In human interaction, the face is a “window” for emotional and information transmission, but the face spasm is like a haze that obscures the original light of the “window” and places many inconveniences and psychological burdens on the patient’s life. Understanding the causes, symptoms and treatments of facial convulsions is important for the recovery of a healthy face.
I. Recognition of facial spasms:
The symptoms are hidden in the “distant” distress.
Face convulsions, medically referred to as facial spasms, are common diseases of the brain nerve. At an early stage, the symptoms are mild, and they may be merely sporadic convulsions, like “jumping” in the eyelids, which many people mispercept are the result of fatigue and lack of attention. With the development of the condition, convulsions spread from the eye to the other muscles on the same side, with the possible involvement of the mouth, cheek and even the neck muscles, with a higher frequency and intensity, the patient’s inability to control his or her own face when he or she is serious, and with the disruption of everyday behaviour such as speech, food, closed eyes and so forth.
ii. Pathological causes:
Complication of the disease network
Its causes are complex and multifaceted. Angiogenesis of angiogenesis is a common cause. As age increases, the vascular sclerosis, shifting, etc., and the vascular “single-free” from the ergonomic nerve tend to oppress the neurological side, leading to a disruption in the transmission of signals between neurofibres, anomalous distribution of impulses and muscle spasms. Predominant pathologies, such as hearing neuromagnosis, meningitis, etc., also interfere with the normal function of the nerve by crowding out the face of the skull; in addition, facial neurosis, brain trauma, genetic factors, etc., also play a role in some cases of morbidity, with a variety of factors, either alone or in synergy, in the form of a single hair and a “balance” in the facial muscle.
Traditional treatment: Conservative response to mitigate “standards”
1. Drug treatment: initial line of defence. Drugs are often the first place of treatment, and commonly used drugs are anti-eclampsia drugs such as Camasipin and Ocasipin, which regulate the stability of the nerve membrane, inhibit the abnormal discharge of neurons and mitigate muscle convulsions. In some cases, the initial effects are evident, with the frequency and magnitude of convulsions, but long-term use, with side effects such as dizziness, sleeping addiction, rashes, and damage to the liver and kidney function, while the efficacy of the drug is reduced as the course of treatment progresses, and it can be repeated.
Meat toxin injection: short-acting saviour. Botoxin is a neurotoxin that, by being accurately injected into the muscle part of a spasm, can disrupt the release of acetylcholine at the head of a nervous muscle, thereby allowing the muscle to “relax” and reduce convulsions quickly. The operation is simple and quick and is an “emergency” treatment for moderate- and moderate-sized patients. However, botulinum is short-lived, with three-to-six months of efficacy sustained, repeated injections, multiple injections leading to antibodies, decreasing therapeutic effects, and complications such as low eyes, tears, stiff facial expressions, which are not long-term solutions, nor long-term calculations, but long-term and long-lasting.
IV. Surgery treatment: addressing the root causes and seeking a “treatment” breakthrough
Microvascular decompression: the relief of oppression “reborn”. This technique is called a “gold standard” for facial convulsion treatment, operating under a microscope, opening small cuts behind the patient’s ear, drilling the skull into the small part of the brain of the bridge, carefully separating the blood vessels of the oppressive neurological side, extending, fixing, removing the blood vessels from the neurological “tight spell”, restoring “freedom” to the neurological side and addressing the root causes. The procedure is mature and over 80 per cent efficient, most patients disappear from post-operative convulsions immediately or in the short term, and has good long-term effects that can significantly improve their quality of life, but the operation has the risk of a skull opening, the possibility of complications such as hearing loss, facial palsy, internal haemorrhage, and high technical requirements for doctors, hospital equipment.
2-Face Neural Combination: Adjusting the nerve “disorder”. This can be done in the case of myelium damage and fibrous muscular disorder in the face-to-face nervous system due to prolonged oppression. The physico-psychology is carefully combed, the “sorted” abnormally transmitted neurofibre is improved, and although the operation is relatively complex and the post-operative recovery is slower, it provides an additional treatment path for patients who are partially unfit for microvascular decompressive or post-operative re-emergence, helping to mitigate symptoms and reduce the risk of relapse.
V. Rehabilitation and psychological support:
Physically and psychologically.
Rehabilitation is a “back-to-back” treatment during which training in facial massages, heat dressings and facial muscles is combined to promote blood circulation, muscle strength and coordination and to enhance the effectiveness of the treatment. Psychological care is also indispensable, and people with facial convulsions are vulnerable to the vortex of low self-esteem, anxiety and depression due to facial abnormalities, and family and friends are required to provide support for understanding and, if necessary, to engage in professional psychological counselling to help them rebuild their self-confidence and combat disease with a positive mindset.
Frontline exploration:
Technology dawns. Lights hope for the future.
Today, medical research is making a breakthrough in facial spasm treatment. Neural physiology surveillance techniques are more precise in locating pathologies, assisting in the operation of the operation and reducing the risk of injury; stem cell research is also visible, using stem cells to repair the potential of damaged neurological tissues, and is expected to lead to a completely new course of treatment, which, although it is now mostly in the laboratory stage, is expected in the near future to bring more traumatic and effective treatment to patients.
Face-convulsion treatment is a “long-lasting war” from tradition to the front, with both advantages and disadvantages. After the patient has been diagnosed, he/she shall communicate fully with the doctor and weigh the scheme according to his/her condition. With the advances in medicine, the challenge is brightening, and the patient is committed to scientific treatment, physical and mental, so that he or she can spare the convulsions, so that he or she can smile and return to normal life.
Noodle spasm.
