Antibiotic treatment for bacterial liver sepsis
In the abdominal sepsis spectrum, bacterial liver sepsis is one of the more difficult infectious diseases that seriously threaten the health of patients. The liver, as the largest physical body of the human body, carries a number of critical functions, such as detoxification, synthesis and metabolism, such as a bacterium attack, which, if it is not treated in time and disposed of properly, can cause dangerous complications such as sepsis and infectious shock, which endanger life. Antibiotic treatment, as a cornerstone of the bacterial liver sepsis treatment system, goes through the whole course of treatment and its rational application is of great importance for improving the prognosis and reducing the rate of death.
The mechanism and pathological characteristics of bacterial liver sepsis
Normal liver is more resistant to bacterial intrusion by having an inherent immune line of Kupffer cell composition, a full liver haemoglobin barrier and a bacterium resistance to bacterium. However, when the organism suffers from “break-proof” factors, such as cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, cholesterol, diarrhea, diarrhea, diarrhea, diarrhea, diarrhea, diarrhea, diarrhea.
Its pathogen composition is diverse and varies according to geographical location, underlying disease of the patient and route of infection. In community access cases, intestinal glycosis, such as intestinal fungi and creberoccus, is most common in intestinal glycosis, which takes advantage of the larvae or cystal “invasion” of the liver, which induces inflammation of the liver by its own virulent factors (e.g. internal toxin, fungi) and incendiary response, while in hospitals it increases the difficulty of anti-infection treatment in the group of sexually bacterial hepatitis, in particular low immune capacity, long-term hospitalization and inoculation treatment patients, with the exception of intestinal fungi, gland positive fungus such as golden fungus, skin fungi and intestinal fungus, as well as imprecipitous fungus such as copper, Breman inacteria, which cover the production of ultra- and ultra-extremamamine (ESBLs), metallic ethylamamine (MBLs), etc.
Basic principles of antibiotics treatment
Empirical drugs: In view of the acuteness and rapid progress of bacterium hemorrhagic hemorrhagic sepsis, the onset of experiential antibiotics at the earliest possible stage, and the dose of the first dose, to ensure that the drugs are rapidly effective inactivated or antibacterial concentrations, “pressure” the outbreak of the pathogenesis, such as three generations of enzyme enzyme (head sorbing, head thorium) and co-inflation (mionitrogenes) for common disease in the community. Physico-oriented precision drugs: blood-treatment, sepsis development and drug sensitivity test results are produced (usually 3 – 5 days) and the use programme is adjusted in a timely manner by switching “targeted” to sensitive antibiotics, abandoning the “widespread net” model of the empirical drug phase and entering a precision “right track”. If the pneumocococcal is sensitive to carbon acryacne (US, Utrapenan) and is replaced immediately, it is consistent with the concept of “individualized” precision treatment to improve antibacterial efficacy and reduce the pathology. Footsteps ensure a complete cure: antibiotic control is the key to success or failure of treatment, short- and easily re-emergence of infection, and excessive length increases the liver and kidney burden and trigger resistance. In general, treatment needs to be consolidated after the patient ‘ s temperature is normal, signs are significantly abated, laboratory inflammation indicators are regenerated and visual evidence of a reduction in swollen and liquidization is available, with a total of 4 – 8 weeks of treatment to ensure that bacteria are hidden in the liver and that the inflammation is fully dispersed and the liver tissue is repaired.
Types and characteristics of antibiotics commonly used
1. Beta – intraamide: Includes penicillin, capisculin, carbon methacrylate, etc., and antibacterial activity by inhibiting the synthesis of bacterial cellular walls. The algebrain changes, the antibacterial spectroscopy increases in size, the stability of the beta-neamide enzymes, the strength of the three generations of cystals against the gerranes, the four generations of cystals (heads) against the geran positive, the vaginal, and the anti-colate cystasy; the carbon pyroacne as an “acete”, the antibacterial spectrosity, the extreme antibacterial activity, and the severity of the multiple antibacterial strains produced by ESBLs, MBLs, are serious diseases, antibacterial infections of “savid stars” but their adverse effects such as the induction of hard-to-symosis diarrhoea cannot be ignored. Nitromoxazole: Metrazine, nitrazine-focused anaerobic bacteria, destruction of bacterial DNA spiral structures by retrogents, “one-sided” in the prevention of anaerobic tract infections such as the abdominal cavity, pelvis and liver, often in coordination with other anti-aerobic drugs, forming “unilateral forces” to counter mixed infections, although side effects such as gastrointestinal discomfort and oral metal taste are more common during drug use. 3. quinone type: Left-oxen fluoride, Moxisa Star has inhibitive effects on the gland cactus and part of the gland-positive fungi, trigen, chlamydia, by inhibiting the effects of bacteria DNA rotor enzyme (expulsive isomerase II) and diaphragm IV, with good use of oral organisms and high tissue penetration, and with infection in deep tissues, such as hepatic anesthesia, but is limited to mild and moderate infections and is prohibited for use in children, pregnant women, ostrich bones and arthropodic development.
Joint drug strategy
Given that bacterial liver sepsis is often a mixed infection, the joint use of the drug is a “circular” to increase the cure rate. Classic combinations such as three generations of cystactin, the former “capturing” eclastic aerobics, such as intestinal enzymes, and the latter “cleaning up” aerobics, such as eclactactus, with a two-pronged approach; and, for patients with drug-resistant risk or severe diseases, carbon methacin (for gland-positive resistant fungus, such as methoxysilincin golden fungus), with a full “preserve” to deal with complex mutagenic pathogens, but when combined, it is necessary to weigh the interaction of drugs (e.g., drug efficacy enhancement, stress or drug by-effect superheating) and to carefully adjust doses and monitor adverse effects.
Monitoring and adjustment in treatment
Indicators such as body temperature curves, blood patterns, liver and kidney functions, and condensation should be closely monitored throughout the treatment process, and ultrasounds should be periodically reviewed and CTs should follow up on sepsis changes. In the case of persistent temperature and inflammation indicators, there is a need to reflect on the effectiveness of antibiotics, to screen for resistance, to screen for unreachable drugs (e.g. sept sepsis, pustosis infestation limits), to re-advertise second-infection, to adjust, as appropriate, the variety of drugs, the type of agents (modified to IVS injections), the dose and the frequency of delivery, taking into account the overall state of the patient, to help with a combination of nutritional support, and to correct hydrolytic disorders, so as to facilitate the “infection” of the patient and to restore normal functioning of the liver.
Concluding remarks
The treatment of bacterial hepatic sepsis is a “persistent battle” and “precision battle” with pathogens, requiring a dynamic optimization programme, closely monitored, based on the mechanism of morbidity, evidence based on the pathogen, strict application of the principle of medicine, flexible deployment of soldiers (choices and combinations). With the advances in microbiological diagnostics and the development of new antibiotics, it is hoped that in the future more efficient and less toxic treatments for these dangerous diseases will be brought to light and the health of the patients will be restored.
Bacteria hepatic swollen.
