Inflammatory intestinal science.
Inflammatory enteropathy (IBD) is a group of chronic, non-specific intestinal diseases that have not yet been fully described, mainly including ulcer colonitis (UC) and Cron disease (CD).
I. Causes of morbidity
1. Genetic factors
– Inflammatory enteropathy has certain genetic tendencies. The study found that people with family history were at higher risk of inflammatory enteropathy than the general population. If there are IBD cases in the family, the risk of a first-degree relative ‘ s illness increases relatively. For example, one of the same twins is suffering from Crohn disease, while the other is much more likely to suffer from the disease than the other.
2. Environmental factors
– The modern way of life may be linked to the incidence of inflammatory enteropathy. For example, changes in dietary structure, high sugar, high fat and low-fibre Westernized dietary patterns may increase the risk of disease. In addition, smoking has a different impact on Cron and ulcer colonitis, which increases the risk of Cron disease, while ulcer colonitis may have some protection.
3. Immunization factors
Human immunodeficiency system anomalies play a key role in the incidence of inflammatory enteropathy. Under normal circumstances, the intestinal immune system is able to identify and resist the symbiotic bacteria in the intestinal tract, but in IBD patients, the immune system may wrongly attack the intestinal tissue and cause inflammation. The impairment of the intestinal mucous barrier makes it easier for bacteria, antigens, etc., within the intestinal tract to enter the inner membranes, activate immunocellular cells, and release inflammatory factors such as cancer causes – alpha, white-cell media, etc., leading to the persistence of intestinal inflammation.
II. Symptoms
1. Exulsive colonitis
– Diarrhoea, mucous sepsis, abdominal abdominal pain, etc., which is the main manifestation of persistent or repeated outbreaks. Diarrhoeal diseases vary from 3 to 4 times a day to more than 10 times a day. The abdominal pain is mostly in the lower left abdomen or lower abdominal pain, which can also be exhausted and fully abdominal. Patients often have a sense of excrement, i.e. defecation. In addition, all-body symptoms of fever, wasting and anaemia may be associated.
Cron disease
– More diverse symptoms. It is common to have abdominal pain, mostly in the lower right abdomen or umbilical weeks, which can be of accommodative, corrosive or blunt nature. Diarrhoea is also one of the common symptoms, with a large number of paste and generally free of sepsis or slime. In addition, particular manifestations of abdominal blockages, fistula formation and anal disease may occur. Patients can also experience whole-body manifestations such as fever, nutritional disorders, and child patients may experience stunting.
Diagnosis and treatment
Diagnosis
– The diagnosis of inflammatory enteropathy requires a combination of multiple factors. Doctors are asked about patients ‘ medical history, including the frequency, duration, family history, etc. A series of examinations, such as colonoscopy, are also carried out, which is an important means of diagnosing ulcer colonitis and Cronnia. Under the colonoscopy, pathologies of the intestinal mucous membranes, such as ulcer, inflammation, salivation, etc., can be observed directly. In addition, blood tests may be carried out, inflammation indicators such as C-reactive proteins, blood sunk, excreta tests to remove infectious enteritis etc. In the case of Crohn, tests such as intestine imaging, CT or MRI (MRE) help to assess the condition of the intestines.
Treatment
The objective of treatment is to induce and sustain clinical decomposition, improve the quality of life, prevent and treat complications, etc.
– Drug treatment is the main means. For mild ulcer ulcer coliitis, amino-water can be used as an acid, e.g., mesala. For moderately severe patients, the use of sugar cortex hormones, immunosuppressants, such as sulfur, or biological agents, such as inflation, may be required. The same is true for the treatment of Crohn ‘ s disease, with different medications depending on the severity and extent of the disease.
– In addition to drug treatment, nutritional support is important. Because of the potential for poor nutritional intake among people with inflammatory enteria, reasonable nutritional supplementation, such as vitamin supplementation, micronutrients and, where necessary, intestine or intestine nutritional support can improve the nutritional status of patients and contribute to their recovery. Serious complications, such as narrow intestinal tracts, perforation and haemorrhage, may require surgical treatment. However, surgical treatment does not cure inflammatory intestinal diseases and there is still a risk of relapse after surgery.
Inflammatory intestinal disease is a chronic disease that requires long-term management, and patients are required to follow the medical ” Inflammatory intestinal pathology ” .
Inflammatory enteropathy (IBD) is a group of chronic, non-specific intestinal diseases that have not yet been fully described, mainly including ulcer colonitis (UC) and Cron disease (CD).
I. Causes of morbidity
1. Genetic factors
– Inflammatory enteropathy has certain genetic tendencies. The study found that people with family history were at higher risk of inflammatory enteropathy than the general population. If there are IBD cases in the family, the risk of a first-degree relative ‘ s illness increases relatively. For example, one of the same twins is suffering from Crohn disease, while the other is much more likely to suffer from the disease than the other.
2. Environmental factors
– The modern way of life may be linked to the incidence of inflammatory enteropathy. For example, changes in dietary structure, high sugar, high fat and low-fibre Westernized dietary patterns may increase the risk of disease. In addition, smoking has a different impact on Cron and ulcer colonitis, which increases the risk of Cron disease, while ulcer colonitis may have some protection.
3. Immunization factors
Human immunodeficiency system anomalies play a key role in the incidence of inflammatory enteropathy. Under normal circumstances, the intestinal immune system is able to identify and resist the symbiotic bacteria in the intestinal tract, but in IBD patients, the immune system may wrongly attack the intestinal tissue and cause inflammation. The impairment of the intestinal mucous barrier makes it easier for bacteria, antigens, etc., within the intestinal tract to enter the inner membranes, activate immunocellular cells, and release inflammatory factors such as cancer causes – alpha, white-cell media, etc., leading to the persistence of intestinal inflammation.
II. Symptoms
1. Exulsive colonitis
– Diarrhoea, mucous sepsis, abdominal abdominal pain, etc., which is the main manifestation of persistent or repeated outbreaks. Diarrhoeal diseases vary from 3 to 4 times a day to more than 10 times a day. The abdominal pain is mostly in the lower left abdomen or lower abdominal pain, which can also be exhausted and fully abdominal. Patients often have a sense of excrement, i.e. defecation. In addition, all-body symptoms of fever, wasting and anaemia may be associated.
Cron disease
– More diverse symptoms. It is common to have abdominal pain, mostly in the lower right abdomen or umbilical weeks, which can be of accommodative, corrosive or blunt nature. Diarrhoea is also one of the common symptoms, with a large number of paste and generally free of sepsis or slime. In addition, particular manifestations of abdominal blockages, fistula formation and anal disease may occur. Patients can also experience whole-body manifestations such as fever, nutritional disorders, and child patients may experience stunting.
Diagnosis and treatment
Diagnosis
– The diagnosis of inflammatory enteropathy requires a combination of multiple factors. Doctors are asked about patients ‘ medical history, including the frequency, duration, family history, etc. A series of examinations, such as colonoscopy, are also carried out, which is an important means of diagnosing ulcer colonitis and Cronnia. Under the colonoscopy, pathologies of the intestinal mucous membranes, such as ulcer, inflammation, salivation, etc., can be observed directly. In addition, blood tests may be carried out, inflammation indicators such as C-reactive proteins, blood sunk, excreta tests to remove infectious enteritis etc. In the case of Crohn, tests such as intestine imaging, CT or MRI (MRE) help to assess the condition of the intestines.
Treatment
The objective of treatment is to induce and sustain clinical decomposition, improve the quality of life, prevent and treat complications, etc.
– Drug treatment is the main means. For mild ulcer ulcer coliitis, amino-water can be used as an acid, e.g., mesala. For moderately severe patients, the use of sugar cortex hormones, immunosuppressants, such as sulfur, or biological agents, such as inflation, may be required. The same is true for the treatment of Crohn ‘ s disease, with different medications depending on the severity and extent of the disease.
– In addition to drug treatment, nutritional support is important. Because of the potential for poor nutritional intake among people with inflammatory enteria, reasonable nutritional supplementation, such as vitamin supplementation, micronutrients and, where necessary, intestine or intestine nutritional support can improve the nutritional status of patients and contribute to their recovery. Serious complications, such as narrow intestinal tracts, perforation and haemorrhage, may require surgical treatment. However, surgical treatment does not cure inflammatory intestinal diseases and there is still a risk of relapse after surgery.
Inflammatory intestine disease is a chronic disease that requires long-term management, and patients are required to follow the advice of a doctor, to be reviewed regularly and to maintain good living habits in order to control the development of the condition. The recommendations are periodically reviewed and good living habits are maintained to control the development of the condition.
