The description of antibacterials as antibacterials or antibacterials, including various antibacterials and chemical synthetics, is given in terms of their common types such as beta-nimamine and large cyclic esters.
(i) Beta-nimide
Beta-nitromamine antibacterials are highly active, diverse and widely used. Among the common drugs of penicillin are penicillin G, methoxysicillin, ammonia sicillin, ammosicillin, etc. Penicillin-related proteins (PBPs) in bacteria are highly proximate and, when combined, interfere with the synthesis of bacterial cell walls, leading to the cessation of bacteria ‘ growth, dissolution and death. Drugs such as head acne are acne, sodium acne, sodium acne, hair acne, hair acne, hair accelerant, etc. Such drugs have the advantage of broad antibacterial spectrometry, low toxicity, less allergic reactions to penicillin, high antibacterial efficacy, penicillin enzymes and high clinical efficacy. The atypical β-neamides include carbon methacne, carcinin, etc., and represent the drugs of amphetamine, sepamide, etc.
(ii) Large ringed esters
The large mercuric esters are commonly used for the treatment of infections such as oxygland positives and gland vaginal sphericals. Common drugs are erythacin, achicin, gitacin, erythrin, carcinin, etc. The first generation of Great Ringed Iester antibiotics include erythrin, amethrin, etc. The most representative of the second generation are Archicillin, erythroacin, Kracin, etc. The third generation of the Great Ringed Iester, which represents the drug with Telicin. It is relatively narrow and works well for a wide range of pathogens, usually antibacterial, and at high concentrations.
(iii) Lincoxin
The Lincoxin category includes drugs such as Lincoln and Clincin.
(iv) Amino sugar
Amino sugar is an effective antibiotics, especially for aerobic glycol. Common drugs include cythin, Tobcocin, Amikane, etc. This drug has characteristics such as a good water solubility, stable nature and anti-bacterial spectrometry, but with varying degrees of ear and kidney toxicity.
(v) Tetracycline
The tetracyclic group is a rapid antibacterial agent for the gland positive and the vaginal bacteria, and also has a strong inhibition effect on secondary, chlamydia, etc. The most common drugs are supercycline, Minocycline, tetracycline, earthin, etc.
(vi) Synthetic antibiotics
Artificial antibiotics commonly include quinone-like substances such as cyclopropa and quinol; sulfamide-like substances such as sulfamide. Drugs such as quinone have a wide spectrum of antibacterials, a wide distribution in the body, a long half-life elimination and a reduced number of adverse effects.
II. Mechanisms for the functioning of anti-bacterial drugs
(i) Suppression of bacterial cell wall synthesis
Antibacterial drugs that inhibit the synthesis of bacterial cytowalls have the effect of microbicide by destroying the cell wall, bringing the hydrometric material outside the cell into the cell, causing cell swelling and death. Commonly used are penicillin, headgillin, phosphoxin, cyclosylic acid, vancin, bacterium, etc. For example, penicillin-related proteins (PBPs) in the cycin-like drug and bacteria have a high degree of affinity, which, when combined, interferes with the synthesis of bacterial cell walls, leading to the cessation of bacteria ‘ growth, dissolution and death. Drugs such as capisculin have the advantage of a wide spectrum of antibacterials, low toxicity, less sensitive reactions to penicillin, high antibacterial effects, penicillin enzymes and high clinical efficacy. Vancocin is an antibiotic of aluminum, which inhibits the synthesis of bacterial cell walls, alters the permeability of cellular membranes and is used in clinical treatments for serious infections caused by, for example, methoxysyltin yellow grapes.
(ii) Change in cytochrome permeability
The antibiotics that alter the permeability of the cytogens are mainly caused by the destruction of the cytogens, resulting in the excretion of proteins, amino acids, etc. in the cytogens, leading to bacterial deaths. Antibiotics such as polymixer E, whose anion combines with phosphorus resin in the cystal membrane, impairs the membrane function; anti-facteria pepcin B can selectively combine with the ecdote steroids in the fungal membrane to form a pore, alters the membrane permeability and causes the death of fungi by leaks in proteins, amino acids, nucleic acids, etc. in fungi.
(iii) Depression of protein synthesis
Antibiotics that inhibit protein synthesis serve the purpose of microbicide by inhibiting the synthesis of normal platinum chains. For example, Clocin, Lincoln. This type of drug works at different stages of bacterial protein synthesis: in the initial stage, amino-sugar antibiotics prevent 30S and 70S sub-synthesis complexes; in the cylindrin extension phase, tetracyclic antibiotics combine with nuclose 30S and nuclei, hindering the formation of peptonic chains, producing antiphylactic effects, chlorinoxin and lycin inhibiting pyramide transferases, and in the macro-cyretanone inhibiting transcontinent enzymes; in the end phase, the amino-sugar antibiotics prevent the termination of the factor with A, hindering the nuclei cycle, and synthesizing abnormal or non-functional pelican chains, thus contributing to bacteria.
(iv) Impacting nucleic acid and folic acid metabolism
Antibiotics that affect nucleic acid and folic acid metabolism prevent bacteria from synthesizing folic acids and affect the synthesis of nucleotide, thus preventing bacteria from breeding for fungi purposes. For example, quinone, such as an oxyfluza, inhibits bacterial DNA revolving enzymes, thus inhibiting bacteria’ DNA reproduction and fungicide; and rifpint inhibits bacteria’ DNA-dependent RNA polypolytic enzymes, which prevents mRNA synthesis and kills bacteria. Different antibacterial drugs have different mechanisms and different bacteria are targeted, so they should be used under the guidance of a doctor and not on their own.
Attention to the use of antibacterial drugs
(i) Identification of pathogens
Prior to the use of anti-bacterial drugs, it is essential to conduct relevant examinations to determine whether the patient is bacterial. Pathological examinations, for example, can help identify pathogens and provide a basis for subsequent precision treatment. In the case of cases of fever and cold warfare, a combination of bacterial infections is generally considered, but it is not possible to use antibacterial drugs blindly on the basis of symptoms alone, which must be confirmed by scientific means of examination.
(ii) Understanding of drug-sensitive strains
Different antibacterials have specific extinction effects on different strains. For example, penicillin is mainly active in the Gelancian positive fungi and the Gelancne vaginal fungus; in the Gelancian positive fungus and some Gelancella cactus, the second generation is more active in the Gelancian vaginal fungi, and in the third generation, it is very active in the Gelancella cactus, but less active in the positive fungi. It’s also like corrosive esters, a narrow spectrovelocity antibacterials, which are similar to penicillin G, mainly aerobic gland-positive, gland-negative and anaerobic fungus, legionary fungus, steroids.