How do people with heart failure control water?

The disproportionate “water” intake of heart failure increases the heart burden, i.e. the excess capacity of the heart, followed by clinical manifestations such as respiratory difficulties and body oedema, and is the main reason for the hospitalization of heart failure patients. It is therefore hoped that reasonable control of “water” for heart failure patients will benefit the patients better.First, to accurately assess the state of capacity1. Initial determination of capacity based on patient ‘ s symptoms and signsSymptoms of pulmonary siltosis due to incomplete left-heart overloads of heart disease include respiratory difficulties in labour (sensitivity: 66 per cent, special 53 per cent), chronic respiratory difficulties or post-crazy cough at night, static breathing difficulties or end breathing (sensitivity: 66 per cent, special 47 per cent); signs of circulatory haemorrhage such as oedema, abdominal swelling, and necrosis, which are not due to full right-heart function.If “water” for heart failure can be assessed at a qualified hospital by the following tests; CVP: the normal range is 5-12 cm H2O, the examination is vulnerable to multiple factors such as left heart function, heart rate, heart adaptability, pulmonary intravenous pressure; CVP trends need to be observed dynamically and cannot be determined on the basis of a single measurement.Drifting tube examination: Pneumocular wedge pressure (PCWP), pulmonary artery pressure, heart output, central intravenous pressure, etc., low blood pressure, viable float tube examination in the event of difficulties in determining the state of cardiac capacity. Pulse indicator continuous heart output monitoring: Indicators that reflect the “water” load and pulmonary oedema of the heart can be measured with a higher sensitivity than the pressure indicator.II. Choosing appropriate treatment. 1. Self-health management for people suffering from heart failureThe increase in body weight and the reduction in urine suggest “water” larvae; persons with heart failure are required to exercise strict control over “water” intake, patients with chronic heart failure D can control liquid intake at 1.5 – 2 L/day, and acute heart failure can be controlled more closely; patients with heart failure are subjected to self-weight and urine monitoring if they are found to have sustained body weight increase (e.g., 2 Kg increase of 3 days) to indicate that the heart “water” is overloaded, and people with heart failure can be self-monitored if abnormal weight increases are detected and urine reductions are needed and medically available. Declining blood pressure, accelerating heart rate, or possibly as a result of increased heart failure due to “water” overload, can be measured by a stand-by test, with the patient lying down for two minutes to measure the blood pressure and heart rate, waiting for the patient to stand for one minute to measure the vertical blood pressure and heart rate, and if the constriction pressure falls significantly (>20mmHg) or the heart rate increases significantly (>30bpm), to suggest that the heart “water” load is inadequate. The rational use of urea and assistive urea to treat urea is one of the only drugs that can adequately control “water” retention in patients with cardiac failure. It is one of the cornerstones of the treatment of acute, chronic heart failure, with multiple oral small doses of urea sufficient for long-term maintenance; acute urea or acute increase in chronic urea requires adjustment of the urea dosage or delivery method. The urea currently used in clinical use is lyrea, e.g. urea, e.g. potassium urea, vascular pressurizer V2 receptor stressant. Different types of urea function mechanisms and urea strength are different, and the use of urea by patients with dystrophy is subject to the guidance of a specialist physician. Small to medium doses of dopamine and angiogens can reduce “water” loads by increasing renal blood flow, spreading blood vessels, etc.The rational use of water “overloading” aids for the treatment of blood overfiltration is attracted by the overfiltration piping negative pressure. Small molecular substances such as water filtering from both sides of the diaphragm are used to achieve drainage efficiency, and when ultrafiltration dehydration treatment is initiated when the “water” of a heart failure patient is overloading is subject to the judgement of a professional physician.In summary, “water” management for people suffering from heart failure is a complex and dynamic process in which “water” is accurately assessed in real time, drugs are used rationally, equipment aids and self- “water” management for people with heart failure can be better managed.