Proximate spinal disease is a major attack on the spinal column and can be chronically inflammable to varying degrees of stress and cosmos and the surrounding joints. In the case of symptoms, the early stages of direct spinal disease tend to be hidden. The pain in the lower waist is a common first-time symptom, which is usually increased after sitting or sleeping, but is reduced after the activity. As the condition progresses, the activity of the spine is gradually limited. Patients may find it increasingly difficult to bend, turn around, etc., and even to view the front when it is serious, with only a limited range of feet. Some patients also suffer from external arteries, such as hips, knees, ankles, which are characterized by pain and swelling of the joints, which can occur at or after the spinal symptoms. In addition to the joints, direct spinal diseases can overwhelm the eyes, causing iris, causing eye pain, fear, loss of vision, etc.; may also affect the heart, such as cardiac changes such as partial aneurystic valve closure; and can cause lung problems such as restricted aerobic disorders when the acoustics and lungs are exhausted. The causes of morbidity are complex, and genetic factors play a key role in the incidence of direct spinal disease. The main link is to human white cell antigens – B27 (HLA – B27), most of which are antigen positive, but not all HLA – B27 positives have disease, and environmental factors, infections, etc. may cause disease based on genetic background. For example, some bacterial infections in the intestinal or urinary reproductive tract may trigger the immune response of the organism, which in turn triggers the onset of a strong straight spinal disease. Diagnosis of direct spinal disease requires a combination of multiple factors. The doctor first asks for details of the patient ‘ s symptoms, including the part of the pain, its nature, duration, etc. The medical examination focused on the activity of the spinal column, and whether there was pressure on the hip. The visual examination is an important basis for showing the pathology of the joints at an early stage, which may be characterized by a blurring of the joints, and at a later stage, where the gap between the joints may be narrowed, integrated, etc. The CT inspection shows more clearly the fine structural changes in the hips, and MRI is of high value for the early detection of hysteria and soft tissue changes in the spinal column. In the area of blood testing, in addition to HLA-B27, indicators of haemorrhagic inflammation, C-reactive proteins, etc., are also examined, which can reflect the activity of the disease. The aim of the treatment of direct spinal disease is to control inflammation, mitigate or mitigate symptoms, maintain normal position and optimal functional position and prevent malformation. Drug treatment is at the core, and non-inflammatory anti-inflammation drugs are effective for the relief of pain and inflammation and are commonly used to improve the quality of life of patients. Rheumatizing drugs, such as nitrous sulfate, can slow progress to some extent. Biological agents, such as tumour-dysplasia stressants, can be more effective in cases of severe illness and poor response to routine treatment, but are relatively expensive and may have some side effects. In addition, the self-management and rehabilitation of patients is essential, and routine exercise in the spinal, chest, hip and other areas can maintain joint activity and prevent spinal malformations. At the same time, maintaining the right standing, sitting and resting positions, avoiding overwork and cold and humid environmental irritation, also helps to control the situation. In short, direct spinal disease requires long-term integrated treatment and management to improve the quality of life of patients and reduce the incidence of complications.
Straight spinalitis.
