What caused the atrophy?

The atrophy of stomach inflammation is a chronic type of stomach inflammation, mainly manifested in the reduction or disappearance of the gland within the gastric mucous membrane, which can lead to indigestion, appetite and weight loss. The following is a detailed explanation of the probable causes of atrophy, the mechanism of morbidity, clinical performance, diagnosis and treatment.Reasons for contracting stomachitisSphinx infection: Helicobacter pylori, known as H. pylori, is the most common cause of atrophy of stomachitis. The bacteria can produce toxins, damage protective layers of the gastromus, cause inflammation, and long-term infections can lead to atrophy of the gland.Self-immunization response: In some cases, the human immune system wrongly attacked its own gastric mucous cell, leading to a contraction of the mucous membrane. This is common in the case of autoimmune gastroenteritis and in the case of patients, there is a high incidence of self-antiphyte for gastromus cell.Long-term adverse eating habits: Long-term consumption of irritating foods (e.g. spicy, greasy, overheated or overcooled foods), high-salt diets, pickled foods, etc. can damage stomach mucous membranes and increase the risk of atrophy.Drug factors: The long-term use of inflammation drugs (NSAIDs), certain anti-oncological drugs, iron agents, etc., may cause damage to stomach mucous membranes and induce atrophy of stomachitis.Genetic factors: Genetic factors may play a role in the incidence of atrophy of stomachitis. The risk of individual morbidity may increase in families with a history of atrophy of stomachitis or stomach cancer.Environmental factors: Long-term exposure to harmful substances, such as heavy metals, pesticides, asbestos, may cause damage to stomach mucous membranes and induce atrophy of stomachitis.Smoking and alcohol consumption: Smoking and alcohol consumption are considered to be a risk factor for atrophy of gastroenteritis, which can damage the stomach mucous membranes through different mechanisms.Incidence mechanismIncidence mechanisms for atrophy of stomach disease are complex and involve inflammation response, oxidation stress and cell failure. Inflammatory reactions caused by cholesterococcal infections can lead to stomach mucous cell damage and death, and long-term inflammation can also cause atrophy in the stomach mucous gland. Self-immunisation responses directly attack gastric mucous membranes, causing cell damage and loss of function.Clinical performanceSymptoms of atrophy can be less visible and may include indigestion, upper abdominal pain, saturation, appetite and weight loss. Some suffer from complications such as anaemia and malnutrition.DiagnosisDiagnosis of abbreviated stomach disease is usually dependent on stomach mirror examination and the organization of pathology examinations. Stomach mirrors directly observe the conditions of the gastric mucous membranes, while tissue pathological examinations determine gland atrophy.TreatmentEradicating fungus: The eradication of fungi is the primary treatment for atrophy caused by fungus.Drug treatment: The use of gastric mucous membranes, anti-acids, gastrokinetics, etc., to alleviate symptoms and improve stomach mucous membranes.Dietary adjustment: avoid stimulating foods and adopt digestive, nutritious diets.Regular monitoring: As a result of some association between atrophy of stomachitis and stomach cancer, patients are required to perform regular gastroscope examinations to monitor changes in conditions.Surgery: In very few cases, serious complications may require surgical treatment.ConclusionsThe atrophy of stomachitis is a multifactorial chronic stomach disease with complex mechanisms and treatment requires a combination of causes, symptoms and the overall condition of the patient. Through reasonable treatment and lifestyle adjustments, symptoms can be effectively controlled and the risk of complications reduced. For high-risk groups, regular gastroscopy is an important means of preventing stomach cancer.