The hearing of a neuromass (former neuroplasma) is a benign tumour originating from the eighth pair of neural tumours, mostly from the frontal neurological. The neoplasm accounts for 8-10 per cent of the tumours in the skull, 80-90 per cent of the tumours in the small brain, most of them among adults aged 30-60 years, with a male-to-female ratio of 2:3 and an annual increase of 2.28 per 100,000 per year, thanks to advances in diagnostic methods. Most of them are single-sided, and very rare on both sides. The growth of tumours is generally slow, increasing by an average of 0.25 – 0.4 cm per month, with individual tumours increasing by 2 cm within one year.
At outpatient clinics, “hearing a nervous tumor” and “crash deafness” are often confused. Actually, the two are very different.
Listen to neuromas and sudden deafness.
The hearing of patients with neuromagnosis can decline gradually for several years until they are fully deaf. There are three forms of hearing decline, with chronic performance at 87 per cent, sudden deafness at 10 per cent and volatile hearing at 3 per cent. At an early stage, the hearing decline for hearing patients with neuromagnosis was one-sided or asymmetric, tiring and high frequency. The decline in the recognition rate for neuromagnosis is disproportionate to the loss of pure audio.
In addition, the ringing of the ears is a common symptom of the hearing of patients with neuromas. Patients sound in high-frequency (HF) sound, which sometimes changes, and sometimes disappears for some time. The sound can be intermittent, it can be continuous, it can be intransigent, and it continues until the hearing is completely lost. An ear ringing can be the only symptom and can be accompanied by other symptoms.
Hearings have suddenly declined and are often diagnosed as “crash deafness”, although only 1 to 2 per cent of sudden deaf persons have finally been confirmed to have a hearing tumor.
Sudden loss of hearing may be the result of an internal hearing artery convulsion or blockage caused by tumour oppression, which can be induced by head trauma or severe motion. Hearing decline can be fully deaf and has the potential to recover, but the restoration of hearing does not preclude the possibility of hearing a neuromagnosis. Twenty-six per cent of patients with neuromagnosis have a history of sudden deafness. It is therefore essential that patients with sudden-onset deafness be treated with a positive attitude that precludes the hearing of neuromas.
Listening to neuromas, although a benign tumour, only early detection can provide a good prognosis and retain hearing through active treatment to preserve the function of facial and hearing nerve. Therefore, an MRI examination should be carried out on the inside when there is an unknown single ear ringing and deafness.
As the condition develops, deafness among patients with neuromagnosis increases. Due to the gradual expansion of the tumour internal hearing, neuropressure of the inside of the skull can occur when it reaches the small brain corner of the bridge.
1 The cochlear and front court symptoms are in the form of ringing ears, deafness, dizziness and dizziness;
2. Discomfort in the area where the head pain of the forehead is accompanied by a large hole in the side of the pillow;
3. Motivation and incoherence;
4. Symptoms of conjunctive neurological damage, such as facial pain, facial convulsions, diminished facial feelings, and sexual palsy in the surrounding area;
5. Symptoms of increased intracircle pressure, such as osteoporosis, increased headache, vomiting, re-vision, etc.
In the event of the above-mentioned symptoms, the patient should visit the neurosurgery in a timely manner for examination and treatment in order to prevent further consequences of the delay.
