The soluble streptococcus, a common type of gland positive fungus, is widely distributed in nature, in water, air and healthy human mouths and nasal cavities, with complex and diverse mechanisms and a rich clinical performance. The following is a detailed discussion of the mechanism for the occurrence and clinical performance of the soluble streptococcus.
I. EMERGENCY MECHANISMS
Synthetic streptocytocytococcus is produced mainly in the areas of immuno-response, genetic susceptibility, skin mucous membrane damage, blood condensation disorders and inappropriate use of antibiotics. 1. Immunization response:
• In case of over- and abnormally immune responses to an antigen, allergies, self-immuno-diseases, etc., may result in tissue damage and inflammation, which in turn can cause scabies infection.
• Certain protein components produced as antigen irritant bodies produce antibodies, form circulatory immunocomposables and are deposited in tissue organs and cause inflammatory reactions after infection. 2. Genetic susceptibility:
• An individual’s ability to carry certain genetic mutations or defects may increase his or her vulnerability to specific types of immune response, thus increasing the risk of infection with soluble streptocyccus. Skin mucous membrane damage:
• When the skin or mucous membrane is damaged, the body defence function is reduced and bacteria are vulnerable to intrusion and infection. Soluble streptocycoccus enters the blood cycle through wounds, causing infection. 4. Blood condensation disorder:
• Blood condensation disorders affect the ability of the organism to defend the pathogens, making the soluble streptococcal fungi susceptible to spreading and infecting the surrounding tissue. 5. Inadequate use of antibiotics:
• The long-term use of broad-spectral antibiotics can lead to normal fungus disorders, which can facilitate the planting and reproduction of more resistant soluble streptocystella.
II. Clinical performance
The clinical performance of soluble streptocyccus infection varies according to the area of infection and the individual. 1. Upper respiratory infections:
Soluble streptocyccus is transmitted through foam into humans’ upper respiratory tracts, rapidly breeds under suitable conditions and produces toxins and enzymes, resulting in local mucous membrane bulge, oedema and seepage. Patients may experience cold fighting, fear of cold, throat pain, headache and fatigue. Skin and soft tissue infections:
Soluble streptococcus can cause sepsis in skin and subcutaneous tissues such as diarrhea, beeweed, etc. Patients show symptoms such as local skin swelling, pain and fever.
• Serious skin infections can lead to serious complications such as carnalitis. 3. Utility system infections:
The soluble streptococcal fungus can reverse through the urethrocyte to the bladder, vasectomy, kidney, etc., causing urology system infections. Patients may have symptoms such as urination frequency, urination acuteness, urinary pain and, in serious cases, whole-body symptoms such as fever and back pain. 4. Rheumatism:
• Thermal factors in the metabolic products of soluble streptocyccus, after infection, act in the organism, causing functional impairment of the body temperature-regulating centre, and thus heat. Long-term or repeated soluble streptocycoccus infections can cause rheumatism, as shown by arthritis, heart disease, etc. 5. Acute kidney pelvis:
• Some of the protein components produced by soluble streptocycoccus infection produce antibodies as antigen irritant bodies, form circulatory immunocomposers, settle in kidney balls, activate patches, and cause renal inflammation. Patients may have symptoms such as blood urine, protein urine, oedema and hypertension. Other performances:
Soluble streptococcal infections can also cause diseases such as red fever and infectious endometriitis. Chlamydia fever is manifested in the symptoms of fever, rashes, etc., while infectious endocrinitis can lead to abnormal manifestations such as cardiac disorders and cardiac noises.
In the light of the above, there are many aspects to the mechanism for the development of soluble streptococcus and the clinical performance is quite rich. Therefore, patients with suspected soluble streptocyccus infection should be treated in a timely manner and in a targeted manner. At the same time, measures such as enhancing personal hygiene, improving the body’s immunity and rational use of antibiotics also help to prevent the occurrence of soluble streptococcal infections.
Soluble streptococcus noma
