Among the many stomach diseases, HP correlations are more common, and the fungus (HPs) are like hidden “invisible killers” in the stomach, launching silent attacks on stomach health, causing a series of symptoms of discomfort and potential risks.
Sphinx is a grenacella fungus that lives mainly in the human stomach and in the 12-finger bowel. It has a strong ability to survive in bad conditions of stomach acid and is closely bound to the surface of the gastric mucous membranes through its spiral structure and special motion, thereby causing damage to the gastric mucous membranes and causing stomach inflammation.
HP Incidence mechanisms related to stomach disease are complex. Once the fungus is settled in the stomach, a variety of enzymes and toxins, such as urea enzymes, empty-bulb toxins, are released. The urea enzyme is capable of dissecting urea to produce ammonia, which can moderate and gastric acid, making its surrounding environment more suitable for bacterial survival, while it has a direct toxic effect on gastric mucous cells and can damage the barrier function of the gastric mucous membrane. An empty bubble toxin can lead to the formation of an empty bubble sample by upper skin cells of the gastric mucous membrane, which impairs the integrity of the tissue of the gastric mucous membrane and makes it more susceptible to digestive erosion of stomach acids and gastric proteinases, thus triggering inflammatory reactions. In addition, cholesterocococcal infections can cause the activation of the human immune system, resulting in an immune response that further exacerbates the damage to the gastric mucous membranes, and long-term inflammation irritation can lead to changes in the gastric mucous membranes, i.e. atrophy, intestinal in vitro, and increase the risk of stomach cancer.
The clinical manifestations of HP-related stomach inflammation are not specific, and some patients may not have visible symptoms or have only mild abdominal disorders, abdominal pain, abdominal abdomen, gas, appetite, nausea, vomiting, etc., which can easily be ignored or confused with other stomach diseases. In the case of more serious or long-term conditions, haemorrhage may occur in the upper digestive tract, in the form of black defecation or vomiting, but relatively rare.
The diagnosis of HP relevance for stomach inflammation is usually combined in multiple ways. Stomach spectroscopy is one of the important diagnostic tools through which pathologies such as the morphology of the gastric mucous membranes, colours, carving and ulcer can be directly observed, and pathological examinations may be desirable in the stomach mucous tissue to determine the presence of cholesterosomiasis infection and to assess the inflammity of the stomach mucous membranes and other pathologies. In addition, there are a number of non-intrusive detection methods, such as carbon – 13 or carbon – 14 excretion tests, where a patient is able to detect the content of decomposition products in a gas after oral urea containing a specific mark, thus determining whether or not he is infected with the cholesterosomiasis, which is simple, pain-free and widely applied to clinical screening; serocytological detection of the antibodies of the cracobacteria can also be used as an assistive diagnostic method, but it does not distinguish between current or prior infections.
Once diagnosed as HP-related gastroenteritis, the key to treatment is the eradication of fungus. The treatment currently commonly used is a combination of proton pump inhibitors (e.g., Omera, Lansola, Pitola, etc.), beryllium (e.g., potassium acetate) and two antibiotics (e.g., Amosilin, Craccoline, Metrazine, Furan, etc.), which generally lasts 10 – 14 days. This tetratherapeutic treatment can serve the purpose of root treatment by effectively inhibiting gastric acidization, protecting the gastric mucous membranes and eliminating the fungus of the fungus through the synergy of antibiotics. In the course of treatment, the patient should take the medication in strict compliance with the medical instructions, and avoid leakage or self-disposal to ensure its effectiveness. At the same time, after treatment, there is also a need for review, usually after four weeks of detoxification, for a carbon – 13 or carbon – 14 exhalation test to confirm whether the fungus has been successfully eradicated.
The prevention of HP relevance to stomachitis is equally important. It is essential to develop good personal hygiene practices, as the fungus is mainly transmitted through mouth – mouth and faeces – mouth. In daily life, care should be taken to keep the diet healthy, avoiding the consumption of unclean food and raw water, and to minimize the use of cold foods; to promote the use of public chopsticks, spoons and meal-sharing to reduce the risk of cross-infection between family members; and to keep the mouth clean and regularly change toothbrushes to prevent the growth and spread of the fungus of the mouth to the stomach.
While HP is common, if we raise awareness of it, make early diagnosis, early treatment and take effective preventive measures, we can effectively control the infection of the fungus, protect stomach health and reduce the risk of stomach disease, and keep our stomachs away from this “invisible killer” and enjoy a healthy life.
Stomachitis
