What do you know about gout?

Arrhythmia is a disease that results in increased levels of blood urea acid due to metabolic disorders, resulting in the crystallization and sedimentation of urineate in joints and surrounding tissues, which causes a range of symptoms, such as acute arthritis, chronic goutosis and kidney damage. The onset of arrhythmia is often unexpected. The most typical symptoms are the acute pain of the joints, most of which occur at night or in the morning, and are acute. First-time hair is usually tired and single joints, most common with the first toe joint, which is the big toe root that we often say. Patients may feel the pain of cutting and burning the joints, with a rapid swelling of the joints, a tight and bright skin, and an unbearable touch. This pain peaks in hours and seriously affects the ability of patients to move, even if it becomes extremely difficult to walk and stand. During acute onset, if prompt and effective treatment is not provided, the pain may last for days to weeks and then gradually alleviate. But this does not mean that a gout disappears, it is like a hidden “time bomb” and can be repeated at any time, and as the number of go-lives increases, the burden of the joints increases, with the ankles, knees, wrists, etc. being tired as well. The root cause of this is increased levels of haematosis in the body. There are two main sources of urea acid in the human body: internal, nucleic acid and other argon compounds derived from cell metabolism, which account for about 80 per cent of total urea acid in the body; and external, i.e., urea acid derived from ingestion in the food and metabolism in the body, which is about 20 per cent. When the body is over-generated or excretion is reduced, the level of blood urea is higher than its solubility in blood, and the urea acid crystal is distilled and deposited in joints, soft tissues, kidneys, etc. Over-ingestion of foods rich in larvae, such as animal internals, seafood, meat, beans, and so forth, as well as the long-term high consumption of alcohol, especially beer, and the impact of certain drugs on the excretion of urea acid can induce ache. In addition, metabolic syndromes such as obesity, hypertension, diabetes mellitus and high blood resin are closely related to the occurrence of gouts, which interact with each other and form a vicious circle. Diagnosis is based on tests of the patient ‘ s symptoms, signs and levels of urea acid. During acute dysentery, it is generally not difficult to diagnose high ureaemia based on typical joint symptoms. However, for a number of unusual cases, there may also be a need for joint ultrasound, dual energy CT, etc., which can detect urea acid crystals in the joint and provide a more precise basis for diagnosis. The treatment of gout is a long-term process aimed at controlling blood urea levels, reducing the number of strokes and preventing and treating complications. During acute dysentery, the main use is to relieve joint pain and inflammation, using drugs such as non-paralytic anti-inflammation, aqueous alcophate and sugary cortex hormones. In the case of intermittent and chronic periods, the level of urea acid needs to be reduced through lifestyle adjustments and the use of urea acids. The lifestyle adjustment includes severe restrictions on high-pretium food intake, alcohol abdication, drinking water to facilitate urinary acid excretion, and appropriate exercise to control weight. Pre-ureic acids are divided into those that inhibit the production of urine, such as beryllol, non-Busta and those that promote the excretion of urine, such as phenylbromomalon, for which the doctor selects the appropriate drug for treatment based on the patient ‘ s specific circumstances. Plumbing is not just a joint disease, but it can also cause kidney damage, such as urealine kidney disease, urine-acid urinate road stones, etc., if it is not given attention and treatment. Therefore, knowledge about gout and active prevention and treatment are essential for patients.