In some cases, the recovery period of patients in the brain can be characterized by retardation, loss of memory, loss of focus, loss of attention, loss of speech flow and difficulties in understanding. The decline in cognitive capacity, in addition to the loss of the post-minority direct hemorrhage or haematoma oppression, also includes brain water, epilepsy, high blood pressure, and high blood sugar leading to many secondary damage. Therefore, improving the post-middle cognitive function is an integrated and multi-faceted approach to comprehensive rehabilitation, requiring the removal of secondary encephalus and the control of complications and basic diseases such as epilepsy, hypertension, high blood sugar and high blood resin, on the basis of which dietary restructuring is carried out, nutritional support is strengthened and continuous training is repeated in reading, hearing, etc.
In the event of a continuous reduction in the cognitive function of the patient ‘ s mid-career, be wary of cerebral sepsis, especially in the case of the elderly, who are vulnerable to the misdiagnosis of Alzheimer ‘ s dementia in the form of a decline in mentality, unstable movement and defecation, and are then returned to the hospital for further clearly diagnosed and treated for brain sepsis surgery, such as head MR examinations, diagnostic vertebraepsy, etc. Basic diseases such as hypertension, high blood sugar and high blood resin continue to cause continuous damage to brain tissue, and it is essential to continue the treatment programme during hospitalization, to control “three heights” and to regularly review and adjust the treatment programme at community health centres or hospital clinics. Studies have shown that the anti-sculpable tablet drug, Cilotaz, reduces the risk of dementia for people with no history of dementia [1]. It is therefore necessary to continue anti-sculpture treatment.
In addition to the removal of damage factors, there is an active effort to improve the brain structure and to promote cognitive function restoration. Nutritional support is essential. There is growing concern about the preventive effects of healthy food on brain function. Observatory studies have shown that dietary proteins can improve cognitive capacity, including the long-term effects of mental motion speed and attention, performance function, memory and overall awareness. Protein-rich foods or supplements may prevent or reduce the decline in cognitive capacity [2]. Protein-rich foods include beef, chicken chests, fish, etc., and supplements include protein powders such as anthon, which are sold in formal supermarkets or drugs. These nutrients do not include all health products. The Mediterranean diet has been a well-respected and healthy diet, and the MIND diet, which has been improved in recent years on the basis of the Mediterranean diet, has been confirmed by studies that have slowed down neurodegenerative pathologies and improved cognitive functioning, and similar studies have been published in a number of professional journals [3] [4]. This diet is characterized by green leaves and other vegetables, preferring berries rather than other fruits, supporting whole grains, beans and nuts, eating fish at least once a week, and limiting fried foods such as cheese, red meat, candy, fast food and oil bars.
When patients are often discharged from hospital, doctors are asked if they can still drink tea and drinks such as coffee. Research has shown that often non-canned beverages such as tea and coffee can improve cognitive functions, especially in cases of cardiac tremors, which cause patients in the brain, which are an independent risk factor in the development of cognitive disorders, and that frequent coffee is beneficial to the cognitive capacity of healthy individuals. Coffee for persons with room tremors may be associated with improved cognitive capacity and reduced signs of inflammation.[5] There are also studies of excessive high-salt diets that may damage neurons in the mid-seahorse region of the brain, where neurons are responsible for our memory, emotional control, and so on, reducing salt intake can mitigate neurotic degenerative disorders, including cognitive functions, among the elderly, so that the diet of the sick in the pawn can be reduced [6].
Some of the pawns become lazy and sleep for long periods of time, but long sleep is associated with a decline in the overall cognitive function. Among people who combine depression, there is a strong correlation between sleep time and cognitive ability, suggesting that sleep time may be a variable risk factor for reduced cognitive ability. Such patients should be encouraged to change their way of life, maintain regular sleep and be actively rehabilitated [7]. Most of the pawns, with varying degrees of loss in reading and hearing after the onset of the disease, are able to distinguish the basic structure of the written language at first sight, which is much faster than it is expressed loudly, so that the first drop in the pawns is a decline in the ability to read, less a decline in the speed of reading, and more of the ability to read with sound. In the early stages of rehabilitation among pawns, reading training can begin at an early stage, starting with the simplest words and gradually increasing the magnitude and difficulty of the patient ‘ s literacy and reading skills. This can facilitate the remodelling of neurons in the cortex of the brain language, while at the same time contributing to the restoration of linguistic thinking and the improvement of cognitive levels. Hearing training may be conducted in substance at the same time as reading training, with the addition of musical methods such as music intervention.[8] In addition to this, intensive body and body sports and skills training is an important means of rehabilitation to improve cognitive functions, and systematic rehabilitation is recommended with the help of rehabilitation trainers.
References:
[1] X. Cheng, Q. Ren, J. Zhi, et al. Effecs of cilostazol on cognition funcing and dementia risk: A systematic review and meta-analysis, J Medicine (Baltimore) 2024; 103:50; e40668.
[2] M. S. Adams, R. P. Mensink, P. J. Joris. Effects of diagnosive proteins on cognition performance and brain vascular funcing in opinions: a systematic review of ranting controlled trials. Jnutr Res Rev 2024;
Impact of the MIND Diet on Communications with Dementia.
[4] M. Hollander, N. Jariwala, L. Sotelo, et al. The impact of MIND diet regulation and physical activity on health promotions.
[5] M. Barbagalo, A. Springer, C. Vanetta, et al. Coffee Consorption Corretions With Better Cognitic Personality in Patients with a High Institute for Stroke. J. Am Heart Assoc 2024; e034365.
[6] K. Ma, C. Zhang, H. Zhang, C. An, G. Li, L. Cheng, et al. High-Salt Acelerates Neuron Los and Anxiety in APP/PS1 Mice Through Serpina3n. J Mol Sci 2024; 25;
V. M. Young, R. Bernal, A. A. Baril, et al. Long Sleep Durance, Cognitive Performance, and the Moderating Roll of Depresion: A Cross-Sectoral Analysis in the Framingham Heart Study.
[8] J. Rodakowski, J. L. Dorris, D. J. McLaughlin, et al. Music Intervention Pilot Studiy: Assing Teaching Methods of Group Marimba Classes Designed to Train Spatial Skills of Old Ages with Changers in Regulation. J Music Med 2021;
