Acute appendicitis is a common acute abdominal condition in surgery, with rapid onset and, when diagnosed, usually requires timely treatment, of which anti-infection treatment is a key component.
First, it is clear that the fungi is the basis for precision resistance. Acute appendicitis causes most of the co-infections of gland cactus with anaerobics, with common gland cactus, e.g., e.g., e.g., in the intestinal tract, with cortex and intestinal cortex, which can easily enter the ancillary tissue once the pathology of the appendix has changed, such as ischaemic blood, etc.; and the fragile bacterium in anaerobic bacteria, which thrives in an anaerobic conditions, with local tissue insufficiency of oxygen, providing a hot bed for their breeding.
In the choice of treatment drugs, a combination of broad-spectral antibiotics against anaerobics is commonly used. A wide range of applications, such as second-generation septoxin, e.g. fraffoxin, have a certain level of activity for the geran positive fungi, while increasing antibacterial efficacy for the geran cactus and effectively inhibiting common pathogens, such as the intestinal Eshic. The mechanism of action is to remove the bacteria from the protective barrier by inhibiting the synthesis of the bacterium cytowalls, and internal permeability is unbalanced, resulting in the break-up of death. When used, appropriate doses are usually given based on the severity of the condition, and intravenous drops are usually given every 8 hours to maintain stable blood concentration.
Nitromazole is the main force for anti-aerobic bacteria and is represented by mitraz. It can break or prevent the synthesis of DNA in anaerobic cells and kill bacteria. Metrazine is widely distributed in the body and can rapidly reach the appendicitis area, with a conventional dose of 0.5 g per 8 – 12 hours of intravenous drip. In order to improve the efficacy of treatment and to expand the antibacterial spectrum, many applications are combined with head enzymes, which work in concert to provide full coverage of common pathogens of acute appendixitis.
In terms of access to medication, it depends on the progress made and the treatment response. Pure appendicitis, mild signs, and anti-infection treatment is usually three to five days. At this time, the inflammation is at an early stage, the appendix tissue has not yet suffered from severe death, perforation, the drug has been able to control the infection more quickly, the abdominal pain and fever of the patient have gradually eased, and the white-cell count has returned to normal range, and a stoppage may be considered.
If it develops into a septic appendix, with a swollen appendix, and a pustically permeated surface, the treatment will have to be extended to 5-7 days. At this stage of infection, which increases the bacterium load, drugs are needed both to stem the growth of existing bacteria and to prevent the spread of the infection, and to provide adequate treatment to ensure that the inflammation is completely abated and does not recur.
Noma perforated appendicitis is the most dangerous, causing serious complications, such as perimenitis, and anti-infection treatments often last 7-10 days or more. Large quantities of bacteria and toxins enter the abdominal cavity, and in addition to a strong antibiotic combination of intravenous applications, there may be a need to adapt the drug programme to the bacterium culture of abdominal fluids and to the results of the acoustic sensibilities, so as to accurately combat the drug-resistant bacteria, while strengthening the whole-body support for treatment and helping patients to survive.
The efficacy of treatment needs to be closely monitored throughout the treatment process. Observe changes in the patient ‘ s body temperature, and reflect on whether the drug covers the fungus, the dosage is adequate and the antibiotics are replaced if necessary, if the substance ‘ s temperature continues to remain high after 2 – 3 days, or if the temperature decreases and then rises again. Check for abdominal signs, reduction of abdominal pain, reduction of abdominal abdominal stress, in conjunction with laboratory tests, dynamic monitoring of indicators of albino count, meso-particle-cell ratio and C-reaction inflammation, combined assessment of anti-infection effects, optimization of treatment strategies in a timely manner, and guarantee of successful recovery.
