Osteoporosis is a complication that cannot be ignored in the long-term management of diabetes. Many diabetics are aware of the risk of osteoporosis and insist on daily calcium tablets in the expectation that they will be used to maintain their bones ‘ health, yet osteoporosis continues to occur in the opposite direction. Behind this phenomenon are many underlying reasons.
Diabetes itself can interfere with calcium metabolism. High blood sugar conditions are one of the main characteristics of diabetes, and this abnormal metabolism directly affects the intestine absorption of calcium. Under normal conditions, calcium is absorbed into the blood circulation system through a specific trans-shipment mechanism in the intestinal tract and is transported to bone tissue to maintain bone strength and density. In diabetes patients, however, a series of metabolic disorders caused by high blood sugar, such as insulin resistance, increased oxidation stress and intestinal and vascular pathologies, can disrupt the normal structure and functioning of intestinal mucular cells, causing damage to the absorption and transshipment routes of calcium, even if sufficient calcium tablets are replenished, and the amount of calcium actually absorbed into the body by the intestinal tract may not be sufficient to meet the need to maintain the normal mineralization of the bones.
Diabetes complications are also a significant factor in osteoporosis after calcium is replenished. Long-term high blood sugar can induce microvascular disease, which strains the whole body of organs and tissues, including bones. Microvascular damage to the skeletal tissue can significantly hinder the blood circulation, resulting in a partial ischaemic oxygen in the skeletal skeletal tissue and inadequate supply of nutrients. In this bad environment, the activity of skeletal cells is inhibited, their capacity to generate new bones is reduced, and the functioning of broken bones is relatively enhanced, accelerating the process of bone absorption and causing a gradual loss of bone mass. Even with additional calcium, in such a state of skeletal metabolic imbalance and poor blood supply, calcium cannot be successfully deposited into the bones, but may be released from the bones by changes in the microskeletal environment, further exacerbating osteoporosis.
For diabetics, the problem of osteoporosis in the face of calcium refilling requires a comprehensive response. The first task is to control the level of blood sugar in a strict manner, and to keep it as close as possible to normal, by means of reasonable dietary control, regular exercise and standard medication, in order to reduce the adverse effects of high blood sugar on calcium metabolism and bone health. At the same time, there are regular bone density checks to detect early signs of osteoporosis and timely intervention. In addition, the exercise of moderate heavy weights, such as walking, jogging, tao-poo, etc., can stimulate bone growth, increase bone strength and resilience and help to prevent and improve osteoporosis.
Osteoporosis continues to occur in the case of calcium recharge by diabetics as a result of a combination of factors. A better understanding of these factors and active and effective interventions are essential to improve the bone health and quality of life of diabetes patients.
Diabetes
