Common causes, clinical performance and common induction factors for obese obstructive myocardiosis

Fatty obstructive cardiomopathy, HOCM is a primary myocardial disease characterized by myocardial hemorrhoids, which can lead to an outflow of the left heart chamber.I. CausesGenetic factorsHOCM is a common chromosome genetic disease, most of which has family genetic history. A number of pathogenic genes have been identified, the main coding of which are myocardial myocardial proteins, such as the β-sculinary cylindrical weight chain, the myocalcium protein T. When these genes mutate, they cause abnormalities in myocardial myocardial structure and function, leading to cyst fattening and organization disorders, which eventually lead to cystular fattening and left ventricular displacement.2. Other possible factorsWhile genetics is the main factor, there are other possible factors involved in the development of the disease. For example, neuroendocrine factors may play a role in the process of myocardiology. Over-stimulation of neurotransmitters, such as chlorophenolamine, may contribute to the obesity of myocardial cells. In addition, long-term high-intensity movements may contribute to myocardial obesity, but the relationship between this weight and HOCM is unclear.II. Clinical performance1. Respiratory difficultiesThis is one of the most common symptoms. As a result of an outlet of the left heart chamber, there was a decrease in blood from the left heart chamber, which resulted in cycling of blood. Patients experience breathing stress when they are active or emotional, and in serious cases they have difficulty breathing in a state of rest. The degree of respiratory difficulty can be measured from the end of a mild short-to-heavy air (the patient is forced to take the end seat or half-bed to alleviate the breathing difficulty).Breast painschest pains are usually of a labour force, similar to graft. This is due to the increase in myocardial oxygen requirements due to the thickness of myocardial muscles, while the blood supply of the coronary artery does not meet the demand and causes myocardial hemorrhage. The nature of chest pain may be squeezing, irritating or stinging. The pain is mostly in the front of the heart and can be emitted to the left shoulder, the inner arm of the left arm or the neck.3. Stifler and premonal fainterStunning is a more serious symptom of the HOCM. When patients suddenly stand up, move hard, or get emotional, they become fainter because of a sharp reduction in heart output due to increased outlet barriers to the left heart chamber. Premonition dizziness refers to symptoms such as dizziness, blackness, etc. of the patient before dizziness.Cardiac disordersHOCM patients are prone to cardiac disorders, such as indoor precipices and hypercardial hyperactivity. This is due to myocardial cell organization and myocardial fibrosis, which alters the electrophysiological properties of myocardial muscles and tends to form a circuit and cause heart disorders. Cardiac disorders not only exacerbate the heart attack symptoms of patients, but also increase the risk of sudden death.Based on the risk of sudden death, the common trigger is known, avoided and the incidence of serious complications, including sudden death, reduced.Fatal obstructive myocardiasis (HOCM) is commonly induced by a number of factors, as follows:I. Physiological factorsPhysical activity(a) Extremism: Extremism is one of the major triggers of the HOCM outbreak. High-intensity competitive sports, such as sprinting, basketball and football, can dramatically increase the body ‘ s demand for oxygen. This can lead to an acceleration of the heart rate, increased myocardial contraction and increased resistance to left ventricular outlet.Scrambling: sudden and hard moves, such as heavy liftings and heavy defecation, can lead to similar situations.Bit changeA sudden change in the body position may result in a lack of sufficient blood supply to the brain for a short period of time when the person is on his or her feet or when he or she suddenly rises from his or her position. In the case of HOCM patients, such changes in blood flow mechanics can also cause cardiac retributive acceleration and contraction, which may induce symptoms.Heart failure.The HOCM patients themselves suffer from cardiac-cellal disorders and are prone to heart disorders. When a heart disorder occurs, such as an early indoor stroke, an excess of the room heart movement or a tremor, the rhythm of the heart is disrupted, which leads to an under-filled heart room or an uncoordinated heart contraction.II. Psychological factorsEmotional.Strong emotional fluctuations and a sense of neurological excitement can release epinephrine and de-adrenalin as a teacupine-like substance, which induces pain, respiratory difficulties or fainting symptoms.III. Environmental factorsClimate changeCold weather causes a constriction of the blood vessels and increases the resistance of the exterior vessels, leading to increased blood pressure. Similarly, in hot weather, the body is dissipated, skin veins expand, the blood capacity is relatively inadequate, and the heart needs to be pumped faster and can induce symptoms.Other factorsThe adverse effects of drugsSome of the drugs may induce a HOCM attack. For example, yellow-type drugs in the ocean may increase myocardial constriction and increase left-heart ventilation barriers.InfectionViruses or bacterial infections, especially respiratory infections and infectious endometriitis, can cause inflammatory reactions in the body. Inflammatory response increases the metabolic demand for the heart and may also affect the normal functioning of the heart, which induces the HOCM.Awareness of the common clinical manifestations of the disease has been given priority, with emphasis on avoiding common triggers and outbreaks.