Unstable heart pain is a complex and serious type of coronary heart disease in the transition between stable heart pain and acute myocardial infarction. Because of their precariousness and potential dangers, timely diagnosis and accurate understanding of heart pain are of great importance in improving patient prognosis and reducing the incidence of cardiovascular events.
The symptoms are more complex and varied and vary in severity than the symptoms of the stifling. Patients generally feel squeezing pain, stifling or constriction in their chest cavity. The pain is mostly in the back of the chest and can be directed to the left shoulder, the inner arm of the left arm, the neck, the jaw or the upper abdomen. The pain tends to be suddener and the pain is generally more severe than the stable CPR, usually lasting between 3 and 15 minutes, but may be longer.
In contrast to stable heart pains, the frequency of heart pains is significantly increased during rest periods or when there is a slight activity, and the frequency of unstable attacks, which were caused only by factors such as physical activity or emotional agitation. For example, some patients are suddenly awakened by chest pains during night sleep, or suffer unbearable chest pains during light activities such as daily walking, up and down stairs.
In addition, some patients may suffer from respiratory difficulties, sweaty sweats, vomiting, dizziness and inactivity. The emergence of these symptoms reflects the effects of insufficient heart blood supply on myocardial function.
The reason for this is mainly the instability of coronary porridge. The coronary artery is an important vessel for blood and oxygen to the heart, and when porridge sclerosis changes in the coronary membrane, lipid sedimentation and cell growth in smooth muscles, among other factors, lead to the gradual formation of specks. Among stifling patients, the specks are relatively stable, the cavity of the tube is relatively fixed, and myocardial blood is to some extent balanced.
However, fibre caps with coronary porridge sclerosis, which are subject to certain factors, such as blood pressure fluctuations, abnormally high blood resins, poor blood sugar control, smoking, overwork, emotional agitation, etc., may all break down, causing the nucleus in the plaque to emerge and then activate the cumulator system to form a blood clot. Part of the hemorrhage or coronary artery is completely blocked, causing a sharp reduction in the blood of the heart muscles and, consequently, an unstable attack on the heart ‘ s pain.
In addition, coronary artery convulsions are also an important pathology, and coronary artery convulsions may occur alone and may occur on the basis of the hardening of coronary porridge samples, resulting in temporary narrowness or closure of the coronary artery, which in turn leads to myocardial hemorrhages and CPR symptoms. Some patients can induce coronary artery convulsions, which occur under cold irritation, stress, etc.
A multi-faceted picture is needed to determine the precariousness of the arrhythmia. First, detailed medical history inquiries are very important, and doctors are aware of the characteristics of a patient ‘ s chest pain, including the extent, nature, duration, frequency, induction factors, mode of relief and associated symptoms. What often provides important leads to diagnosis is a typical history of unstable heart pain. An electrocardiogram is a common tool. The electrocardiograms may show signs of myocardial insinuation during an aorexia, e.g., a drop in ST, a reversal of T-waves or an increase in ST-sections (variate cardiac angulation). Some patients may have a EKG back to normal and may need to undergo multiple EKG examinations, including the Dynamic EKG Monitor (Holter), to capture changes in the EKG for patients with high suspicion of CPR instability. In the diagnostic process, myocardial signs are also important indicators. Common signs of myocardial damage include myocalcium protein (CTN) and myomatease (CK-MB). Although there is generally no significant increase in the number of signs of myocardial injuries, as in acute myocardial infarction, there is also a risk of a slight increase. Continuous monitoring of myocardial damage marks helps to determine progress and advance.
A coronary artery is a “gold standard” for the diagnosis of coronary heart disease, which provides an accurate basis for the next treatment decision by visualizing the coronary artery’s morphology, narrowness, plaque and the presence of a clot.
(i) Once an unstable heart analgesic is diagnosed, patients in general treatment should rest in bed to reduce the consumption of myocardial oxygen. Oxygen is also being administered to improve myocardial myocardial deficiency. The vital signs of the patient, including heart rate, blood pressure and breathing, are carefully monitored in order to detect changes in the condition in a timely manner.
(ii) Drug treatment
Anti-shelter drugs: aspirin, chlorprorey. Aspirin acts as a disincentive to condensation by inhibiting the production of sepsis A2, and its effect is to inhibit the cyclic oxidation enzymes of sepsis. And chlorpelle acts as an anti-shelter, with selective and irreversible inhibition for ADP receptors. In order to enhance anti-condensation treatment and to prevent further development of the haemorrhagic clots, the two drugs often need to be combined in the case of unstable analgesics.
2. Anticondensants: The commonly used anticondensants are heparin and low-molecular heparin, and the anticondensants are mainly heparin and low-molecular heparin. They are the means to retard the formation and development of the clots, by inhibiting the activity of the enzymes or by activating the coagulation factors. Condensation treatment plays an important role in the treatment of unstable heart pains, especially for patients at higher risk of leoparding.
Nitrate: The usual drug is nitrate glycerine, which mitigates the symptoms of heart pain. By expanding the coronary artery, the reduction of pre- and post-heart loads and the reduction of oxygen consumption of myocardial muscles is accompanied by an increase in the flow of blood from the coronary artery, so that ecstasy is rapidly reduced. Patients take nitric acid glycerine, which can be worn under the tongue at the time of the heart aching, usually within minutes and with moderate effect. For patients with frequent symptoms, intravenous drip formulations of nitrate-type drugs can also be used to sustain the continued expansion of the coronary artery.
4. Beta receptor blockers: Metolore, Pasilore et al., which slows down the heart rate, lowers blood pressure and reduces myocardial contraction, can reduce the consumption of myocardial oxygen. At the same time, beta receptor retardants are also resistant to cardiac disorders, which reduce the risk of heart disorders in patients with unstable heart pain and improve the patient ‘ s prognosis.
calcium ion channel blocker (calibonicchannel): The calcium ion channel blocker is an effective treatment option for patients who cannot withstand β-receptors or who have coronary artery convulsions. It inhibits the entry of calcium ions into myocardial and vascular smooth muscle cells, expands the coronary artery and the surrounding veins, reduces oxygen consumption of myocardial muscles and reduces the symptoms of cardiac pain. Common calcium route retardants are nitrous levelling, aminochlor, and calcium route blockers are mainly nitrous.
6. Tatin: Hetin has the effect of geling the plaster. Reducing progress in coronary porridge cholesterol, increasing the stability of the specks, reducing the incidence of cardiovascular events and reducing the incidence of coronary porridge plasters by reducing the levels of blood resin, especially low-density protein cholesterol (LDL-C). Irresistence is subject to long-term LDL-C.
(iii) Interventions should be considered when the patient whose condition is still unstable or whose coronary artery shows that the coronary artery is so narrow (70 per cent narrow) that the treatment of drugs such as coronary artery scyte expansion (PTCA) and coronary artery implants is ineffective. Interventions can directly expand the narrow coronary artery, restore the blood supply of myocardial muscles, rapidly relieve the symptoms of CPR, improve the quality of life of patients and reduce the incidence of cardiovascular events such as myocardial infarction.
(iv) CABG may be a more appropriate treatment option in a few cases, such as when coronary artery disease is severe and widespread, inappropriate for intervention, or when the patient has multiple coronary artery pathologies combining other heart diseases. CABG provides for blood from the heart muscles by taking the patient ‘ s own veins (e.g., large hidden veins, breast artery, etc.) and establishing a new vascular route between the near and the far end of the coronary artery, where the coronary artery is narrow or blocked.
First, maintaining a healthy lifestyle is essential. Rational diet, reduced intake of saturated fatty acid and cholesterol, salt and sugar, and increased ingestion of food rich in dietary fibre and unsaturated fatty acid. Vegetables, fruit, whole valley, fish, etc. There is also a need to control blood resin, blood pressure, blood sugar levels, to refrain from smoking and alcohol, to avoid overwork and stress, to maintain psychological balance, to regularize, to exercise properly, to exercise aerobics, such as walking, jogging, swimming, tao-bok, etc., and to improve CPR function and physical resistance.
Second, patients with cardiovascular risk factors such as hypertension, hypertension and diabetes should be actively treated and blood pressure, blood resin and blood sugar should be strictly controlled to the standard. Periodic medical examinations, including EKGs, cardiac ultrasound, blood resin, blood sugar etc., are conducted to detect coronary porridge sclerosis at an early stage. For those who have suffered coronary heart disease such as coronary heart disease, anti-shelter drugs, cartin-type drugs, beta receptor blockants, and in order to prevent the occurrence and progress of unstable heart pains, medical care should be taken on time, and regular consultations should be conducted and treatment programmes adjusted.
Unstable coronary coronary coronary coronary coronary porridge is a serious cardiovascular disease with different and complex symptoms, and its causes are closely linked to the instability of coronary porridge hardening. Timely and accurate diagnosis and comprehensive and effective treatment are key to improving patients ‘ pre-natal capacity. At the same time, the strengthening of preventive measures, the control of cardiovascular risk factors and the maintenance of a healthy lifestyle are important for reducing the incidence of unstable heart pain.
