Rheumatism is a large and complex category of self-immunizing diseases whose inflammation is by nature the key to understanding the occurrence, development and treatment of such diseases.
The inflammation of rheumatism is not caused by common infectious factors but by sterile inflammation of the immune system. However, in cases of rheumatism, the immune system is misidentified, treating its own tissues and organs as external antigens, which in turn triggers an immune attack and inflammation response.
The onset of the inflammation response is related to a number of factors. First, genetic factors play an important role in the incidence of rheumatism, and the presence of certain genes may make individuals more vulnerable to rheumatism. Second, environmental factors such as infection, trauma, stress, etc. can also induce immunisation imbalances, leading to rheumatism.
When inflammation of rheumatism causes a series of pathological changes. Among them, cytogens and inflammatory media, such as cytogenesis, have been released in large quantities, like “showers”, concentrating inflammation cells, such as meteosat cells, and megacormic cells, which, for example, immerse into the joints, produce increased growth and oedema, form vascular hysteria, thereby destroying the joint cartilage and bone tissue, and eventually lead to joint malformations and functional disorders.
Different types of rheumatism disease have different manifestations and effects. Physico-wettic arthritis is a major violation of the small joints of four limbs, symmetrical distribution, it leads to the continued growth of the joints and the inflammation of inflammation cells, and the patient suffers from joint pain, swelling, morning stagnosis and, as the disease progresses, it causes the gradual destruction of the cortex and bone, which seriously affects the normal functioning of the joints.
The inflammation of rheumatism is also reflected in its chronic and persistent nature. Inflammation tends to persist in the event of an outbreak, with repeated outbreaks and constant damage to body tissue. This chronic inflammation not only leads to direct damage to joints and tissues, but also causes a range of complications, such as cardiovascular diseases, lung diseases, osteoporosis, etc., which further exacerbates the patient ‘ s condition and reduces the quality of life.
With regard to the nature of rheumatism, the key to treatment is to control the inflammation response and to regulate the immune system in order to alleviate symptoms, delay progress, protect joints and organ functions. With regard to medication, acne anti-inflammation drugs can alleviate pain and inflammation; sugar cortex hormones have a strong anti-inflammation effect, but need to be used with caution to avoid side effects; immunosuppressants can be used to control progress; biological agents and target treatments can be more precisely tailored to specific inflammatory factors or immunocellation cells, and can effectively contain inflammatory response and organizational damage. Physical therapy, such as heat dressing, cooling, electrotherapy, etc., can relieve pain and muscle stress; rehabilitation and exercise can help to increase muscle strength, improve joint function and improve the patient ‘ s ability to self-care.
In sum, rheumatism is essentially a disease of sterile inflammation caused by the abnormal activation of the immune system, resulting in a series of pathological changes that can cause damage to joints, bones, skin and other organs. Understanding the nature of rheumatism is important for early diagnosis, sound treatment and patient self-management. Through comprehensive treatment and the active collaboration of patients, it is possible to effectively control inflammation, reduce symptoms, improve the quality of life and allow patients to live with the disease.
