A digestive ulcer is a chronic ulcer that occurs in the stomach or in the inner wall of the larvae (the first part of the intestine below the stomach). Such ulcer is mainly caused by the erosion of gastric acids and gastroprotease on gastric mucous membranes or membranes of the mesmopolis, resulting in local tissue deficiencies. Depending on where it occurs, the digestive ulcer can be divided into two categories: stomach ulcer and 12-finger ulcer. Of these, the ulcer is more common with the 12 fingers.
I. Causes of digestive ulcer
The causes of the disease in digestive ulcer are more complex and relate mainly to the following:
1. Sphinx infection: Helicobacter pylori, H. pylori infection is one of the most common causes of digestive ulcer. This bacteria can break the gastric mucous membrane barrier, making it easier for gastric acid to erode the stomach or the larvae wall, thereby triggering ulcer.
2. Use of non-paralytic anti-inflammatory drugs (NSAIDs): Long-term or excessive use of non-inflammatory drugs, such as aspirin, Broven, etc., can damage stomach mucous membranes, reduce their resistance and promote ulcer formation.
3. Excess stomach acidity: In some cases, such as the Zollinger-Ellison syndrome, there has been an increase in gastric acidity anomalies in patients, increasing the risk of damage to the stomach mucous membranes.
4. Genetic factors: The risk of disease is relatively high among groups with a history of digestive ulcer in the family.
Lifestyle factors: Bad living habits such as smoking, drinking and high stress are also considered as a potential incentive for digestive ulcer.
II. Clinical performance of digestive ulcer
Typical symptoms of digestive ulcer include:
• Upper abdominal pain: The pain is usually in the area between the lower part of the chest and the belly button, with cyclical effects, especially when the abdominal is empty. For the ulcer, the pain occurs more than two to three hours after the meal, while the ulcer may feel uncomfortable immediately after the meal.
• Disgusting and vomiting: Some patients may have symptoms such as nausea, vomiting and possibly blood in serious vomiting.
Weight loss: Patients may experience weight loss due to pain and appetite loss.
• Black defecation or vomiting of blood: When an ulcer penetrates the vein, it can lead to ingestional haemorrhage, in the form of black tar or vomiting.
III. Indigestion ulcer treatment and prevention
Treatment:
1. Eradicating fungus of cholesterol: for patients who are H. Pylori-positive, root treatment is provided with a combination antibiotic proton pump inhibitor (PPIs) or stomach mucous protection.
2. Reduction of stomach acidity: Reduction of stomach acidity by using proton pump receptor inhibitors (PPIs) or a new generation of potassium ion competitive acid retardants (PCAB) to mitigate symptoms and promote ulcer healing.
3. Lifestyle adjustments: cessation of smoking, drinking restrictions, avoidance of overwork, maintenance of good eating habits, help to control the condition.
4. Drug management: For patients requiring long-term use of NSAIDs, doctors assess risks and consider the use of gastric mucus protections to prevent ulcer occurrence.
Prevention:
1. Healthy diet: balanced diet, avoiding spicy, greasy food and reducing the irritation of stomach mucous membranes.
2. Periodic medical check-ups: The group with a history of digestive ulcer should carry out regular stomach lenses for early detection.
3. Rational use of medicines: Medically prescribed use of drugs, in particular those of the NSAIDs type, supplemented, if necessary, by gastric mucous protection measures.
4. Psychological regulation: remain optimistic, exercise appropriate and reduce stress on life and work.
The digestive ulcer is a very common gastrointestinal disease, the treatment of which depends not only on medical means but also on the adaptation of patients in their daily lives. Through comprehensive treatment and active prevention, the vast majority of patients have effective control and recovery.
Indigestion ulcer.
