” Stomach cancer and cholesterol: the hidden link between bacteria and stomach cancer “

” Stomach cancer and cholesterol: the hidden link between bacteria and stomach cancer “

In exploring the pathogen of stomach cancer, this tiny bacteria, the claustrophilus, cannot be ignored. It is inextricably linked to the occurrence of stomach cancer, so let’s unmask the mystery of their connection.

I. Sphinx, “Infrequent visitors” in the stomach

Sphinx is a grenacella fungus that lives mainly on the surface of human stomach mucus. It is very crafty and able to survive in a very acidic environment, such as gastric acid, thanks to its unique spiral structure and its own special enzymes, which help it to resist the erosion of stomach acid.

Moreover, the cholesterocella is very contagious, and it can be transmitted through mouths, such as the sharing of dishes, water cups, or intimate contact behaviour such as adult kissing of a child, which may lead to the transfer of cipococcal from one person to another, thereby “settling” in the stomach of the new host.

II. Disruption of the gastric mucous membranes by the fungus cucumber

Once the fungus is standing on his feet in his stomach, it’s starting to be “floating.” It uses its own various enzymes, such as urea enzymes, which decompose urea to produce ammonia, which can moderate and gastric acid, making the fungus a relatively alkaline environment around which it can survive. At the same time, however, it also alters the original balance of acid in the stomach and affects the normal physiological function of the stomach.

More importantly, the fungus also releases some toxins, such as cytoxin-related protein A (CagaA) and bubble toxin A (VacA). These toxins directly attack epipelagic cells in the gastric mucous membranes, causing damage to the cell membrane, causing the release of the substance in the cell and the disruption of the normal structure and functioning of the cell. Such attacks over a long period of time have led to inflammation of the gastric mucous membranes, which have evolved from mild shallow stomach inflammation, which is an important “prelude” to stomach cancer.

III. Inducing a stomach immune response and cell mutation

The presence of the fungus in the stomach also triggers an immune reaction to the stomach. When the body’s immune system detects this “external invader” in its stomach, it sends immune cells to attack it. However, in the course of this confrontation, some of the substances released by immunocellular cells, as well as some of the chemicals resulting from the persistence of the inflammatory response, may cause damage to the DNA of the epipelagic cells of the stomach mucous membrane and mutation of the genes within the cell.

When the cell’s genes mutate, it’s like the cell’s “run process” is a problem, and cells may begin to grow and divide uncontrollably, and then normal cells may gradually become cancer cells. Although not all those infected with the fungus have stomach cancer, it undoubtedly increases the risk of stomach cancer.

IV. Promotion of micro-environmental formation of stomach tumours

In addition to causing direct damage to the upper skin cells of the gastric mucous membranes and triggering cell mutations, the fungus coliculus encourages the formation of micro-environments of tumours in the stomach. It affects the interaction of various cells in the stomach, alters the composition and properties of the extracellular matrix and makes the environment in the stomach more conducive to the growth, growth and transfer of tumour cells.

For example, it would disable some of the cytogens that would otherwise inhibit the growth of tumours, while at the same time contributing to the creation of a large number of cytogens that are conducive to tumour development, thus creating a relatively “friendly” environment for stomach cancer and development.

Although a small bacteria, the fungus of the fungus can be very damaging to the stomach, and it is closely associated with the occurrence of stomach cancer in a variety of ways, including the destruction of the gastric mucous membranes, the triggering of cell mutations and the promotion of micro-tumour formation. Therefore, in our daily lives, we need to be careful about food hygiene and to avoid, as far as possible, the infection of the fungus, and, if the infection is detected, to be treated in a timely manner in order to reduce the risk of stomach cancer.