The cirrhosis of the liver is long-term damage to the liver, extensive fibrosis of the tissue, repeated destruction of normal lobes, regeneration of the fibrous tissues, formation of false foetals, deformation of the liver and hardening of the liver, which is at the end stage of various chronic, but not the cause of the disease, in which the lives of many patients end.
I. Causes
1. Virus infections: Hepatitis B and C viruses are common. The virus continues to replicate and damage liver cells, causing inflammation and long-term cirrhosis. The rate of hepatitis cirrhosis is high in the country.
2. Chronic alcohol hepatitis: chronic alcohol consumption and alcohol metabolic acetylene damage to hepatic cells, causing hepatic cell fatty deformity, bad death and, consequently, fibrosis. The daily average of alcohol consumption and the number of years of drinking are associated with morbidity, and this type of liver disease is on the rise in the country.
3. Courage silt: cholesterol in and around the liver, lack of cholesterol, cholesterol damage to liver cells and cirrhosis of the liver. Such as original cholesterol, primary cholesterol.
4. Drug or toxic injuries: Long-term hepatoplasmic drugs, such as ammonium butterflies, or exposure to toxic substances such as carbon tetrachloride, cumulative damage to liver cells and development into liver cirrhosis.
5. Self-immuno-hepatitis: The attack on the liver by the own immune system causes hepatitis, necrosis and cirrhosis. It’s like autoimmune hepatitis.
II. Symptoms
1. Substitute period: light symptoms and lack of specificity. There may be a lack of strength, reduced appetite, abdominal swelling, increased fatigue or anger, and rest or accommodation can be mitigated. The liver is slightly swollen and part of the spleen is swollen.
2. Period of default
– Declining liver function: inadequate digestive absorption, nausea, vomiting, anorexia. Yellow, skin and membrane yellow. Hemorrhage tends to occur, with teeth and nose hemorrhages, with reduced coagulation factors for the liver. Endocrine disorders, male breast development, female menstrual disorders, and spider moles and liver palms.
– High pressure of the frontal veins: spleen size, spleen bulging, blood cell reduction. The side circulation is established, such as a oesophagus of the oesophagus, which breaks to cause haemorrhage in the digestive tract; a diarrhea of the abdominal wall; abdominal water, abdominal swelling; and mobile silt positive.
Diagnosis and treatment
Diagnosis: combined with medical history, signs and examinations. Laboratory check liver function, coagulation function, virus marker. Image science uses ultrasound, CT, MRI to observe liver form, structure and abdominal water. Liver puncture biopsy is the gold standard and has been initiated to determine the extent of liver fibrosis.
2. Treatment: treatment for causes of illness, such as anti-virus and alcohol. Hepatitis cytorrhea is treated, hepatitis is mitigated with hepatitis, and abdominal urine is used. Treatment of complications, fracture of the edible larvae and bleeding, and use of drugs, endoscopy, intervention or surgery to stop the bleeding. Hepatic cirrhosis is considered for liver transplant.
Preventive measures
1. Vaccination: Hepatitis B prevention. Newborns should be vaccinated against hepatitis B in a timely manner; adults are not infected and have no antibodies, and vaccinations are recommended. Hepatitis A and E vaccines can be administered on an as-needed basis in epidemic areas or high-risk populations.
2. Healthy diets and living habits: balanced diets, fruits and vegetables, whole grains, control of fat sugar intake. To avoid alcohol consumption, men consume less than 25 grams of alcohol per day and women less than 15 grams per day. Regularity, avoidance of lateness, proper exercise and physical improvement.
3. Precautionary use and protection: medically prescribed drugs are not self-reducing or abusive. Protect against occupational exposure when exposed to chemicals and toxins.
4. Periodic medical examinations: early detection of early intervention by high-risk groups, such as persons infected with hepatitis B, C, chronic alcoholics, periodic examination of liver function, liver ultrasound, etc.
The cirrhosis of the liver is preventable and irreversible once it is cirrhosis, focusing on early detection of risk factors and early intervention. The cirrhosis of the liver can also extend its life by controlling the delay process.
Hepatic cirrhosis
