulcer ulcer enteritis is more common). Weight loss: due to absorption disorders and inflammation consumption. Anal ecstasy: e.g. anal fistula, anal ectoplasm (mostly Crohn). 2. All-body symptoms. Heat: Inflammation activity may be low or high. Wearyness: reduced physical strength due to chronic inflammation. Malnutrition: Vitamins and minerals in particular. 3. Intestinal manifestations Arthritis: Tired and large joints, possible joint pain and swelling. Skin disease: e.g. erythrocyte or noma sepsis. Eye diseases: e.g., grapes, filaments. Hepatic cholesterol disease: PSC. Diagnosis of inflammatory intestinal diseases 1. History and medical examination 2. Endoscopy: colonoscopy: observation of inflammation and ulcers in the colon and rectal, to be clearly diagnosed by a biopsy. (c) Capacitors: used for the examination of intestines, especially for Crohn. 3. Visual screening MRI and CT: used to assess the extent of intestinal inflammation, fistula and intestinal infarction. Intestine imaging: help to detect intestines, especially in the case of Crohn. 4. Laboratory tests Blood routines: anaemia, white cell altruism. C Reacting Protein (CRP): Reflects the state of inflammation. Calcium faeces: assessment of intestinal inflammation activities. Pathological examinations confirm pathological changes, such as gland structural damage or bulge formation, through active examinations. Treatment of inflammatory intestinal diseases The objective of treatment is to control symptoms, mitigate inflammation, prevent complications, including medication, nutritional support and surgical treatment. 1. Drug treatments 5-amino-acrylic acids (5-ASA): e.g., metallazole, for the control of mild moderate inflammation of ulcer colonitis. Sugar cortex hormones: e.g., Peneson, applicable during acute onset but not suitable for long-term use. Immunosuppressants: e.g. sulfur, ammonium butterflies, which are used to sustain the mitigation and reduction of hormone dependence. Biological preparations, such as anti-tumour cause of death (TNF) drugs, for moderately severe patients, with significant results. Antibiotics: e.g., americium, which can be used for co-infections with Kronn or fistula. 2. Nutritional support Intestine nutrition: high protein, low fat diet helps control inflammation. Vitamin supplementation: for example, vitamin D, B12 and iron. In vitro nutrition: for patients with severe malnutrition or enteric infarction. 3. Surgical treatment ulcer colonitis: may require colon removal if the drug treatment is ineffective or serious complications (e.g., mesotoxic coliform, cancer). Cronn Disease: While surgery is mainly used to treat complications such as narrowness, fistula and sepsis, it cannot be cured and will continue to require medication.
1. intestinal complications 1. intestinal infarction, fistula, sepsis, haemorrhage, perforation. Long-term ulcer colonitis may increase the risk of colon cancer. 2. Osteoporosis, anaemia and malnutrition. Increased risk of infection due to drug side effects, such as sugar cortex hormones.
1. A healthy diet that avoids high fat, sugary foods, adequate intake of dietary fibres and digestible foods. The diet is as light as possible, avoiding alcohol and stimulating food. 2. Regularity. Adequate sleep and appropriate exercise enhances immunity. 3. Psychological support. Chronic diseases can lead to anxiety or depression and patients should seek professional psychological help or join the patient support group. 4. Regular follow-up visits and periodic medical examinations to assess the efficacy of the drug and to prevent complications. As medical research advances, new treatment methods and biological agents are increasingly being developed, providing better control for IBD patients. Although not entirely curable at present, scientific management can help patients maintain a good quality of life. Inflammatory intestine disease is a complex chronic disease, but with drug treatment, dietary adjustment and improved lifestyle, most patients are able to control symptoms and maintain a normal life. Patients with digestive symptoms should be treated as early as possible, clearly diagnosed and individualized. Scientific management and proactive response are key to addressing IBD.
Inflammatory enteria
