The digestive ulcer is a common disease of the digestive system, posing many ills and potential risks to patients. Knowledge of this knowledge helps us to better prevent and respond.
I. What’s a digestive ulcer?
The digestive ulcer is mainly related to chronic ulcer in the stomach and the 12-finger bowel, i.e. ulcer in the stomach and ulcer in the 12-finger. These ulcer formations are closely related to stomach acid and gastroprotease digestion. When there is an imbalance in the protection mechanisms of the mucous membranes, the stomach acid and gastroprotease have a potentially invasive effect on the stomach or on the membranes, leading to ulcer formation after long periods of accumulation.
II. Symptoms
Pain: As the main clinical manifestation of the digestive ulcer, most of the pain caused by the stomach ulcer occurs 30 minutes to 1 hour after the meal and is gradually alleviated after one to two hours until the next meal repeats this pattern. In contrast, the pain caused by the ulcer of the 12-finger intestine is mostly caused by an empty abdominal state, which is common between meals or at night, and can be mitigated by eating or taking antiacid drugs. The nature of pain is diverse and can be manifested in blunt pain, burns, pain, severe pain or a sense of discomfort similar to hunger.
Other symptoms: Symptoms of digestive disorders such as anti-acid, gas, nausea, vomiting and anorexia may also be associated. Some of the patients may have symptoms of digestive haemorrhage, such as vomiting, black poop, which can lead, in serious cases, to symptoms of anaemia such as dizziness, panic and inactivity, and even to signs of acute abdominal pain and abdominal stress caused by perforation.
III. Common causes of illness
1. Sphinx (Hp) infection: This factor is one of the key causes of digestive ulcer. The fungus can be planted in a gastromus membranes, creating multiple enzymes and toxins, disrupting the barrier function of the gastromus, and causing inflammation, which in turn leads to ulcer formation.
2. Drug factors: Long-term ingestion of non-paramoxin anti-inflammatory drugs (e.g. aspirin, Broven, etc.) and sugary cortex hormones can lead to inhibition of biosynthesis of the stomach mucous prostate and impairment of the protective barrier of the stomach mucous membrane, increasing the incidence of ulcer.
3. Stomach acids and gastroproteases: excessive gastroacidity and increased activity of the gastroprotease, with its own digestive effects on the stomach and on the membrane of the membrane, are direct causes of ulcer formation.
4. Life habits and mental factors: Long-term smoking, alcohol abuse, irregular eating patterns (including severe diets, excessive diets, frequent ingestion of spicy irritating foods, stress, anxiety, depression, etc.) can have a negative impact on the blood cycle of the gastric mucous membrane and its neuroregulation, thus weakening the mechanisms for the defence of the stomach mucous membrane.
IV. Diagnosis
1. Stomach mirror examination: As a preferred means of identifying digestive ulcer, endoscopy can accurately reveal key information such as the anatomic position, size, morphological characteristics and quantity of the ulcer and can be examined for pathology in the direct view of the tissue in order to exclude malignant pathologies.
2.X Concourse examination: applies to cases where there is a taboo in the examination of the stomach or where the patient is unwilling to undergo the examination of the stomach. A typical expression of an ulcer, such as a ulcer in the stomach and a septide, can be detected through an oral sulphate cavity, followed by an X-ray or film, but may be neglected for smaller ulcer or early pathologies.
Cyclops: Common methods of detection include carbon-13 or carbon-14 exhalation tests, rapid urea enzyme tests, serology tests, etc. The detection of cholesterocella helps to identify the causes and guide treatment.
Treatment
1. Drug treatment:
1. Acid inhibitors: such as Proton Pump Repressants (including Omera, Lansola, Rebelazole, etc.) and H2 Receptor Resistants (covering Simibutin, Renitatine, Famotetine, etc.) can inhibit stomach acidization, reduce the stimulation of stomach acid to the ulcer surface and promote ulcer healing.
2. Anti-ghost snail treatment: For the positive digestive ulcer patients, clinical treatment programmes typically include a proton pump inhibitor, americium acetate (e.g. potassium acetate) and a combination of two antibiotics (e.g., Amosicirin, Kracin, Metrazine, etc.). Joint treatment, usually 10 – 14 days, to eradicate the fungus and reduce ulcer recurrence.
3. Stomach mucous membrane protection agents: medicines such as sulfur aluminium and magnesium aluminium carbonate, capable of forming protective barriers on the stomach mucous surface and of effectively preventing the effects of stomach acid and gastroprotease on mucous membrane tissue. , promote membrane restoration.
2. Surgical treatment: In most cases, a digestive ulcer can fully heal through drug treatment. However, in specific clinical situations, such as uncontrollable haemorrhages, acute perforations, scarred cavities and suspected cancers in the stomach ulcer, surgical treatment programmes need to be considered. The main methods of surgical treatment include large stomach ectoptomy and locomotive neurocutters.
VI. Preventive measures
1. Dietary adjustment: Maintaining regular dietary patterns to ensure regular food consumption in order to avoid over-eating. Ingestion of spicy, greasy and irritant foods, such as pepper, coffee, tea and fried food, should be minimized in order to reduce the irritant effects on stomach mucous membranes. Ingestion of nutrient-rich foods, such as fresh vegetables, fruits, milk and eggs, such as vitamins and proteins, has a positive effect on the protection of stomach mucus.
2. Changing lifestyles: Stop drinking, smoking and alcohol can damage stomach mucus and increase the incidence of ulcer. Take care of the rest, avoid overwork and stress, keep the mood open, and ease stress through appropriate sports, music, tourism, etc.
3. Reasonable use: Drugs that may damage the gastric mucous membrane, such as non-polymers, need to be used for a long period of time, as far as possible, to select drugs that are less harmful to the gastric mucous membrane and, under the direction of a doctor, to be administered simultaneously with gastric mucous membrane protection to prevent ulcers.
4. Prevention of cholesterol infection: attention to dietary hygiene and promotion of the use of public chopsticks and spoons to avoid cross-infection. Try not to eat raw, cold foods, such as uncleaned vegetables and fruits, raw water, etc., to prevent the passing through the mouth of the fungus.
