In the extended family of liver diseases, autoimmune Hepatitis is a hidden “killer” that threatens people’s health. Today, let us learn more about the disease and add to the intellectual power of safeguarding liver health.
I. Recognition of Hepatitis Inoculation
Self-immuno-hepatitis, known by name, is the result of a chronic hepatitis disease caused by the body’s own immune system’s wrongful use of liver tissue as an external “enemy”. Under normal circumstances, the immune system, like a loyal guardian, is able to identify and resist alien pathogens with precision and to protect every inch of the body’s “territory”, which can be broken in the self-reventing liver, with its own antibodies and immune cells continuing to “attack” the liver cells, and the liver plunged into a “civil war”.
II. Investigation of causes of morbidity
Its causes are complex and are the result of a combination of genetic, immune and environmental factors.
In the area of genetics, studies have found that there is a certain tendency to congregate and that the mutation or polymorphology of certain genes makes individuals more vulnerable to disease. For example, people carrying specific HLA gene subtypes are at relatively high risk of disease, and they are like “time bombs” in their bodies that are “activated” under certain conditions.
Immunization anomalies are central. The original harmonious immuno-resistance imbalance, T lymphocytes, B lymphocytes and other immunocytes, “Six-Friendly Unrecognize”, produced a large number of self-antigens for liver cells, such as anti-nuclei (ANA), anti-silient muscle antibodies (SMA), which are like “precision-guided weapons”, which lead to attacks on the liver cells by the immune cells, followed by inflammatory reactions and continued damage to the liver cells.
Environmental factors often act as “triggers”. Virus infections, such as hepatitis B and C, may “lost in the direction” of the organism’s immune system in the course of the fight against the virus, and instead attack the liver’s own tissue; the long-term use of certain drugs, such as methyldobars and furan duel, and drug-induced immune responses may also cause risk to the liver; and even long-term exposure to chemical contamination can increase the incidence of disease.
III. Clinical performances
Symptoms of self-exempting livers are diverse, with early periods likely to be characterized by minor inefficiency and fatigue, often mistakenly perceived by patients as a result of labour and easily neglected. As the disease progresses, there are signs of digestive systems, such as abated appetite, nausea and vomiting, due to impaired liver function, which affects the genomic and food metabolism of digestive fluids. Yellow slurry is also one of the most common symptoms, with patients having yellow skin and gills, and urine colours deepening like thick tea, as a result of hepatic cell damage, cholesterol metabolic disorders, a large back flow of cholesterol into the blood and excretion of urine. In addition, free livers are often accompanied by out-of-past appearances, which can be described as “the worst of all”. Artificial pain, swelling and similar rheumatism, due to inflammation caused by the sedimentation of immune complexes in the joints; red spots, rashes and irritation in the skin; and combinations of thyroid diseases, such as hyperthyroidism or decomposition, which stem from a disordered immune system “spills” throughout the body.
IV. The path to diagnosis: precise identification
Diagnosis of the liver is not easy and requires a comprehensive and multifaceted examination. Serophylological examinations are essential, and doctors focus on various self-antibody indicators, such as the ANA, SMA mentioned above, and the anti-hepatic renal granular antibodies (LKM), whose positive combinations provide key indicators for the stylization of diseases. At the same time, abnormal increases in liver function indicators, such as e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e., e., e., e., e.g., e., e.g., e.g., e., e., e., e.g., e., e., e., e.g., e.g., e.g., e., e., e., e., e., e., e.,
Hepatitis biopsy is the “gold standard” for diagnosis. Inflammation, bad death, fibrosis and lymphocyte immersion are observed under the microscope, which provides a strong basis for the development of follow-up treatment programmes by obtaining a sample of liver tissues through needle puncture, visual and precise diagnosis of the condition.
V. Therapeutic programme: fighting the disease
The primary means of treating the liver is drug treatment, with immunosuppressants bearing the brunt. Sugar cortex hormones, such as Pennithon, can rapidly inhibit immune response and reduce hepatocellular damage, but long-term use of side effects can lead to osteoporosis, hypertension, diabetes, etc. At this time, immunosuppressants such as combined sulfur can both enhance the efficacy of treatment and reduce hormone use and the risk of side effects. Bear deoxychoric acid can play a unique role in the case of patients with cholesterol symptoms, which promotes excretion, protects liver cells and improves liver function.
In cases of extreme severity and ineffective drug treatment, liver transplants become the last “life-saving straw”. By transplanting a healthy liver, replacing the damaged liver, the patient is reborn. However, the long-term use of anti-tetanus drugs after surgery is a challenge, such as infection and detoxification.
VI. Prevention and day-to-day management points
Prevention begins at the point of life. Maintain good hygiene practices to prevent viral infections, such as timely vaccination against hepatitis B; be cautious about using drugs to avoid substance abuse, especially those known to cause liver damage, and be unwell and timely in the process; and stay away from the environment and reduce chemical exposure.
Self-management is equally critical for patients. Eating high proteins, high vitamins, low sugar, low fats, eating more fresh fruit and vegetables, skinny meat, fish, etc., provides adequate nutrition for liver restoration; moderate exercise, such as walking, Tai Chi boxing, promotes metabolic, but avoids overwork; smoking is restricted and the damage to the liver is aggravated by tobacco and tobacco; regular rest is provided to ensure adequate sleep and the liver is fully rested at night; at the same time, optimism is maintained, treatment is actively cooperated, periodic review is undertaken and the disease is fought with doctors.
In short, as complex as it may be, our own immune hepatitis can be best managed to embrace a healthy life by raising awareness, making early detection, early diagnosis, early treatment and caring for the liver in our daily lives. Together, let us build strong lines of defence for the health of the liver, away from the liver.
Self-immunizing hepatitis
