The digestive system, which is the “life path” for human ingestion of nutrients and discharge of waste, is constantly at risk of bacterial infestation, and antibacterials are an important “resistance weapon” in our hands once the “protection line” is broken and the infection is triggered. However, the “acting” methods of different strains vary, and only precise identification and treatment can effectively repel the infection and restore gastrointestinal health.
The gastrointestinal bacterial infection: the fungus is different, the drugs are exclusive. In common bacterial infections in the gastrointestinal tract, acute gastrointestinal inflammation is a “frequent visitor”, most of which, due to an unclean diet, “drive in” coli, salmonella, etc. Patients often suffer from abdominal pains, diarrhoea and excreta in the form of water or slime, accompanied by nausea and vomiting, with different levels of severity. For this type of infection, quinone-type drugs (e.g., left-oxen fluoride) were commonly used as “warlords”, which suppress bacteria’ DNA revolving enzymes, interfere with the synthesis of genetic material, direct to grenacella, such as coliculus, “intensity” and then rapidly absorb and work in the gastrointestinal tract to relieve abdominal abdominal pain. But in recent years, given the drug resistance problem, three generations of enzymes (like chrystals) are more appropriate for children and young people, with antigen spectroscopy covering common intestinal bacteria, safeness clinically certified to effectively control inflammation and reduce gastrointestinal symptoms. Another thorny “competent” is a hard-pressed scaffolding, often resulting in pseudo-film inflammitis following the long-term use of broad spectrum antibiotics and intestinal disequilibria, in the form of severe diarrhoea, abdominal swelling, and even heat and bloodbath. And at that point, the unicorn and the vancomycin became the “savage straw.” Americium is based on nitrocorporatium, which destroys large molecules, such as hard-to-earth DNA, oral administration to the intestinal pathology, where prices are proximate and the efficacy of the treatment is accurate; vancomicin orals are almost non-absorbed, high concentrations are formed in the intestinal tract, by inhibiting the synthesis of bacterial cell walls, fine hanging of the hard-to-exclude, preventing the deterioration of the disease, and remaking the micro-ecological balance of the intestinal tract.
Sphinx infection: “Quadruption” working together. The fungus is like a “ghost” hidden in the stomach, closely related to chronic stomach inflammation, stomach ulcer, stomach cancer. A single antibacterial drug is difficult to “leach” and clinically more often uses “tetratherapies”, i.e. a proton pump inhibitor (e.g. Omiraazole, Lansolarazine) combined acetate (Pyramid potassium) and two antibacterials, which form a powerful “fouling” formation. Antibacterial drugs are often used in combinations of Amosicillin and Kracin, which destroy bacterial cell walls, Kracin acts on bacterial nuclei, inhibits protein synthesis, and the two come from different target points and work together to inhibit the growth and reproduction of the fungi; proton pump inhibits gastric acidation, increases the pH value of the stomach, creates a favourable “operational environment” for antibacterial drugs, creates a protective membrane on the stomach muclin surface, binds the fungus, enhances antibacterial effects, promotes mucous rehabiliation, and the therapeutic process is usually 10 – 14 days, which is administered in a manner that significantly increases the eradication rate of the fung.
Intestine fungus infections: antifluents, “Scrambling.” People with low immunity, long-term use of sugar-coated hormones or broad-spectral antibacterial drugs are vulnerable to intestinal fungi, and white membrane is one of the “probators” that causes fungus intestinal inflammation in the form of diarrhoea, abdominal swelling, tofu slag and mucous membranes, such as goose scabies. Fluconium as a front-line antifluorus “enabling hand”, by inhibiting fungal cell pigmentation P450 relying on woolen oxen 14α-demethylide enzymes, disrupting fungal platinum synthesis, damaging cell membrane integrity, effective penetration of intestinal tissues after oral or intravenous dripping, “elimination” of white dysentery and restoring normal intestinal gynaecology; and, in the case of resistance strains or complications, the use of new antifluents, such as voliacinol, Carpofon, can be chosen to widen antibacterial spectrometry, enhance antibacterial activity and ensure precision against fungi pathogens.
Infections of the digestive system are complex, and it is self-decided that medications are subject to error and delay. In cases of persistent gastrointestinal disorders and abnormal defecation, it is important to have timely medical access, to identify the causes of the disease through tests such as faecal routines, gastroscopes and bacterial culture, and to regulate, under the guidance of a doctor, the use of antibacterial drugs and to build a “fortress” for the health of the digestive system.
