In the context of cerebrovascular disease, brain infarction is like a pair of “brothers” with short-term ischaemic haemorrhages, all of which are closely related to the abnormal supply of blood in the brain, but are fundamentally different. A clear understanding of the differences between them is crucial for the timely detection of the situation, the right treatment and the prevention of serious consequences.
I. EMERGENCY MECHANISMS: POSSIBLE SPECIFIC ISSUES
The mechanism of brain infarction is mainly due to the sclerosis of porridge in the artery of the brain, the thickening and hardening of the vascular wall and the narrowness or even total clogged of the cavity. Just as the river is severely blocked by silts and impurities, and the flow of water is not smooth, the slabs, fibre proteins, etc. in the blood accumulate to form a clot in a narrow or clogged area, leading to the death of local brain tissues due to haemorrhage and aerobics. This is a relatively slow but continuous process, with permanent damage to the functioning of the brain if it dies.
Short-term ischaemic haemorrhage is the result of a short-term convulsion in the brain ‘ s veins or a short-term constriction of the veins by a small slug, which reduces the blood supply of local brain tissues in an instant. However, this situation is usually reversible, with an vascular convulsion likely to ease itself, or a micro-breeding may be rapidly dissolved by the body’s natural embolism mechanism, and the blood supply of brain tissues quickly returning to normal, like traffic congestion, which is only a short “red light” and then the road is back to work, with no substantial brain failure.
II. Symptoms Performance: Different length and degree
Symptoms of brain infarction tend to be more severe and longer. Patients may suddenly suffer from the inability or paralysis of one side of their limbs to walk normally, lift hands or grab objects; facial muscles may also suffer from paralysis, which leads to slanting mouths and running water; speech and understanding can be significantly affected, as they speak vaguely and cannot understand others; and may be accompanied by dizziness, headache, nausea, vomiting, blurred vision and even blindness. When these symptoms occur, they do not usually disappear on their own in a short period of time, but rather persist and gradually increase, seriously affecting the daily lives and self-care capacities of the patients and requiring urgent medical attention and systematic treatment.
Symptoms of short-term ischaemic haemorrhage are similar to brain infarction, but are of a relatively short duration and generally fully abated within minutes to hours, up to a maximum of 24 hours. For example, patients may suddenly feel a brief numbness or weakness on one side, which may return to normal only after a few minutes; or there may be a brief lack of clarity, but it can be expressed quickly and clearly; and the vision may be blurred in an instant, but it can then recover clarity. Because the symptoms disappear faster, many patients tend to ignore the situation, arguing that it is merely physical discomfort and that it is not timely, but it is a dangerous signal that there is a potential problem with the cerebral veins that could cause more severe brain infarction in the future.
III. IMAGES CHECKING: FINDING OUT THE INTEREST
There are also different manifestations of brain infarction and short-lived ischaemic haemorrhage in video-testing. For people with brain infarction, the skull C.T. may not be visible at an early stage of the disease, but with time, usually 24 hours after the outbreak, low-density strangulation stoves can be seen on CT images, which are visual features of brain tissue. The MRI is more sensitive, and abnormal signs of infarction can be detected within hours of the onset of the disease, facilitating early diagnosis.
In cases of short-term ischaemic haemorrhage, there are often no visible brain lesions during CT or MRI tests. This is due to the fact that short-lived ischaemic haemorrhages do not result in substantial brain failure, but only in short-lived blood supplies, making it difficult for images to capture anomalies. However, a number of special tests, such as cerebrovascular imaging, cranial doppler ultrasound, provide an important basis for diagnosis by detecting the presence of constrictive, convulsive or micro-embolisms in the brain.
Treatment and planning: coping strategies and the way forward
The treatment of brain infarction emphasizes a combination of early slurry, anti-blood panels, improved brain circulation, protection of brain tissue and rehabilitation. Patients who have developed within 4.5 – 6 hours of the onset of the disease can be treated with a solution if they meet the conditions of solubility. This is like the use of routing tools in blocked river lanes to clean up the haemorrhage in a timely manner and restore the blood supply of brain tissues, with the potential to save the brain tissue that is dying. At the same time, patients need to take long-term anti-sphygmophyllic drugs (e.g., aspirin, chlorprorey) and fatty-deductive drugs to prevent further brain infarction, and actively engage in rehabilitation training to restore, to the extent possible, impaired body function, language skills, etc. However, as brain tissues have already died, even after active treatment, there is still a significant proportion of patients with different levels of disability, such as physical and motor impairment, reduced cognitive function, speech impairment, etc., which seriously affects the quality of life and has a high rate of relapse into brain infarction, requiring long-term secondary preventive management.
Treatment for short-term ischaemic haemorrhages focuses mainly on preventing brain infarction. Patients need immediate medical attention to conduct a comprehensive assessment of the causes of the disease, such as the presence of risk factors such as hypertension, diabetes and high blood resin, and to actively control them. Anti-sphygmophyllic drugs (e.g. aspirin, chlorprorey) and titanoids are usually treated to stabilize inner-vascular cells and prevent the formation of haemoboles. At the same time, patients need to change their poor lifestyle, such as the cessation of alcohol and alcohol, a reasonable diet, adequate exercise and weight control. If the causes of short-term ischaemic haemorrhage can be detected and treated in a timely manner, the patient ‘ s prognosis is relatively good and it is possible to avoid brain infarction. But if this “alarm” is ignored and no active preventive measures are taken, about one third of patients may progress to brain infarction in the short term.
While both brain infarction and short-lived ischaemic haemorrhage are related to the availability of brain blood, there are clear differences in the mechanisms of morbidity, symptoms, visual examination and treatment and prognosis. Both diseases should be clearly understood, both by the patient himself and by his family, and, in the event of suspected symptoms, should be treated in a timely manner so as to enable early diagnosis and treatment, minimize the health hazards of cerebrovascular diseases and safeguard the normal functioning and vitality of the brain.
