Clinical performance and treatment of arrhythmia
I. Clinical performance
(i) Acute aroma arthritis
1. Developmental characteristics
o Sudden illness often occurs at midnight or early in the morning and the pain increases, peaking in hours. The joints are sore, they are torn, cut or bitten, and they are unbearable.
o First-time hairing and single joints are most common in the first toe joint, i.e., the joint of the big toe thumb root, followed by the joints of the back, heel, ankle, knee, etc.
2. Partial symptoms
o Red, swollen, hot, pain and functional impairments of the arteries. The soft tissue around the joints is clearly red and swollen, the skin temperature rises, the pressure is palpable, the activity is restricted and the patient cannot even bear a slight touch.
(ii) Period
1. Symptoms mitigation
o In the interim following an acute outbreak, symptoms can disappear completely and joint activity resumes. This period is of varying length, lasting several months, years and even more than a decade.
2. Potential risks
As the situation progresses, the occasional breaks will be reduced, the frequency of outbreaks will increase, and the number of arteries may increase.
(iii) Chronic gout
1. Plumstone formation
– Long-term hyperureaemia, with urea acid crystal deposits in joints, cartilage, scythes, larvae and subcutaneous tissues, which form a gourmet. It’s not the same size, small as sesame, big as eggs. 2. Artificial damage
o Painstone can cause arthropod damage, arthropod deformation and affect joint function. For example, pejorative stones at the finger joints may distort fingers and seriously affect normal movements of the hand.
3. Kidney disease
o The kidney is the main organ excreted by urea, and hyperureaemia can also exhaust the kidneys. It can cause urea-acid kidney stones, and patients can suffer from symptoms such as blood urine and kidney sting. It can also lead to chronic urinate kidney disease, with increased night urine, protein urine, oedema and other symptoms, which can develop into kidney failure in serious cases.
Treatment
(i) General treatment
1. Adapting lifestyles
o On the diet, restrictions are placed on the intake of high-eating foods, such as the internal organs of animals, seafood, meat soup, etc. Reduced consumption of alcohol, especially beer, as alcohol promotes urine production and inhibits urinary acid excretion. At the same time, the amount of drinking water has been increased and it has been recommended that 2000 – 3000 ml per day be used to facilitate the excretion of urine acid.
– Controlling weight and avoiding obesity. Obesity leads to increased urine acid production and reduced excretion. Appropriate exercise and choice of the appropriate way of moving, such as walking, swimming, etc., should be avoided, since intense exercise may induce arrhythmia.
2. Avoiding triggers
o Be careful to keep warm and avoid cooling of joints. Avoid overwork and stress, which can lead to ailments.
(ii) Acute onset treatment
1. Inflammatory drugs (NSAIDs)
o The pain and inflammation can be effectively alleviated by the example of Brophen, Piming, etc. However, such drugs may have adverse gastrointestinal effects, such as nausea, vomiting and ulcer, which require attention when used.
2. Autumn pixie base
o Traditional medication for acute pain, which is used more effectively in the early stages of ailments. However, the side effects of aqueous alcophagus, such as diarrhea, vomiting, abdominal abdominal intestinal reactions, are also more evident, and may cause serious adverse effects such as bone marrow inhibition.
3. Sugar cortex hormones
○ Sugar-coated hormones, such as piston, can be used for patients who cannot withstand NSAIDs and Autumn cactus, or who have inadequate kidney function. However, the long-term use of sugar cortex hormones may cause adverse effects such as osteoporosis and increased blood sugar.
(iii) Intermittent and chronic treatment
1. Uric acid reduction
o Inhibiting urea acid-producing drugs: e.g., peptoxol and non-bbutta. It may cause allergies, and genetic testing is best before use. Non-bubbles had a better effect but relatively high prices.
o Promote urea acid excretion drugs: e.g., phenylbromomalon, which need to be used in a manner that ensures sufficient urine, with care to avoid rock formation.
alkaline urine medicine
o It is common for sodium carbonate to keep the urine pH at 6.2 – 6.9, to promote urea acid solubility and excretion and to reduce the deposition of urea acid crystals in the kidneys.
