Comprehensive treatment for acute coronary syndrome
Acute coronary syndrome (Acute Coronary Syndrome, ACS) refers to a group of clinical symptoms of myocardial insufficiency, anaerobics due to narrow or obstructive coronary arteries, including unstable aorexia and acute myocardial infarction. The severity of the disease and the magnitude of the complications have become one of the major threats to human health. The treatment of acute coronary syndrome requires an integrated treatment strategy to minimize morbidity and mortality rates and improve patient prognosis. i. The mechanism for the onset of acute coronary syndrome and the mechanism for the occurrence of acute coronary syndrome are closely linked to the sclerosis of coronary porridge samples, the narrowness of the coronary vein and the formation of a thrombosis. When the coronal is narrow or blocked, the blood supply of the myocardial muscles is reduced, resulting in myocardial insufficiency, anaerobics and, in turn, infarction, etc. The danger of acute coronary syndrome is the rapid progress of the disease and the high rate of death, which seriously threatens the life of the patient. ii. Comprehensive treatment strategy for acute coronary syndrome 1. Acute treatment Re-infusion treatment is the key to acute coronary syndrome acute treatment, which reduces myocardial myocardial damage by rapidly restoring coronary blood flow. Common re-infusion treatments include coronary artery intervention therapy (PCI) and intravenous embolism treatment. Anti-sculpture and anti-condensation treatments are designed to prevent the formation of clots and reduce the rate of disease and death. Stabilisation treatments include, inter alia, the use of carcasses to reduce blood resin, stabilize plasters and prevent cardiovascular events. Anti-embolism treatment: The inclusion of both the anti-blood plate and the anticondensation components prevents the further formation of a coronary internal embolism, thus preventing the occurrence of a complete closure of the cavity of the coronary artery and reducing the risk of progress and heart-borne death. Anti-sphygmopolytic cyclic oxidation inhibitors, non-ST lift acute coronary syndrome patients should be rapidly given aspirin to inhibit slab accumulation. Diphosphate gland receptor agonizer, which was chosen as Greilo ‘ s replacement during the resistance of chlorpyrey. Carbine membrane protein receptor blocker, for Rophebane. Diesterase inhibitor for cyclonuclear phosphate, as an alternative to a patient who cannot be applied by aspirin and chlorplycerine (Ciloxazole). Condensation treatment Sulphur to sodium, with limited recommended use for anticondensation treatment throughout the period of hospitalization. Hepatin, currently recommended for Hepatrin, 2-8 days. Bivalulud, a direct anticondensation drug, reduces the risk of intervention in the treatment of acute coronary thrombosis during the immediate surgery. Anti-myocardial ischaemic treatment Nitrate esters, with nitrite glycerine under the tongue, with long-term oral exposure to nitrates, with regard to the possibility of nitrate resistance. Analgesics, such as nitrates, do not relieve pain and morphine treatment should be provided immediately. The beta receptor blocker, in the case of heart failure, low output, heart-borne shock risk or other taboos, is to begin to take the beta receptor blocker for the first 24 hours, with the use of the amber acid metolor and the pesorol. Calcium route blockers, calcium condensers of thiomers, but the combined heart failure should be used with caution for the time of the failure and for the apparent slowness of the heart. Nephrine-vascular stressors-alphatesterone inhibitors, such as ACEI/ARB-type drugs, including Inapli, Peppli, Zolzatan, Temishatan, etc. Plasma-based treatments, such as Atophate, Rishuptadine, etc. Chronic treatment The medications include the long-term use of aspirin, carpentine, calcium ion stressants, etc., to reduce blood resin, expand the coronary artery and improve myocardial insemination. Interventions and coronary artery bridgings improve the quality of life of patients by re-establishing coronary blood flow, improving myocardiosis. Rehabilitation treatment is an important component of acute coronary syndrome treatment. When the condition is stable, the patient should participate actively in rehabilitation, including appropriate physical activity, psychological guidance, health education, etc. Rehabilitation treatment helps to improve the cardiovascular function of patients and reduce the risk of reoccurrence of cardiovascular events. Lifestyle adjustments. Lifestyle adjustments are important for the treatment of acute coronary syndrome. Patients should refrain from smoking and alcohol, proper diet, weight control, regularity, good mentalities, etc. These measures help to reduce the risk of cardiovascular events and improve the prognosis.
Coronary heart disease. Acute myocardial infarction.
