Duodenal stasis, also known as duodenal stasis or duodenal stasis, is a rare disease characterized by obstruction of the passage of duodenal contents, resulting in dyspepsia, epigastric pain, vomiting and other symptoms. Here are some details about duodenal stasis:
Etiology and classification:
Duodenal stasis can be divided into two categories: primary and secondary.
1. Primary duodenal stasis: It is mostly caused by abnormal anatomical structure, such as too short suspensory ligament of duodenum, too many congenital circular folds of duodenum, abnormal position of duodenum, etc.
2. Secondary duodenal stasis: It is mostly caused by external compression or pathological changes of adjacent organs, such as tumor, inflammation, postoperative adhesion, superior mesenteric artery compression, etc.
Clinical signs:
1. Epigastric pain: It is aggravated after meals, and the pain can radiate to the back, showing persistent dull pain or drilling pain.
2. Vomiting: It often occurs after meals. The vomitus is gastric contents and contains bile.
3. Indigestion: loss of appetite, abdominal distension, belching, constipation or diarrhea.
4. Weight loss: Long-term vomiting and indigestion lead to malnutrition and weight loss.
5. Electrolyte disorder: Due to frequent vomiting, hypokalemic and hypochloremic alkalosis may occur.
Diagnostics:
1. Medical history inquiry: find out whether the patient has related symptoms, such as epigastric pain after meals, vomiting, etc.
2. Physical examination: check for abdominal tenderness, mass, etc.
3. Imaging examination: Abdominal X-ray, barium meal radiography, CT scan, magnetic resonance imaging (MRI), etc., can show duodenal dilatation and stasis.
4. Endoscopy: direct observation of duodenal mucosa through gastroscopy or duodenoscopy.
5. Angiography: For the stasis caused by superior mesenteric artery compression, angiography is feasible.
Treatment:
1. General treatment: diet adjustment, a small number of meals, avoid overeating, reduce the intake of stimulating food.
2. Medication: Use gastrointestinal motility drugs and spasmolytic drugs to relieve symptoms.
3. Surgical treatment: For severe duodenal stasis, especially secondary stasis, surgical treatment may be required. The operative methods included duodenal reduction, duodenojejunostomy, adhesiolysis and so on.
4. Interventional therapy: For stasis caused by superior mesenteric artery compression, interventional therapy is feasible, such as vascular stent implantation.
Prevention and care:
1. Dietary management: Eat digestible food, avoid overheated, too hard, spicy and other stimulating food.
2. Living habits: regular work and rest, avoid overwork, and maintain a good attitude.
3. Regular review: For patients who have been diagnosed, regular review should be carried out to monitor the changes of their condition.
4. Health education: to popularize disease knowledge and improve patients’ self-management ability.
Prognosis:
The prognosis of duodenal stasis depends on the etiology, severity of the disease and whether the treatment is timely. The prognosis of primary stasis syndrome is better if it is treated in time, while the prognosis of secondary stasis syndrome depends on the nature of the primary disease and the effect of treatment.
In a word, duodenal stasis is a disease that needs comprehensive diagnosis and treatment by various means. For suspected cases, they should seek medical treatment in time and follow medical supervision for treatment. With proper treatment and care, most patients can relieve their symptoms and return to normal life.
