Illustrative disease.
This is a complex category of self-immunological diseases.
Definitions and characteristics
Communicable conjunctive tissue diseases are defined as a large group of diseases with multiple system irritation (e.g. skin, joints, muscles, heart, kidney, blood-building system, central nervous system, etc.), long duration, complex and persistent conditions, and multiple self-antibodies in serum.
II. Common types
Rheumatism
– Main assault on the joints. Patients often suffer from arthritis pain, swelling and symmetry, with small joints such as near-end joints of hands, palm joints, etc. being the first to suffer. As the condition progresses, the cartilage and bone of the joint can be damaged, leading to joint deformities that seriously affect the mobility of the patient. Self-antibodies such as rheumatist factors can be detected in most patients.
2. Systemic red hemorrhoids
– It is an autoimmune disease of a multisystem, multi-organistic nature. The skin of the patient can be red-stained, typical of butterflies, cross-nose beams and double cheeks. It can also be tiring and renal, with protein urine, blood urine, etc.; when tiring and the blood system, there may be anaemia, a decrease in white cells or a decrease in slabs. Various self-antibodies, such as anti-nuclei, are positive among the systematic erythalamus.
Dry syndrome
– Main attacks on the excreta, especially saliva and tear. Patients often feel dry and need to eat dry foods with water to carry them, and the rampant cartilage is one of their characteristic manifestations; the dry eye, with a sense of sand grinding, can lead to a cornea ulcer when severe. The anti-SA and anti-SSB antibodies are detected in patients.
III. Causes
1. Genetic factors
– The presence of certain genes can make individuals vulnerable to widespread conjunctive tissue diseases. For example, among patients with rheumatism, specific human white cell antigen (HLA) genes are associated with disease susceptibility.
2. Environmental factors
– Including infections (e.g., certain viruses, bacterial infections that could trigger an immune response), exposure to chemical substances, etc. For example, long-term exposure to some organic solvents may increase the risk of self-immunizing diseases.
3. Immunization anomalies
– Disruption of the patient ‘ s own immune system, wrongly identifying the patient ‘ s own tissue as an external antigen, thereby initiating the immune response, producing an antibody, and attacking its own tissue and organs.
IV. Diagnosis and treatment
Diagnosis
– Diagnosis is carried out by doctors in combination with symptoms, signs, laboratory examinations (e.g. self-antibody tests, inflammation indicator tests, etc.), visual examinations (e.g. joint X-rays, CT, MRI imaging, etc.).
Treatment
– Comprehensive treatment is generally used. These include the use of sugar-coated hormones to mitigate inflammation; immunosuppressants (e.g., aminophosphate, cyclophosphate, etc.) to regulate immune functions; and biological agents (e.g., drugs for tumour cause of death – alpha, white cell media – target points – 6, etc.), which can specifically disrupt key molecules in the immunitis route and improve treatment effectiveness. At the same time, there is also a need for treatment of organs and systems that suffer from stress to improve the quality of life of patients.
Consistency tissue diseases are a complex category of diseases that require long-term management, and patients need to actively cooperate with doctors and review them regularly to control their development.
