Summary: The purpose of this paper is to inform the reader about the disease of arterial hypertension, including its definition, causes, symptoms, diagnostic methods and treatment and preventive measures. Through in-depth and shallow statements, public awareness of high-tension corrosive disorders is increased, early detection and reasonable intervention are promoted, and the health risks of the disease are reduced. 1. In the introduction, the more common and serious type of hepatic circulatory disorder is pulsive corrosive disease, which causes a complex set of pathological physiological changes and clinical symptoms that pose a significant threat to the health of patients. Knowledge of the subject of arterial hypertension is of great importance, both for the prevention of diseases in the general population and for the early treatment and health management of patients. 1. Hepatic cirrhosis is the most common cause of cystalcosis. When hepatic cirrhosis occurs, hepatic tissue is fibrogenic and conjunctivated, causing damage to the normal liver structure, obstructing the flow of veins and increasing pressure. Common factors that give rise to hepatitis cirrhosis include chronic high levels of alcohol consumption, hepatitis B and C virus infections, and self-immuno-hepatitis. 2. Non-hepatic cirrhosis of the door veins, such as the formation of a door vein, can lead to the formation of an internal vein of the door, inter alia, through high blood condensation, abdominal inflammation or tumour oppression, hindering the flow of the cavity of the door veins, which in turn triggers high pressure. Congenital membrane, such as a narrow membrane, can also affect the normal flow of blood from the membrane, leading to increased pressure. 3. Burgar syndrome of obstructive hepatovascular retipation is a typical representation, where the retrenchment of the hepatic vein or lower cavity veins above its openings results in a lack of retrenchment of the blood of the liver and increases the passivity of the door vein. Symptoms 1. Spleen spleen swollen and spleen hypertensive, resulting in spleen silt and increased spleen size. Spleen inflammation can reduce white, plated and red cells, and patients are prone to infection, haemorrhage, such as frequent colds, haemorrhaging of their teeth and skin bruises. 2. The abdominal water forms abdominal internal vascular pressure which is increased and the internal vascular fluids seep into the abdominal cavity to form abdominal water. The abdominal swelling of the patient can be accompanied by abdominal swelling, abdominal abdominal pain, appetite and respiratory difficulties, which seriously affects the quality of life. 3. Increased oesophagus cirrhal pressure opens the side-cycle between the door vein system and the cavity vein system, which is one of the important side-cycles. These twisted veins are thin and prone to fractured bleeding, manifested in vomiting, black defecation, which can cause shock and endanger life. 4. Anal veins at the lower end of the cysts can also be twisted by the high pressure of the door vein, forming hemorrhoids, and the patient can suffer symptoms of defecation, anal swollenness, etc. Diagnosis 1. The medical history and medical examination details the patient ‘ s history of liver disease, alcohol abuse, viral infections, etc., and whether there are any of the above related symptoms. The medical examination focused on liver size, mass, spleen swollen, abdominal incisor and abdominal vein. Laboratory blood tests provide information on the reduction of blood cells due to spleen hyperactivity; liver examinations help to determine the extent of liver damage, such as reduction of pure blood protein, increase in chlamydia, abnormality of retinoids, etc.; and coagulation tests reveal abnormalities of condensed blood caused by reduced condensation. In addition, hepatitis virus markers need to be tested for viral hepatitis-related arteries. 3. Visually – Ultrasound: The detection of liver form, size, mass, door veins, spleen diameter and blood flow, spleen size and the presence of abdominal water, etc., is a common method of screening for high-tensiveness of the door vein. – CT and MRI: This is important for determining the cause and assessment of the disease. – Diagnosis of the stomach: it is important to observe directly the extent, extent and availability of red signs in the oesophagus, to predict the risk of haemorrhage and to guide treatment. 1. Antiviral treatment is required for the treatment of diseases resulting from the infection of hepatitis B or C; persons with alcohol cirrhosis are required to stop drinking; and their own immunosuppressants, etc., are necessary for the treatment of their own immunosuppressants to slow the cirrhosis of the liver and to fundamentally reduce the pressure on the door. The medications commonly used for the treatment of membrane cytostatics include vascular pressurizers and their analogues, growth inhibitors and their analogues, which can be used to prevent and treat oesophagus in the oesophagus by constricting the internal veins and thus reducing the flow of blood from the veins. Beta receptor retardants, such as Punirol, can reduce the flow of veins by slowing the heart rate and reducing the heart output, and long-term applications can reduce the risk of first haemorrhage and re-emorrhage rates. 3. Treatment of bleeding from dysenteral diarrhea: In case of acute haemorrhage, use of the above-mentioned medication for the reduction of door dyslexic stress, together with the application of acidics such as proton pump inhibitors, to increase pH values in the stomach and to promote stop bleeding. – Endoscopy treatment: this includes hysterectomy of the oesophagus under the inner oesophagus, injection of scortizers, etc., for the purpose of stopping and preventing haemorrhage through the interruption of hysteria or the formation of haemorrhages within the veins. – Tertiary cystals to stop the bleeding: if drugs and endoscopy treatments are ineffective, they can be used to impregnate the stomach and oesophagus by inflating the dystrophagus, but not for too long, so as to avoid complications such as mucous membranes. – Cervical intracircular sepsis (TIPS): by creating a traverse between the frontal veins and the hepatic veins in the liver, to reduce the pressure of the door veins, which applies to patients whose medications and endoscopy are ineffective, who have repeated haemorrhages or who are waiting for a liver transplant, but may have complications such as liver cerebral disease after the operation. 4. Treatment of abdominal water limits sodium salt intake and uses urinants to facilitate abdominal discharges, such as propyl amalgam sermi. In the case of a large quantity of abdominal water and a poor urea effect, abdominal puncture may be considered, but care may be taken, for example, to supplement protein to prevent circulatory disorders. Severely persistent abdominal water may be considered for TIPS or liver transplant treatment. 5. The treatment of spleen surgeries may be subject to spleen ectopsis or partial spleen aneurysm when medical conditions permit, but may increase the risk of infection after spleen ectopsis, subject to careful assessment. Prevention of hepatitis B vaccine against hepatitis B, unnecessary blood transfusions and blood products, and prevention of hepatitis B, hepatitis C virus infection. Maintain a healthy lifestyle, with a proper amount of drinking or sobering, avoiding substance abuse and reducing the risk of liver damage. 2. The management of liver patients who are already suffering from chronic hepatosis should be actively treated, with periodic reviews of liver function, abdominal ultrasound, etc., to detect and deal in a timely manner with possible cirrhosis of the liver, as well as with early pathologies of door pulsation. Control of the development of the condition in accordance with the doctor ‘ s dietary and therapeutic recommendations. VII. CONCLUSION: A disease that seriously affects the health and quality of life of the patient, with its diverse causes, complex symptoms and a combination of diagnostic and therapeutic factors. Increased public education, increased awareness of and prevention of high-tensiveness at the door, as well as early screening, diagnosis and sound treatment for high-risk groups and patients, can effectively reduce morbidity and mortality from high-tensiveness at the door and improve patient prognosis and reduce the medical burden on society and families.
Unscientific high-pressure syndrome
