Common symptoms and diagnosis of acute kidney damage

Acute renal damage is a common clinical syndrome, with a sharp decline in renal leaching from multiple causes in the short term (a few hours to days) and a drop in renal ball filtration to less than < 50 per cent, leading to the rapid accumulation of nitrogen metabolites in the body, water, electrolyte and acid balance disorders, and clinical syndromes of various manifestations of urine poisoning.Common symptoms of acute kidney damage include reduced urine, oedema, inactivity, appetite, vomiting, breathing difficulties, chest pain, heart attack, blurred consciousness or coma. These symptoms are usually associated with electrolyte disorders and acid alkali balance disorders resulting from a reduction in kidney function. In serious cases, acute kidney damage may also give rise to complications such as potassium haematosis and metabolic acid poisoning, which are complex and dangerous and may further threaten the life of the patient. Therefore, as soon as these symptoms occur, they should be referred to the hospital for timely diagnosis and treatment. The doctor conducts a series of examinations, such as blood, urine and video examinations, to determine the extent and cause of the kidney impairment, depending on the patient ' s circumstances. The treatment of acute kidney damage typically includes support treatment, medication and dialysis, which aims to restore kidney function, correct electrolyte disorders and acid alkali balance disorders and prevent complications. The acute kidney damage is usually divided into three periods based on changes in serum acetic anhydride or urine, the higher the period, the more serious it represents. Of course, acute renal damage can be classified as pre-renal, renal and post-renal causes of disease, with acute renal damage due to different causes, with treatment not identical, and severe acute renal damage usually requires dialysis.The diagnosis of acute kidney damage (Acute Kidney Injury, AKI) is mainly dependent on the increase in seroperal acetic anhydride levels and/or a decrease in urine. The diagnostic criteria usually include the following:The 1st serostatic acetic anhydride level increased 1.5-1.9 times more than the baseline, or increased thallium 0.3 mg/dL or more (mg/dL) or 26.5 μmol/L, or decreased urine to <0.5 ml/(kgčh) for 6-12 hours. This usually indicates that the kidney function is beginning to suffer. This change in serocelline acetic anhydride levels may be associated with a number of factors, including but not limited to dehydration, infection, exposure to drugs or toxins, post-operative complications, etc. Doctors therefore determine the severity and probable cause of acute kidney damage by taking into account the patient ' s clinical symptoms, medical history and possible exposure factors.The 2-stage serocelline acetic anhydride levels increased by 2.0-2.9 times above the baseline, or the amount of urine decreased to <0.5 ml/(kg§h) for 12 hours and above. This change usually means that kidney damage is already severe and requires immediate medical intervention. The doctor assesses the severity of the kidney damage in the light of the increase in this indicator and develops a treatment programme that takes into account the clinical performance of the patient and the results of the examination.The serostatic acetic anhydride levels increased by 3.0 times more than the baseline in the 3rd period or increased to 4.0 mg/dl (≥353.6umol/L) or decreased urine in the serostatic acetic anhydride, less than 0.3 ml/kg/hour for adults within 24 hours, lasting more than 24 hours or 12 hours without urine and more. This indicates that the excretion function of the kidney has been severely affected and that a significant reduction in urine is a warning sign of progress in acute kidney damage. In such cases, doctors closely monitor changes in the patient ' s urine and may take measures such as adjusting liquid intake, using urinants or renal substitution treatment to ease the kidney burden and try to restore its function. At the same time, doctors will assess whether further diagnostic tests, such as urine analysis, blood or video-testing, are required to determine the specific causes of the decrease in urine and to adjust the treatment strategy accordingly. Acute kidney failure. Chronic kidney disease.