Abdominal water, in short, refers to excessive accumulation of liquids within the abdominal cavity. As a rule, there is only a small amount of liquid in the human abdominal cavity, which acts as a lubricating internal organs. But when certain diseases strike, this balance is broken, and the abdominal water forms like a silent “water condition” inside the body.
The causes of abdominal water formation are diverse, with liver diseases being one of the most common factors. The liver cirrhosis patients suffered from chronic damage to liver tissue, increased fibrous tissue and damage to liver structure, resulting in high pressure of the door veins. When the pressure of the door vein rises, the static pressure of the vascular bed in the abdominal cavity increases, so that the liquid can readily seep into the abdominal cavity to form abdominal water. At the same time, hepatic cirrhosis can lead to a reduction in the capacity of the liver to synthesize proteins and lower plasma pneumatic pressure, making it easier to transfer liquids within the veins to the abdomen. In addition, the reduction of the hepatogenic effect on formaldehydesterone and anti-urea hormones, both of which have increased their levels, will further increase sodium sodium retention and contribute to the generation of abdominal water.
Cardiovascular diseases can cause abdominal water in addition to liver diseases. For example, when chronic pneumatic heart failure occurs, the heart pump function is reduced, the circulation of the veins is hampered, the circulation of blood is silted, and the vein pressure in the abdominal cavity increases, and the fluids seep to abdominal water. Kidney diseases, such as kidney syndrome, can also lead to abdominal water due to the loss of large amounts of protein from urine and lower plasma gelatinous pressure. Malignant neoplasms are also one of the major causes of abdominal water, and tumours in the abdominal cavity, such as stomach, liver and ovary cancer, can infringe on the peritoneal membrane, leading to increased peritoneal permeability, or inhibition of lymphobar tubes and obstruction of lymph flow, causing abdominal water. In addition, infectious diseases, such as tuberculosis peritoneum, and tuberculosis bacteria that are infected with the peritoneum, cause inflammation, resulting in increased peritoneal seepage and abdominal water.
Symptoms of abdominal patients vary according to the quantity of abdominal water and the rate of formation. A small amount of abdominal water may have a slight abdominal swelling which is not easily detected. As abdominal water increases, the abdominal abdomen will increase significantly, like balloons, and patients will feel their abdominal swelling, appetite, nausea, vomiting, etc. A large amount of abdominal water also oppresses the surrounding organs, causing respiratory difficulties, as it increases the abdominal muscles and limits the expansion of the lungs; it may also cause an oedema to the lower limbs, as the abdominal water further exacerbates the flow of the veins, increases the pressure of the lower limbs and leaks into the inter-organizational gap.
For the diagnosis of abdominal water, the doctor first performed a detailed medical examination, which revealed a mobile sound, an important sign of abdominal water. At the same time, there are a number of complementary means of inspection. Ultrasound is the most common and easy method of diagnosing abdominal water, which clearly shows the volume and location of abdominal water, as well as the morphological structure of abdominal inner organs such as liver, spleen and so forth, and helps doctors to determine the probable causes of abdominal water. Laboratory tests for abdominal perforation are also crucial, and the analysis of the nature of the abdominal water, such as colour, transparency, weight, protein content, cell count and classification, can further clarify the causes of abdominal water. For example, the cirrhosis of the liver is typically leaking fluid, while the abdominal water due to tuberculosis peritoneal inflammation or malignant neoplasm is mostly seepage fluid. In addition, there may be a need for blood tests, including liver function, kidney function, blood protocol, tumor markers, etc., as well as other visual examinations such as CT, MRI etc., to fully assess the patient ‘ s condition.
Treatment of the abdominal water is mainly for the cause of the disease. For patients with cirrhosis of the liver, sodium salt needs to be restricted, generally not to exceed 2 g per day, with the rational use of urinants, such as co-appliances with fursamy, to facilitate abdominal discharge. However, during the use of urinants, the electrolyte balance is closely monitored to prevent complications such as potassium haematosis. For patients with high levels of low-protein haematoma, the protein can be adequately supplemented to increase plasma glycol penetration pressure and reduce the formation of abdominal water. Cardiovascular diseases are caused mainly by active treatment of primary diseases, improvement of heart function and reduction of cyclic blood silt. In the case of malignant neoplasm, the abdominal water may be treated in a combination of surgery, chemotherapy and plaster treatment for the tumour, as well as palliative care such as abdominal puncture and abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdomen to alleviate the symptoms of the patient. Tuberculosis perimenitis requires a standard anti-tuberculosis treatment, which is long and typically 6-9 months or longer.
Despite its complexity, abdominal water is not insurmountable. The key is early detection, accurate diagnosis of the causes and targeted treatment. At the same time, patients must be careful to rest in their daily lives, avoid overwork, follow healthy lifestyles such as low-salt diets, provide complementary treatment, improve the quality of life and work together against “water problems” in this body.
Abdominal water.
