As the spring festival approaches, so does the gathering of family and friends. In these joyful occasions, the promotion of cups has become a common scene. However, after a cup of corruption, some may be drunk and even suffer from acute alcohol poisoning, some of whom may be at risk of acute brain haemorrhage or brain damage, such as Vineique. As a senior medical practitioner, I am deeply convinced of the need to focus on the important health topic of science for all, in terms of causes, morbidity mechanisms, pathological physiology and prevention, highlighting the risk of brain damage that acute drunks may face.
First, the cause of the disease, which explores the brain damage caused by acute intoxication, is due to excessive alcohol intake. As a neurotoxin, over-ingestion can have significant negative effects on the nervous system. For acute brain haemorrhage, alcohol ingestion leads to increased blood pressure and increases the risk of cerebrovascular fractures. At the same time, alcohol inhibits the performance of coagulant factors, making it difficult to stop the bleeding quickly after a broken vein, thus increasing the severity of brain haemorrhage. Venik ‘ s brain disease is closely related to the shortage of sulfur (vitamin B1). Long-term or large-scale alcohol consumption can interfere with the absorption and use of sulfur, leading to a decrease in levels of sulfur in the body. Sulphamine is an important nutrient for maintaining the normal functioning of the nervous system, and its lack can cause a range of nervous system symptoms, including the Vinek brain disease.
II. Analysis of the mechanism of morbidity
1. Mechanisms for acute brain haemorrhage The mechanisms for acute cerebral haemorrhage are multifaceted. First, when alcohol is ingestion, its metabolites can lead to an expansion of the blood vessels and increase the permeability of the vascular walls, thus reducing their resistance to pressure. When blood pressure rises sharply, cerebral vessels are prone to fractures and bleeding. Secondly, alcohol inhibits the accumulation of slabs and the release of coagulation factors, making it difficult to form an effective hemorrhage after haemorrhage, which leads to continued bleeding.
2. The mechanism for the development of the Venik cerebral disease The mechanism for the development of the Venik cerebral disease is mainly related to the energy metabolic disorder caused by the lack of sulfur. Sulphamine is the precursor of oxyphosphate, which is an important enzyme in cell metabolism. The lack of sulphate leads to insufficient synthesis of oxyphosphate, which in turn affects the activity of key enzymes such as acetone dehydrogenase, α-ketonete dihydrogenase and transgenase. The reduced activity of these enzymes can disrupt the aerobic oxidation of glucose and lead to insufficient energy supply. At the same time, the lack of sulfur can also disrupt the synthesis, release and ingestion of neurotransmitters, further affecting the normal functioning of the nervous system.
III. Pathological changes
1. Pathological changes in acute cerebral haemorrhage. After acute cerebral haemorrhage, haematoma forms and oppresses the surrounding tissue, resulting in pathological changes in sponges, degenerative deaths and edema. These changes not only directly harm brain tissue but also cause secondary damage such as increased intracranial pressure, cerebral oedema and blood circulation disorders. In addition, vascular activity in haematoma and free radicals can exacerbate brain tissue damage.
2. Pathological physiology changes in Venik ‘ s cerebral disease The pathological characteristics of Venik ‘ s cerebral disease include neuroneurological variability, necrosis and deficiency, mestrogenicity and necrosis, as well as the growth of star-shaped glue cells, less gel cells and cardiacs. These changes have led to a serious impairment of the functioning of the nervous system, which is manifested in the symptoms of cognitive disorders, eye tremors and communion disorders.
Preventive measures. With regard to the risk of brain damage due to acute intoxication, we can take the following preventive measures:
1. Limiting alcohol intake: Avoiding excessive drinking is key to preventing acute intoxication and brain damage. In places such as gatherings, drinking should be consciously controlled and cups avoided.
2. A balanced diet: Before and after drinking, appropriate supplements to nutrient-rich foods such as whole grains, skinny meat and beans, such as vitamin B1, can help to reduce the risk of sulphur shortage.
3. Periodic medical examinations: For people with alcohol habit, the detection of indicators such as blood pressure, blood resin and blood sugar should be carried out on a regular basis to detect and control potential cardiovascular disease risks in a timely manner.
4. Timely access to medical care: medical treatment should be provided immediately in the event of intoxication symptoms or suspected manifestations of brain damage.
Timely treatment is essential for emergencies such as acute brain haemorrhage. In short, we should maintain a rational drinking attitude and avoid the health risks of excessive drinking during spring holidays. At the same time, we can effectively prevent serious consequences such as brain damage caused by acute intoxication through measures such as a balanced diet, periodic medical examinations and timely medical access. Let us all enjoy a healthy, happy holiday!
