The difference between the normal and the strong straight spinal column is mainly reflected in the anatomy structure, function and pathological changes. Understanding these differences contributes to a better understanding of the pathological mechanisms of direct spinal disease and its impact on the lives of patients.
The spinal column is not only an important structure that supports the body, but also protects the spinal cord and nerve roots and participates in the regulation of movement and position. The normal spinal cord has a good level of activity and allows for a wide range of movements, including front, back, side and spin, which are essential to daily life and sport.
Pathological changes in the vertical spinal column
Strong vertical spinal cord is a chronic inflammatory disease that mainly affects the spinal column and the hip. Histopathological changes, which include, inter alia, inflammation, bone weight formation and ultimate bone integration, have led to significant changes in the structure and functioning of the spine.
Inflammatory response: Inflammation occurs in the early stages of AS at points attached to the spinal cords such as ligaments, muscular scrotums and joints, in particular the gill joints and spinal cods. This inflammation leads to pain and rigidity, especially in cases of increased symptoms after early morning or prolonged absence.
Bones formation: As inflammation continues, the mechanism for repair of the body leads to the formation of the bony (new bones). These femurs are formed along the outer side of the spinal column, gradually connecting the adjacent vertebrae, known as the “gui specs”. This corrosive formation is one of the features of AS, leading to a decrease in spinal flexibility.
Osteoporosis: In the later stages of the disease, the small joints of the spine may be fully integrated, leading to a significant decrease in the activity of the spine. This bone-based integration makes the spinal cord rigid, and the patient ‘ s position may be fixed to a hunchback or head-on position, seriously affecting the quality of life.
The function of the normal spinal column differs from that of the AS spinal column.
As a result of these pathological changes, the spinal function of AS patients differs significantly from that of the normal spine:
Decreased activity: Normal spine activity is good, while the range of activity in the spine is significantly limited by AS patients due to bone integration and calcification of radon. Patients experience difficulties in carrying out actions such as front, back, side and rotation.
Pain and rigidity: AAS patients often feel persistent pain and morning paralysis on their backs, caused by inflammation and rigidity of joints. The normal spinal column will not experience this continuing discomfort when moving.
Changes in posture: Due to bone integration and structural changes in the spinal column, ASA patients may have a hunchback deformation and head forward position. This contrasts sharply with the natural physiology of the normal spine.
Respiratory function limited: AS may result in a limit on the expansion of the chest profile and affect the respiratory function. Under normal conditions, the chest contours can expand and shrink freely while breathing, while the rigidity of the chest profiles of AS patients makes deep breath difficult.
Differences between the normal and the strong straight spinal column are mainly reflected in structural, functional and pathological changes. AS-induced inflammation, osteoporosis and osteoporosis significantly alter the normal anatomy and function of the spine, thus affecting the quality of life of patients. Early diagnosis and integrated treatment, including medication, physiotherapy and lifestyle adjustments, can to some extent slow the progress of the disease and improve patients ‘ symptoms and quality of life. Understanding these differences is essential for clinical management and patient education and contributes to increased awareness of and resilience to diseases.
Straight spinalitis.
