Cardiac disease
Chrysotic heart disease is caused by a series of heart changes caused by the adverse effect of thyroid poisoning on the heart. The main cause of thyroid heart disease is the high levels of thyroid hormones in the body when thyroid function is hyperbole. Excessed thyroid hormones have multiple adverse effects on the heart: – Direct effects on myocardial muscles: significant acceleration of myocardial metabolism, increased oxygen consumption of myocardial cells, resulting in increased myocardial constriction, increased heart rate, etc., and long-term structural changes in myocardial fat. – Impacts on the Electronology of the Heart: Changes in the electrophysiological properties of myocardial cells, which can cause various cardiac disorders, such as common cardiac tremors, and the possibility of early pace. – Surrounded vascular effects: contributing to the expansion of the outer vascular vessels, increasing the respiratory blood mass, increasing the pre-heart load, further affecting the cardiac function, which, under long-term effects, can lead to a heart attack. Clinical performance The clinical manifestations of cardiac disease are as follows: – Heart arrhythmia: most common, with vibrancy in the heart, early paces (rooms early, room early), etc., can cause discomfort, panic, etc. – Cardiac amplification: an increase in the size of the heart can be detected through examination, such as an ultrasound of the heart, which may be unsymptomatic at an early stage and may affect the function of the heart at a later stage. – Heart failure: mostly manifested in right-heart failure, which can be characterized by respiratory difficulties (aggravated activity, severe breaks, etc.), oedema (a common edema of the lower limbs, which can also have edema of the whole body), and inefficiency. – Cardiac pain: A small number of patients may be found, mostly as a result of increased oxygen consumption of myocardial muscles and relative blood deficiency of the coronary, as shown by pain in the frontal zone. – Declination of the doppelgangers: some patients can be found, generally with no visible symptoms, a small number can be shown with chest pain, difficulty in breathing, etc., and can be detected through a heart ultrasound, etc. In the case of thyroid heart disease, the following examinations are usually required: The seroprevalence of the triiodine thyroid aminoacin (FT3) and the exciparant thyroid glandin (FT4) are generally elevated; the thyroid hormones (TSH) decrease is clear for the thyroid state. – Cardiac-related examinations: – EEG: Cardiac disorders, common anomalies such as cardiac tremors, early strokes, can be detected, and changes in myocardial hemorrhage can be shown. – Cardiac ultrasound (ultraheart artery): can look at the structure and function of the heart and see whether the heart is increased, myocardial graft, valve abnormalities and heart function indicators, such as blood-shot fractions. – Dynamic electrocardiogram monitoring (Holter): Changes in electrocardiograms of 24 hours or more can be recorded in a continuous manner, which can help to capture acoustic cardiac disorder. – Breast X-ray: can observe heart size, morphology and lung, and determine if there are signs of heart increase and lung haematosis. – Other examinations: – Antibody examination of the thyroid itself: for example, thyroid receptor antibodies (TRAb), thyroid peroxide enzyme antibodies (TPOAb), which helps to identify the causes of thyroid disease. – Radionuclide screening: for example, thyroid iodization measures provide information on the extent of thyroid hyperactivity and uptake of radioiodine, assistive diagnosis of the iodization and guidance on subsequent radioiodine treatment. – Coronary hysteria: In case of suspicion of coronary hysteria, such as coronary heart disease, it is possible to make it clear whether the coronary coronary is constricted or not, but less so in the case of cardiac pains due to coronary hysteria. Diagnosis needs to be combined with a combination of thyroid history, associated cardiac signs, as well as a supportive examination of thyroid function (e.g., an increase in serum thyroid hormone levels), electrocardiograms (e.g., showing abnormalities such as cardiac disorders), heart ultrasound (see changes in heart structure and function). Therapeutic – Control of thyroid: is a key link in the restoration of thyroid function through drugs (e.g., antithyroids such as mastium, radioiodine treatment or surgical treatment). – Treatment of cardiac symptoms: corresponding anti-cardiological disorders can be used in the case of heart failure; heart failure is treated in accordance with the principles of heart failure treatment, such as urine, blood vessels, etc.
Cardiac disease
