Restorative obstructive anaemia (Aplastic Anemia, known as AA) is a blood system disease caused by the failure of bone marrow blood function. It usually manifests a decrease in the whole blood cell, including red cells, white cells and blood panels. The following are the blood pictograms of regenerative obstructive anaemia, including a detailed description of the exterior blood and bone marrow:Exterior blood-like characteristics1. Decline in whole blood cellsThe most significant feature of regenerative obstructive anaemia is the reduction of red cells, white cells and slabs in external blood.The decrease in erythrocyte: In the form of sexual anemia, the patient has symptoms of paleness, lack of strength and heart palsy.Decrease in white cells: This results in reduced immunity and vulnerability to infection, such as fever, cough and oscillitis.Declining slabs: Increased haemorrhage, such as bruises on skin, bruises, nose bleeding, tooth bleeding, etc.2. Decline in weavered cellsThe reduction in the number of mesh-weaved red cells, which are premature, reflects a decline in the blood function of the bone marrow.Decline in bone marrow blood cellsThe ratio of lymphocytes and lymphocytes may be reversed in external blood smears, with a relative increase in lymphocytes.4. Increased non-blood cellsAs a result of the loss of bone marrow blood function, some non-blood cells, such as plasma cells, tissue cells, etc., may appear in outside blood.The bone marrow character.1. Low bone marrow growthThe examination of bone marrow piercing revealed a marked low bone marrow growth, increased fatification and a lower ratio of blood-forming to fat cells.Reduction in blood cellRed cell systems: Young red cells are rare and mature red cells are normal.Particle cell systems: Moderate and acidist particle cells are reduced, with fewer late-time plume and pole-like nuclear particles.Mega-nucleus systems: mega-nucleus cells are significantly reduced and sometimes disappear altogether.3. Relative increase in non-blood cellsThey include plasma cells, tissue cells, fibre cells, etc., which are relatively small in normal bone marrow.4. OstemosisThe degree of defamination of the bone marrow can be further confirmed by a bone marrow biopsy, usually manifested in the replacement of blood tissue by fat tissue.Cell genetics and molecular biology characteristicsGenetic anomaliesSome, but not all, patients with regenerative obstructive anaemia may have chromosome anomalies.Molecular biology markersCertain genetic mutations or abnormalities may be associated with the occurrence of regenerative obstructive anaemia.Combining clinical performance with laboratory testsClinical manifestations of regenerative obstructive anaemia, combined with blood and bone marrow tests, can facilitate diagnosis.Symptoms of anaemiaPatients suffer from various levels of anaemia as a result of the reduction of red cells.2. Risk of infectionDue to the reduction of white cells, patients are vulnerable to infection and are not easily controlled.3. Hemorrhage tendenciesDeclining slabs lead to haemorrhage symptoms, with the possibility of internal bleeding in serious cases.Diagnostic criteriaDiagnosis of regenerative disorders is usually based on the following criteria:All blood cells are decreasing, and weeded red cells are decreasing.The bone marrow is low and at least one part is extremely low (< normal 25 per cent) or low (normal 25-50 per cent).The bone marrow biopsy showed fatification.Elimination of other diseases that cause a reduction in the whole blood cell, such as systolic haemoglobin urine, bone marrow amplification abnormalities, etc.ConclusionsThe blood and bone marrow characteristics of regenerative obstructive anaemia are manifested in the reduction of whole blood cells and the failure of bone marrow blood function. These characteristics are important for the diagnosis and treatment of diseases. Detailed haematological and bone marrow tests should be carried out in a timely manner in cases of suspected regenerative obstructive anaemia in order to be diagnosed and treated as soon as possible. Treatment usually includes immunosuppressive treatment, blood growth factor support, bone marrow transplants, etc. The patient ' s prognosis is related to factors such as the severity of the disease, the treatment response and complications.
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