Antibacterial drugs can also cause multiple adverse effects while treating infection.
I. gastrointestinal reaction
– Disgusting and vomiting: This is a more common negative reaction. For example, macrocyclists (e.g. erythrin) can stimulate gastrointestinal tracts and cause nausea and vomiting. This is because such drugs increase the release of gastrokines, lead to an acceleration of gastrointestinal creeping and cause stomach discomfort.
– Diarrhoea: Many antibacterial drugs can lead to diarrhoea. Use of broad-spectral antibiotics (e.g., Amosilin) can disrupt the balance of normal intestinal strains, leading to the reduction of beneficial bacteria in the intestinal tract and the excessive growth of harmful bacteria (e.g., hard-to-work) leading to diarrhoea. If not addressed in a timely manner, such diarrhoea caused by the use of antibiotics may develop into more severe pseudofilm enteritis.
II. Allergies
– Herpes: one of the most common manifestations of antibacterial allergy. Penicillin-type drugs are prone to rashes, ranging from slight red spots, maces and severe skin skin skins. This is because the degradation product of penicillin, when combined as a semi-antigen with human protein, stimulates the immune system of the organism and produces an allergic reaction.
– Itchy: often accompanied by rashes, and possibly alone. For example, after the use of sulfamide-type drugs, some patients suffer from skin aching symptoms. This is due to the fact that metabolites of sulfamide-type drugs in the body may act as allergens, activate the immune response of the organism, stimulate the neurological endpoint of the skin, and cause itching.
– Allergy shock: this is a very serious allergy, and although the incidence is low, the consequences may be serious. Penicillin causes an allergy shock, which is more typical and usually occurs within minutes to half an hour of the drug. Patients suffer from respiratory difficulties, loss of blood pressure and loss of consciousness, which requires immediate rescue.
Hepatotoxicity
– Some antibacterial drugs can cause liver damage. Long-term or large-scale use can cause liver fat to degenerate, for example for tetracyclic antibiotics. This is because tetracycline interferes with the lipid metabolism of the liver, resulting in the accumulation of fat within the liver cell. In addition, the anti-tuberculosis drug Lifupin may cause liver damage, mainly in the form of aminoase rise, yellow sluice, etc. This is due to the fact that during liver metabolism, Lifoping produces some hepatotoxic intermediates that affect the normal function of the liver.
IV. Renal toxicity
– Amino sugar antibiotics (e.g. Quintacin) are more typically renally toxic antibacterials. Such drugs are mainly excreted in the kidneys, which can be absorbed by a small kidney cell, which may be damaged by prolonged or high-dose use, as well as protein urine, blood urine, tube urine, etc. In addition, the renal toxicity may be caused by vancocin, the mechanism of which may be related to the alteration of the permeability of the kidney tube cells, resulting in the impairment of the kidney function.
V. Neuro-system reactions
– Declining ear ringing and hearing: the ear toxicity of amino-sugar-type drugs is more pronounced. For example, the improper use of cytocin can damage the hearing cells of the inner ear, cause a sounding, loss of hearing, and cause severe or even deafness. This is due to the accumulation of amino-sugar-type drugs in lymph fluids in the inner ear, with toxic effects on the snails and front-door hair cells.
– Dizziness and dizziness: Qenone-type drugs (e.g. cyclopropsalt) may cause central nervous system reactions such as dizziness, dizziness, etc. This is because such drugs can affect the normal functioning of the central nervous system through a blood and brain barrier, such as disrupting the balance of neurotransmitters.
