Membrane is a tumour originating from the membrane composition, which consists mainly of a cyst or a cell that separates the membrane cell. Fibrous meningitis is a common pathological subtype of meningitis, which is less likely to be re-emerging and invasive.
In 2007, WHO graded and graded menopause on the basis of re-embracing trends and inoculations, divided into 15 pathologies of level 3.
The WHO I class is a benign meningitis (Benign meningoma, BM), which includes epipelagic, fibrous, vascular, osteoplasmic, transitive, microcystal, lymphocyte-rich, genocrine and chemistry;
The nature of the WHO II class is between benign and nefarious, including atypical, transparent cell-type and vertebrae samples;
WO III is a malignant meningitis (Malignant menningoma, MM), which includes inter-transformation, cross-graft and nipple.
1, WHO I-class meningitis
About 90 per cent of the meningococcal, biochemical, microcystal and lymphocytes are rare. The incidence of meningi-oma is about 1.6 per cent.
Microcystical membrane (Microcystic menningoma) is also less common, with a incidence of about 1.6 per cent, and its pathology is characterized by the transparency of tumour cell plasma, which is separated by extracellular fluids into cysts, propos or stars.
The incidence of cytochroma (Metaplastic meningoma) is not clearly reported, and its pathological characteristics are the visible bone, cartilage or fat cell composition between tumour cells, which is non-specific, rather than the real chemistry.
Lymphoplasmacyte-rich menningoma is rare, with 1.7 per cent incidence, and chronic inflammation of the lymphocytes, with a lymphocytes-based lymphocytes associated with a growth centre structure, and tumours in the form of vertebrate tissues, but mostly among children and adolescents.
2, WHO II stage meningitis
Some 4.7 to 7.2 per cent of meningococcal cancers are of a benign and malignant nature, with a high incidence of inoculation and recurrence compared to class I meningomas, dominated by atypical meningitis (Atypical menningoma, AM).
Atypical membranes can be atypical changes in the subtypes mentioned above, but with their particular pathological characteristics, i.e., a diagnosis can be determined by equal to or more than four filamental ectoplasms per 10 high-fold view. In the absence of a high filamental fragmentation rate, at least three of the following five symptoms can be diagnosed as atypical meningitis: one cell-intensive; two small cell components with a high nucleo-slurry ratio; three nucleus that are visible and prominent; four typical structures that disappear into permafrost or fragmentary growth; and five regional or map types that die.
Clear cell metrinyoma, CCM is relatively rare and has a rate of 0.2% of meningitis. One per cent of CCM has a high degree of intrusiveness and recurrence compared to benign meningitis.
Chordoid menningoma is rare and accounts for 0.5-1 per cent of meningitis, and also has a high intrusive and re-emergence rate. Oncological pathologies are similar to vertebrate tumours and converse membrane cells.
3, WHOIII-class meningitis
It is rare, with about 1 to 3 per cent of meningitis, with a high rate of aggression and recurrence.
Intergenerative membrane (Anaplastic menningoma) can be evolved from I and II-grade membrane tumors, or from the outset can be malignant. It is characterized by a marked decomposition of tumour cells, a high degree of filamental fragmentation (twenty or more of the 10 high-view view) or a combination of both.
Rhabdoid menningoma has a transective membrane tumour cell structure, which often retains membrane areas, tumour cell density, cell nucleus, dichotometry, nucleus nuclei, nuclei, nuclei, nuclei, nuclei, nuclei, and nuclei.
Papillary menningoma is marked by tumour cells that are organized around the veins, mammograms and cysts stretching to the vascular wall, and curvatures, which appear to be floating in tissue slices, with reserved perimenary areas different from the classic membrane, in the form of cell-intensive, visible nuclear divisions and dead areas.
