Rheumatism heart disease, also known as rheumatism, is the most common heart valve disease in the country. It is part of the psychotic response caused by the early infection of group A beta streptocyteella and is a self-immunological disease.
The rheumatism activity is often tiring and the inner membrane, the most common of which is cardiac valves, which cause cardiac valves to mutate. In the form of two, three, one or more narrow and/or incomplete valves in the aorta. The clinically narrow or closed heart valves are not always present at the same time, but they are often dominated by one valve disease, and can be combined with two or three valves, most often tiring and arctic, followed by an aortic valve, which is rare and very rare in the case of pulmonary artery. Symptoms Symptoms of illness. Early symptoms of disease are generally not visible, and haematological abnormalities in patients, such as reduced activity endurance, are usually manifested in loss of CPR, irritation, weakness, cough, lower leg oedema, pink foam, etc., when the patient suffers from an incomplete right heart function, such as swelling of the lower limb, premonition that the disease is serious.
The main clinical manifestations after the disease are: 1. Respiratory difficulties. The different levels of respiratory difficulties are the initial feelings of the patient, who has a subjective feeling of air deficiency or respiratory stress, and objectively the frequency, depth and rhythm changes. Respiratory difficulties can be divided into six basic types of heart-borne respiratory difficulties, lung-source respiratory difficulties, central respiratory difficulties, psychoneurological respiratory difficulties, moderate-toxic respiratory difficulties and blood-borne respiratory difficulties, according to the pathology of the disease. Respiratory difficulties due to wind-heart diseases result in chronic or rapid pulmonary bruising due to heart valve disease, reduction in pneumatic elasticity, aerodynamic disorders, reduced heart drains, reduced blood flow rates and increased pressure from respiratory respiration, leading to increased aerobics, carbon dioxide oscillation and lung circulation. Depending on the degree of the disease, they are classified as labour-related respiratory difficulties, night-stage respiratory difficulties, end-sit breathing and heart-borne asthma. At first, it was physical or running, and at first, it was difficult to breathe when it gradually increased, and at last when it was still. chest pain can be caused by a number of causes, depending on the origin of the chest pain, which can be divided into chest pain caused by, inter alia, ecstasy, ecstasy, heart heart pain, which is referred to as heart-borne chest pain, which is mainly caused by heart attack, which reduces the blood supply from the heart blood, e.g. by a narrow or incomplete heart valve. 3. Sudden, brief loss of consciousness, due to a lack of blood supply from the brain ‘ s instant sexual extensiveness. Main causes: reduction of heart discharges (arcal valve pathologies) or cardiac stoppages (transmission retardation), sudden and severe drops in blood pressure (a large number of aortic valves backsliding) or general temporary closure of the cerebrovascular veins (stripping of the blood clots). Severe heart-borne fainting is called acute heart-borne ischaemic syndrome. 4. Cardiac convulsion is accompanied by a feeling of discomfort in the front section of the heart, often due to cardiac disorders, increased heart activity, etc., such as tremors.
Medical examination (1) Long-term chronic heart failure can be manifested in heart-borne malignadies and extreme failure of patients. (2) Severe oxygen deficiency can cause anxiety and even dementia among patients. (3) Patients with severe heart failure are often forced to half-bed or sit. Patients with cardiac enzyme or heart pressure often take a forward seat. (4) Triple cheeks are found in a narrow, so-called second-point petal face. (5) Rheumatism or rheumatism takes the form of excessive sweat; the skin is pale and sweaty when the surrounding cycle collapses; and severe heart failure can have a mild anthrax. Most of the lower knots around the joints are rheumatized. Main assistive examinations: Cardiac colour ultrasound Doppler Hazard Artificial embolisms such as brain infarction, lower limb artery embolisms, abdominal embolisms, frequent tremors of the wind-heart patients, slow bleeding in the left heart chamber to form a thrombosis, which results in sudden death or arterial embolisms.
1. Once clearly diagnosed, there shall be active surgery for the symptoms of a reduction in the heart function and for the emergence of a turban, including for the replacement of the valve and for the restoration of the valve, which shall be divided into mechanical and biological petals. The second petal repair technology has been developed in recent years to restore the second petal to a normal physiological state, to protect its own valves, to avoid the risk of a permanent anticondensation and haemorrhage and to reduce mortality. Oral drugs: Some drugs need to be taken on time at an early stage of the operation, mainly anticondensatives, strong urea, etc. The replacement of biological petals requires treatment for anticondensation in Wafalin in the second half of the operation, while the replacement of mechanical petals requires life-long treatment for anticondensation in Wafalin. After the operation, there is regular monthly out-patient review. A six-month oral valentine anticondensation treatment after the respiration.
Living habits: Be careful to maintain good habits, combine work and avoid overwork.
Prevention of infection: Appropriate exercise, physical enhancement and prevention of respiratory inflammation, such as cold, in cases of dental diarrhea, ulcers, urinary infections, etc. Medical treatment should be provided in a timely manner and doctors should be informed of their experience with heart valves and given an accurate picture of their current use.
