Infectious shock: learning about this serious medical condition
Infective shock, also known as sepsis or sepsis shock, is a serious medical condition caused by pathogenic microorganisms and their toxins. It involves the intrusion of pathogen microorganisms into the blood cycle, the activation of the host’s cytological and body-liquid immune system, the creation of various cytological factors and internal inflammatory agents, which in turn trigger the SIRS and affect the organs and systems of the organism, causing tissue, cell damage and metabolic and functional impairment, and even multi-organ failure. This paper will provide detailed information on the causes of infectious shock, clinical performance, diagnostic criteria, treatment methods and preventive measures to help readers better understand and respond to the disease.
(a) Pathogens: common fungus of infective shock are gland-negative bacteria, such as coli, creber, meningitis, etc., and gland-positive bacteria, such as scrotum, streptococcus and pneumocococcus, can also cause shock. In addition, certain viral diseases, such as epidemiological haemorrhagic fever, are prone to shock.
Host factors: Pre-existing chronic underlying diseases, such as cirrhosis of the liver, diabetes, malignant neoplasms, leukaemia, organ transplants and patients receiving permanent immunosuppressants, metabolics, cytotoxics and radiotherapy, or persons who retain catheters for urine and veins, are susceptible to infective shock. Older persons, infants and young children, women who give birth, and those with poor physical recovery after a major surgery are particularly vulnerable. Special type of infectious shock: Medium-toxic shock syndrome (TSS) is a serious syndrome caused by bacterial toxins. TSS was initially reported as a result of the golden scab, and similar symptoms have been found in recent years to be caused by streptococcus. The main clinical manifestations of the TSS are acute heat, headaches, confusion, red fever rash, skin rinsing after 1-2 weeks, severe hypotensive pressure or vertical fainting.
Clinical performance The clinical performance of infectious shock is diverse, depending on the area and severity of infection. Common clinical manifestations include: neurological changes: irritability, anxiety, stress, skin paleness, dry mouths, wet limbs, severe unconsciousness, coma, etc. Symptoms of the digestive system: abdominal pain, diarrhoea, nausea, vomiting, etc. Symptoms of the urology system: reduced urine and no urine at serious times. Symptoms of the circulatory system: Infective shock can lead to low blood pressure, and even after active liquid resuscitation treatment, blood pressure is maintained by vascular active drugs. Other: Symptoms such as cold limbs, cold sweating of skin, purple laceration or purple tattoos can also occur. Diagnostic criteria
The diagnostic criteria for infectious shock include the following: All-body inflammation response: excessive or low body temperature, abnormal white cell count, high respiratory frequency, etc. Inadequate infusion of tissue organs: reduced urine, higher levels of lactating acid, cognitive disorders, etc. Blood pressure drops: the constriction pressure is less than 90 mmHg or lower than 40 mmHg on the original basis. Bleeding positive: If there is a high level of suspicion of haematological infection, blood should be bled immediately in order to provide timely treatment with antibiotics.
Treatment The treatment of infectious shock is dominated by liquid resuscitation, angiogenesis, antibacterial antivirals, the removal of stoves and support for treatment. Early correct treatment can significantly reduce patient mortality rates. Anti-infection treatment: Intermittent use of antibiotics within one hour of the diagnosis of an infectious shock, with as much coverage as possible of bacteria, fungi, etc. Before antibiotics are given, bacterial training is provided and, if necessary, surgically removed. Anti-convulsive treatment: Includes circulatory function support, recommended use of crystal fluid recovery, two litres of intravenous input within three hours. The recovery goal is to maintain central intravenous pressure, average arterial pressure, urine, blood oxygen saturation of the central vein and blood lactation in the normal range. Support for treatment of disorders: Oxygen treatment, respiratory assisted breathing, kidney function support, nutritional support, etc.
The key to preventing infectious shock lies in enhancing hygiene, preventing infection and timely treatment of infection. Strengthening personal hygiene: hand-washing, avoiding exposure to pathogens, etc. can effectively prevent infectious shock. Prevention of infection: Avoiding exposure to pathogens, e.g., to densely populated sites, to patients, etc. Prevention measures should be strengthened for high-risk groups, such as the elderly, the under-immunized, etc. (b) Timely treatment of infections: patients who have been infected should be treated, examined and treated in a timely manner. Improved nutrition: Malnutrition leads to reduced immune functions, increasing the risk of infectious shock. Nutrition should therefore be strengthened to ensure adequate intake of proteins and vitamins. Reasonable use of antibiotics: Overuse of antibiotics leads to increased bacterial resistance, thus increasing the risk of infectious shock. Therefore, antibiotics should be used rationally to avoid abuse.
Infectious shock is a serious medical condition, but by understanding its causes, clinical performance, diagnostic criteria, treatment methods and preventive measures we can better cope with the disease. In the event of symptoms, timely medical treatment, examination and treatment should be provided in order to reduce the incidence of disease and death and improve the quality of life.
