Hepatic cerebropathy is a functional syndrome of the central nervous system, based on metabolic disorders, caused by severe liver disorders, mainly in the form of cognitive disorders, behavioural disorders and comas. Given their extremely high mortality rates, it is important to know how to prevent and treat them.
Preventive measures
1. Active treatment of basic hepatitis: Most liver cerebral diseases develop from cirrhosis of the liver, such as hepatitis B and C, which require active resistance to the virus. Hepatitis B is treated in Nteikawe and Novowe, which inhibits the replicability of the virus, mitigates liver damage and reduces the risk of liver cerebral disease. Alcoholic liver patients must stop drinking and be treated with medication to repair damaged liver cells.
Control incentives:
– Avoiding ingestional haemorrhage: Disruption of the edible larvae is a common cause. Patients should refrain from eating rough food in order to prevent the cutting of blood vessels. Periodic gastroscopy is performed, and it is found to be severe, irritating, irritating, i.e. plastering, sclerizer injection, and can also be used to reduce the cosmopolitan pressure, but can increase the risk of liver cerebral disease.
– Prevention of infection: infection increases tissue metabolism and increases the production of toxic substances such as ammonia. In the event of infection, medical attention is promptly provided and antibiotics are reasonably selected.
– Prevention of electrolyte disorders: large amounts of urine, Electrolytic disorders due to discharge of abdominal water. The use of urea is strictly subject to medical instructions and electrolyte is regularly monitored. When abdominal water is required, the velocity and quantity are controlled and the protein is properly supplemented.
3. A reasonable diet:
– Control of protein intake: good-quality proteins such as milk, eggs, fish and meat. During acute onset, protein intake is limited to 20 g/d, increasing as the condition improves. Blood ammonia is low but has cerebral symptoms that increase plant protein intake and contain acoustic amino acids, which are competitively inhibiting aromatic amino acids from entering the brain.
– Increased dietary fibre intake: extra vegetables, fruits, promotion of intestinal creeping, for defecation and reduction of the absorption of toxic substances such as intestinal ammonia.
Periodic review: periodic review of liver function, blood ammonia, abdominal ultrasound, etc. of persons with liver cirrhosis. Potential risks can be detected in a timely manner by monitoring levels of ammonia. Once the blood ammonia rises, the treatment will need to be adjusted even if it is not symptomatic.
Treatment
1. Detergents such as haemorrhage in digestive tracts, rapid stop bleeding, resupply of blood capacity, reduction of internal flow of blood by growth inhibitors, stop bleeding under stomach mirrors, etc. For infections, sensitive antibiotics, such as coliform infection, are selected on the basis of pathogens.
2. Reduction of ammonia generation and absorption:
– Lactose: After oral administration, bacteria decompose into lactated and acetic acid in the colon, lower intestinal pH and decimate the ammonia into ammonium ion. It usually starts at a small dose and is adjusted to the number of defecations, with two to three softs per day.
– Prophylactic bacteria: regulate intestinal strains, inhibit the growth of ammonia-producing bacteria and reduce ammonia production. Duplexella, acidic yogurt, etc. are commonly used.
3. Promotion of ammonia metabolism:
– Dominoaminomate bird amino acid: provide urea and azinamide synthesis to promote ammonia metabolism. Following an intravenous drip, part of the ammonia was transformed into urea from the kidneys.
Other treatments:
– Correcting the amino acid imbalance: an intravenous infusion of salino acids, correcting the mismatch between the chain and aromatic amino acids and improving brain functioning.
– Artificial liver support systems: applied to acute liver failure or to acutely increased liver encephalopathy due to chronic liver disease, the elimination of toxins through in vitro devices, the addition of beneficial substances and the replacement of some liver functions.
– Hepatic transplants: effective treatment is provided to persons with serious liver and cerebral disorders who are ineffective in the treatment of internal medicine. However, supply shortages and post-operative immunization exclusion are challenges.
In general, hepatitis is a disease with a high risk of digestive mortality, most of which occurs during the cirrhosis period. Active treatment slows down to the failure period without the cirrhosis period. If the cirrhosis period has occurred or has occurred, the disease should be actively prevented and mortality rates reduced.
Hepatic cerebral disease
