Capone, which is a third-generation antibiotic type of gilloxin, acts as an antibiotic by inhibiting the synthesis of bacterial cell walls.
The importance and structure of the bacterial cell wall, which is essential for the survival and normal physiological functioning of the bacteria. It maintains the shape of bacteria, prevents bacteria from breaking up in a low-permeability environment as a result of over-absorption, and provides a protection barrier for bacteria against external adverse factors. The main component of the bacterial cell wall is aluminum, a complex large molecular polymer. In the gland positive fungus, the pelican molybdenum layer is thick, accounting for 50 – 80 per cent of the stem weight of the cell wall, and there is a high degree of interlinkage between the pelican glycol chains. And in the gerang cactus, the sugar layer is relatively thin, accounting for only 10-20% of the stem weight of the cell wall, and it is also covered by an outer membrane.
1. Integration with penicillin in proteins (PBPs) PBPs are involved in multiple links in the process of the synthesis of pelicans, including the synthesis of pelicans, the conjunctive reaction, etc. • The chemical structure of capone contains a β-nimide ring, which is a central part of its antibacterial activity. When it enters the environment surrounding bacterial cells, it is able to combine with the PBPs in the bacterial membranes on the basis of its own chemical structure properties. The combination is highly specific, as a key is precisely inserted into the corresponding lock. PBPs’ normal functioning is inhibited by the combination of hair aqualone with PBPs. Since PBPs play a critical catalytic and regulatory role in the synthesis of pelicans, key steps, such as the intersectional reaction during the process of pelicans, cannot be carried out normally when their function is inhibited. For example, in gland cactus, Despite its complex cell wall structure, the membrane is still able to penetrate the outer membranes, to the point where it is combined with the PBPs, and to interfere with the synthesis of the pelican. In the gland positive bacteria, synthetic interference with the thicker peptone layer is also significant, as it prevents normal contact of the peptide chain and prevents the cell wall from being successfully constructed. • The integrity of the cell wall has been compromised as a result of the disturbance of the cytocellular wall fusion by the acetone. In such cases, bacteria lose effective protection of cell walls. Infiltration pressure within bacteria is usually higher than in the outside environment and, without the support and protection of a complete cell wall, bacteria can swell by absorbing water. • When bacteria expand to a certain extent, their cellular membranes cannot withstand internal pressure, which eventually causes bacteria to break down and die. This is the rationale for the entropy of the acetone by inhibiting bacterial cell wall synthesis.
1. Antibacterial antibacterial activity of the gebrane • The peptone has a good antibacterial activity of various gerranes, which is closely related to the mechanism of its operation. It can penetrate the outer membrane of the gebrane fungi because its chemical structure and physical properties give it a degree of aqueous and hydrophobicity, so that it can easily enter the bacterial interior and integrate with the PBPs through the outer membranes. For example, it is effective in inhibiting cell wall synthesis for anti-bacterial purposes in common gland fungus, such as enema, creber, and copper-green bogus. • In the case of gland-positive bacteria, although their cell-wall structure is different from that of gland-negative bacteria, the same can be done with the pBPs to interfere with cell-wall synthesis. However, due to the thickness of the cytal gland-positive fungus, the role of the acetone may differ in some respects from that of the gland-positive fungus. It is mainly by preventing the cyanobacteria from conjunction, destroying the structure of the cell wall and, in turn, producing anti-bacterial effects on gland positive bacteria, such as the golden slurry.
The resistance of bacteria to resistance and the mechanism of action may create resistance to the effects of the methadone. Some bacteria can produce beta-endamide enzymes, which can hydrolyse the β-endamide rings of capone, thus depriving them of the ability to combine with PBPs and thus to act as antibacterial agents. In order to overcome this problem, in clinical applications, the acetone is sometimes used in combination with the β-Imamase inhibitor to enhance its antibacterial effects and ensure effective treatment of the infection.
