The effects of diabetes on end-of-pipe vessels are mainly reflected in micro-vascular disease, which can lead to blood circulation disorders, which in turn affect the normal functioning of end-of-pipe tissue. Microvascular pathologies common to diabetes patients include retinal, kidney and neurosis.
Diabetes harms the artery through multiple mechanisms. First, high blood sugar can cause vascular internal cortex function disorders, due to a decrease in nitrous oxide (NO) in the internal oscillation of the cell, which is an important factor in maintaining vascular hysteria. In addition, high blood sugar can lead to increased oxidation stress, resulting in excessive free radicals and damage to inner-vascular cells. These injuries further contribute to the thickening of the vascular wall, the narrow cavity of the tube, and even the formation of a clot.
Diabetes can also cause vascular structural changes, such as a reduction in the capillary diameter and a decrease in microvascular density, which can lead to a disruption of blood flow and affect blood supply from end-of-pipe tissue. This angiogeneity is not limited to large blood vessels, but also affects microvasculars, leading to blood circulation disorders, affecting the nutritional supply of endopolymers and the discharge of metabolic waste.
Diabetes is also closely related to vascular disease. Due to end-circle disorders, diabetes patients often suffer from symptoms such as body numbness, pain, loss of feeling, which may be related to neuroblood flow inhibition and neuron impairment. Diabetes neurosis can also cause muscle paralysis and affect motor capacity.
The effects of diabetes on the end-of-vascular environment are multifaceted and include not only changes in the vascular structure and function, but also obstacles to the blood cycle and damage to the neurological function. Diabetes patients therefore need to keep their blood sugar levels under strict control and undergo regular tests to reduce the risk of complications.
