The ulcer colonitis is a chronic, non-specific intestinal disease, mainly accumulatory and rectal mucous membrane and mucous membrane, which tends to persist, with significant effects on the physical and mental health of patients.
1. Diarrhoea: This is one of the most significant symptoms of ulcer colonitis, with a significant increase in the number of defecation cases, ranging from 3 to 4 times a day for the light, to more than 10 times a day for the heavy. The faeces are mostly mucous sepsis, caused by an increase in local haemorrhage and seepage due to intestinal mucousitis, decomposition and ulcer. 2. Abdominal pain: Mostly in the lower left abdomen or lower abdominal abdominal pain, which is often in the form of pain, pain or oscillation, of varying degrees of pain and is usually mitigated by defecation. This is due to intestinal creeping during defecation, which induces inflammation and gas in the intestinal tract, thereby reducing the irritation of the intestinal nerve. 3. The acuteness of the disease: patients tend to be frequent, but the amount of defecation is low at a time, and it is still not felt after defecation, as a result of rectal inflammation that irritates the rectum mucous membranes and causes continuous excretion of the excretion reflector. 4. Other symptoms: As the condition progresses, the patient may also suffer from whole-body symptoms such as appetite loss, nausea, vomiting, fever, wasting, anaemia and low-proteinemia. The emergence of these symptoms suggests that the disease has had a more serious impact on the overall health of patients, such as chronic haemorrhage, which can lead to anaemia, and intestinal ingestion disorders of nutrients, which can lead to wasting and low-proteins.
1. Genetic factors: ulcer colonitis has a certain family genetic orientation. Studies have shown that if members of the family suffer from ulcer colonitis, the risk of morbidity among other relatives increases relatively. Genetic anomalies may make individuals more sensitive to certain intestine irritation factors, which can lead to intestinal inflammation. Immunisation factor: The human immune system plays a key role in the outbreak of ulcer colonitis. Under normal conditions, the immune system is able to identify and protect against alien pathogens and protect the health of intestinal mucous membranes. However, among ulcer colonitis patients, the immune system may have abnormally activated, wrongly attacking its own intestinal mucous tissue, leading to the persistence and development of intestinal inflammation. Intestine fungus disorders may also be involved, and certain bacteria or their products may trigger immune reactions that further exacerbate intestinal inflammation. 3. Environmental factors: Eating habits, lifestyles and living conditions may be associated with ulcer colonitis. Long-term consumption of high sugar, high fat and low-fibre food can affect normal intestinal creeping and digestive functions and increase the intestinal burden, leading to disease. Smoking, excessive stress and intestinal infections were also identified as possible triggers. For example, long-term stress may affect the intestinal regulation of the neuroendocrine system, weaken the intestinal barrier function and make the intestinal tract more vulnerable to infestation and inflammation of pathogens.
1. intestinal complications: Long-term ulcer colonitis can lead to repeated damage to and repair of intestinal mucous membranes, and can easily cause a series of intestinal complications. In the case of intestinal piercing, which is due to inflammation of the intestinal wall, the thinness of the intestinal wall may result in perforation and serious peritonealitis when the pressure in the intestinal tract increases; intestinal haemorrhage, which can result in vascular haemorrhage due to intestinal mucous membrane, ulcer ulcer and blood vessels, which can pose a serious risk to life; in addition, intestinal resistance may result in the passage of intestinal contents due to narrow intestinal tracts, thickening of the intestinal wall and coagine. Long-term inflammation can also increase the risk of colon cancer, which is more likely than for the general population, especially those with a long-term history and a wide range of pathologies. 2. All-body complication: ulcer colonitis is not limited to local intestinal pathologies, but can also lead to multi-system complications. In the eye, for example, there can be iris, imitation, imitation, etc., in the eye, which can be characterized by eye pain, blurred vision; in the skin, there may be hysteres, noma, sepsis, etc., and red spots, rashes, herpes, ulcer, etc., may be caused in the skin; and in the joint, it can cause external ecstasy, high-relation spina, etc., resulting in joint pain, swelling, limited activity, etc. In addition, hepatic function may be affected, with hepatic conditions such as fatty hepatitis, choreography, etc., in the form of abnormal liver function, yellow sluice, etc.
Diagnosis: the precise search for a comprehensive diagnosis. It is possible to observe directly the pathology of the rectal and colon mucous membranes, such as mucous membranes, edema, ulcers, etc., through colon lenses. It is also possible to perform a biopsy of the mucular tissues and to obtain pathological specimens in order to ascertain the nature and extent of the disease and to determine whether there are other abnormalities such as cancer. 2. Excreta screening: the main tests are for white cells, red cells, hidden blood, etc., in the faeces, in order to understand whether inflammation, haemorrhage, etc. are present in the intestinal tract. Cure culture can exclude intestinal infections, as some bacterial or parasitic infections can also lead to symptoms similar to ulcer enteritis, and it can be clearly established whether specific pathogens exist. Blood testing: Indicators such as detection of white cell count, red cell deposition rate, C reaction protein, in blood routines, can reflect the extent of inflammation. In addition, indicators such as clean blood proteins and red hemoglobins can be checked for nutritional status and anemia. Self-antibody testing, such as anti-neutral cytocellular plasma antibodies (ANCA), is of some assistance in the diagnosis of ulcer colonitis, and some patients may have positive results. 4. X-linker enema: Although the colonoscopy is more intuitive and accurate, X-linker enema is still of use in certain cases. It can show the form, contours, creeping conditions and the presence or absence of a narrow, epiphany and other pathologies, and can contribute to the overall assessment and diagnosis of the disease.
1. Drug treatment:
Amino-water glycol acids, such as nitrous sulfate, are one of the most common drugs for ulcers. It is able to play a local anti-inflammatory role in the intestinal tract and mitigates the inflammation response of the intestinal mucous membrane, which is applicable to patients with mild and moderate ulcers and to maintenance after abating. Its mechanism of action is mainly to reduce inflammation through various means, such as inhibiting prostate synthesis and removing free radicals.
• Sugar cortex hormones: sugar cortex hormonal hormones, such as pennisons, hydrocrylic pines, etc., are frequently used during acute ulcer. Such drugs have a strong anti-inflammatory effect and can rapidly mitigate intestinal inflammation and the symptoms of patients. However, because of the high number of side effects, such as osteoporosis, high blood pressure, diabetes and increased risk of infection, long-term use is generally not appropriate and will require a gradual reduction in detoxification as the condition is mitigated.
Immunosuppressants, such as sulfur, cyclists, etc., are applied to patients who have not been effective or dependent on sugar cortex hormone treatment. Immunosuppressants control intestinal inflammation by regulating the functioning of the immune system and inhibiting the immune response. However, there are also side effects of immunosuppressants, such as bone marrow inhibition, damage to liver and kidney function, which require close monitoring during use of indicators such as blood routines, liver and kidney function.
Biological agents: In recent years, biological agents have been increasingly used in ulcer colonitis treatment. For example, influencing monostemics, it can be specific to inflammation-related cytogens or cell surface molecules, blocking the transmission of inflammatory signals and thus effectively controlling intestinal inflammation. The efficacy of biological agents is significant, but relatively high prices, and some infectious diseases, such as tuberculosis, hepatitis, etc., need to be removed before use, as they may reduce the immune defence of the body and increase the risk of infection. 2. Surgical treatment: Surgical treatment needs to be considered when there are serious complications such as haemorrhage, perforation, cancer, intestine infarction and ineffectiveness of internal treatment. The procedure includes, inter alia, whole colon removal with reintestinal oral surgery, full colon removal with reintestinal recternology, and reintestinal bag acoustic compatibility. While surgical treatment completely removes the pathological intestinal tissue, post-operative patients need to face new problems, such as reintestinal care, changes in intestinal function, etc., so that surgical decision-making requires a combination of the patient ‘ s condition, physical condition and quality of life.
1. Dietary adjustment: development of good eating habits, increased intake of dietary fibres, more fresh vegetables, fruits, whole grain food, etc., helps to maintain normal intestinal moth function and promotes defecation. Reducing consumption of high fat, high sugar, spicy irritating foods and avoiding the use of irritating drinks such as alcohol, coffee and tea, which can stimulate intestinal mucous membranes, increase the intestinal burden and increase the risk of disease.
2. Stress reduction: Excessive long-term mental stress is one of the triggers of ulcer enteritis. It is therefore important to learn to reduce stress in an effective way, such as through moderate physical exercise, such as walking, jogging, yoga, Tai Chi, which not only relieves stress, but also enhances health; to develop interests, such as painting, calligraphy, music, reading, etc., and enriches life and diverts attention; and to ease mental and physical health and to regulate emotions through meditation, deep breath, psychological counselling, etc.
3. Periodic medical check-ups: It is important for people with family history or other high-risk factors of ulcers. Medical examination projects may include colonoscopy examination, routine excreta examination, blood examination, etc., in order to detect intestinal pathologies at an early stage and to intervene in a timely manner. Early diagnosis and treatment can effectively control the development of conditions, improve the quality of life of patients and reduce the risk of complications.
Although ulcer colonitis is a more difficult chronic disease, it can be better dealt with by patients and the population at large through an in-depth understanding of its symptoms, causes, hazards, diagnostic methods, treatments and preventive measures, actively preventing, early diagnosis and regulating treatment, thus effectively controlling the condition, reducing its impact on the quality of life and accompanying it with health.
ulcer colonitis. Chronic ulcer colonitis.
