Spectrum convulsions are muscular, non-autonomous convulsions that occur repeatedly in one or both sides of the facial muscles (eye rim muscles, emoticon muscles, mouth wheel muscles) and increase during emotional or stress periods, with serious eye opening difficulties, slanting of the mouth and hysteria in the ear. The facial convulsions include both typical and non-typical facial convulsions, which refer to the onset of the convulsions from the eyelids and the gradual downward development of lower muscles, such as those with tired and face and cheek expressions, and not to the onset of the convulsions from the lower side of the muscles, and to the progressive development of the last convulsions and the eyelids and foreheads. Clinical atypical aerobic convulsions are relatively low, with most typical aerobic convulsions. Noodle spasms are better in middle and old age, with slightly more women than men, but the age at which they occur tends to be younger. While most of the aerobic convulsions are on one side, it is not uncommon to have a bipolar muscle convulsion.
How do you diagnose and identify facial spasms?
The diagnosis of facial spasms relies mainly on characteristic clinical manifestations. Patients lacking characterizational clinical representation need to be identified with assistive examinations, including electrophysiological examinations, visual examinations and the Camassipin treatment experiment. Electrophysiological examinations include tests of myotromygraphy (EMG) and abnormal muscle response (AMR) or side-speech (lateral spread response, LSR). Visual examinations include CT and MRI, which are used to identify intracranial pathologies that can lead to facial spasms, and help to understand the vascular distribution around facial nerves. At the beginning of the disease, patients with a facial spasm are generally effective for the Camassipin treatment (a small part of the patients can be ineffective), so that the Camassipin treatment experiment is useful for diagnosis.
The identification and diagnosis of facial avulsions requires the identification of both sides of the avulsions, the Mejé syndrome, the bite of the muscle avulsions, and the presence of facial disorders.
1- Double eye worms: Involuntarily, manifested in the repeated inactivity of the eyelids on both sides, often accompanied by both eyes, with patients often displaying difficulties and a decrease in tears, and as the path goes on, symptoms remain limited to both eyes.
2. Mejé syndrome: Patients often suffer from repeated inactivity with double eyelids, but as the path of the disease is prolonged, there is a gradual loss of the eye and an uninvolved withdrawal of the muscles below, in the form of anomalous movements on both sides of the face, and as the disease increases, the extent of muscle spasms increases downward, even to the neck, limbs and torso muscles.
3. Bite myocardial convulsions: For one-sided or double-sided thawing of the muscle, the patient may have different levels of upper and lower twitching disorders, grinding of teeth and opening difficulties, one of the possible causes of trident neuromotor hysteria.
4. Symptoms of palsy: limited activity in the muscles of the same side face, the same side angular discomfort and the associated motion of the mouth and the eye, which can be identified on the basis of a precise history of facial palsy.
