In the area of cardiac health, a special and complex presence exists in the case of obese cardiac disease. Today, we are here to unmask its mysterious veil and explore the secrets behind the extraordinary increase in heart muscles.
Imagine our heart was originally a tectonic “power pump” with the right thickness of the myocardial muscles, which constricted and stretched in a methodical manner and kept the blood flow running around. However, there has been a change in the situation for those with thick myocardiology. Cardiac muscles began to grow so thick that they were as thin as the walls, and in some places they were built with many additional bricks, which became thick and hard.
This is not evenly distributed. Some patients are room spacing, i.e., that part of the heart room between the right and the left is particularly thickened, as is the case with a thicker “barrier” in the middle of the heart. Some of the patients are getting thicker. These thicker cardiac muscles change the normal structure and function of the heart.
What’s the sign of this disease? Many patients experience respiratory difficulties. This is due to the rigidity of the heart as a result of the thickening of the heart muscle, which has had a significant impact on the stretching function. For example, when the heart is constricted, it’s like a soft pocket, but now it’s too thick and too hard to open. As a result, the blood cannot flow back to the heart, the blood of the lungs is not well received, the blood of the lungs is silted, and people find it difficult to breathe, especially after the exercise, when it’s like a weight on the chest, and it’s hard to breathe.
Breast pain is also a common symptom. It’s like a tight heart, and it’s like a sense of squeezing, stifling or austerity. This is because the thicker heart muscles require more oxygen, but the coronary artery that gives blood to the heart muscles does not meet this demand, and the heart muscles suffer from anemia. Just as a car would have required a specific amount of petrol to travel normally, if the gas were not supplied, the car would have been in trouble, as would the heart muscle.
Heart palsy is also a common feature of fat myocardial patients. Patients can clearly feel their heart beating like they’re jumping out of their throats. This is because the increase in heart muscles has altered the electrical activity of the heart, making it prone to heart disorders, which are like a tumultuous drum beating. In addition to this, the patient may have symptoms of dizziness and inactivity. It’s because the heart can’t effectively transmit the blood to the brain and the whole body. The brain suffers from a lack of blood, people are dizzy; other parts of the body are not provided with sufficient blood and energy, they feel weak, and everyday activity becomes very stressful.
Fat myocardia is a great danger! In the long term, the blood pump function of the heart will gradually decline. The heart is like an old water pump, slowly losing its power and not getting blood where the body needs it. This can lead to heart failure, edema for patients, severe swelling of legs and stomachs, and a significant decline in activity tolerance, which can be difficult to lay down even when it is severe and the quality of life is severely compromised. Worse still, the disease is prone to cardiac disorders, the worst of which is room-to-heart hypervelocity and CPR. It’s like the circuit of the heart is completely out of control, and if this happens, the patient may suddenly faint and even risk life if it is not treated in time.
How did you get fat myocardia? Most of the reason is genetic. The disease is highly hereditary and if one of the family has a obese cardiac disease, the risk of the disease increases for other family members. Like a family spell, genetics play an important role in it. There are, of course, other less clear factors that may lead to an outbreak.
If there is a suspicion of obese cardiac disease, it must be examined in hospital. Doctors use a variety of tests, such as electrocardiograms, ultrasound motion maps, to see if the heart muscles are thicker and the heart function has changed. Once diagnosed, active treatment is required. Patients with mild symptoms can use drugs to mitigate symptoms, such as those that slow down the heart rate and reduce myocardial contraction. If the condition is severe and the drug is not working well, the procedure may have to be considered, for example, to remove a part of the thickening heart muscle so that the heart can recover some normal function. It means a lot to us to protect our heart’s health.
Fat myocardia.
