The incidence of ischaemic intestinal diseases, caused by inadequate supplies of intestinal blood or impeded return, has been increasing in recent years, posing a greater risk to the health of patients, and understanding of the disease is essential for early detection, diagnosis and treatment.
The main causes of ischaemic enteropathy include vascular and non-vascular obstructive factors. In terms of angiogenesis, intestinal aneurysm embolism or leopsis formation is more common, such as the disemboweling of a heart patient (e.g. cardiac tremors, myocardial infarction, etc.), resulting in embolism following the blood flow into the intestinal artery; intestinal dysenteral stimulosis can also cause intestinal retrenchment, mostly related to high blood condensation, abdominal inflammation and abdominal surgery. Non-vascular obstructive factors are mainly due to under-infusion of intestinal blood, such as severe low blood pressure, shock and cardiac failure, which keeps the intestinal inundation low and can cause intestinal deficiency for long periods of time.
The clinical performance of ischaemic intestinal diseases is diverse and can be classified according to the severity and the course of the disease. The acute intestinal ischaemic condition is acute, with severe abdominal pains often occurring, most of which occur in the umbilical week or upper abdominal, which is of an incurable nature, and which is often disproportionate to abdominal signs, i.e., the patient ‘ s abdominal pain is severe but relatively light. Patients may also be associated with gastrointestinal symptoms such as nausea, vomiting and diarrhoea, which, as the disease progresses, can result in abdominal swelling, constipation, perforation, peritonealitis, severe complications such as heat and shock, and endanger life. Chronic intestinal amphylogeneity is more manifested in abdominal pain, nausea, vomiting, fear, etc., which is exacerbated by the increased demand for blood in the intestinal tract after eating, which is not met by a narrow intestinal vascular environment, which often reduces the intake due to fear of pain after eating, and causes chronic undernourishment, such as weight loss. The main symptoms of ischaemic colonitis are sudden pain in the lower abdominal, followed by diarrhoea, defecation, mostly in the form of blood or in the form of dark red blood, which can be accompanied by an acute post-hemorrhagic stress, generally relatively light in the whole body, but may develop into serious effects such as intestine death, perforation, etc. if the ischaemic is severe and untreated in a timely manner.
Diagnosis of ischaemic enteropathy requires a combination of multiple factors. The doctor first asks for details of the patient ‘ s medical history, including past cardiovascular diseases, blood system diseases, abdominal surgery, etc., about the cause and symptoms of the disease. Medical examinations focus on abdominal signs, such as pressure pain, anti-jump pain, changes in the intestines, etc., but, as mentioned earlier, early signs may not be typical. In terms of laboratory tests, blood routines can show an increase in white cell count and, in the case of intestine necrosis, an increase in serum starch enzymes, fat enzymes, lactate dehydrogenase, sio acid anase, etc.; faeces submersible blood tests are often positive. Visual examinations are important in diagnostics, and abdominal X-line tablets reveal intestinal resistance such as intestinal expansion, accumulation, liquid plane, and some patients may have distinctive signs such as intestinal dyslexia, internal intestinal accumulation; colour doppler ultrasound can detect blood flow in the intestinal veins and determine the presence or absence of vascular narrowness, embolism, etc.; CT angiography (CTA) and magnetic resonance (MRA) can clearly show the morphology, walking and pathopathy of intestinal membrane vessels, which is the current method of diagnosing intestinal vascular hysteria; and intestinal hysterectural hysteria is the “char standard” for the diagnosis of intestinal membrane diseases, which not only defines the location, extent and extent of vascular changes, but is also able to intervene in the process.
The key to the treatment of ischaemic enteropathy lies in the early diagnosis and timely restoration of intestinal blood supply. In the case of acute intestinal membrane, a intestinal aneurysm is performed as soon as possible after diagnosis; intestinal aneurysm is formed, which may require an vascular reconstruction operation such as an vascular by-pass transplant; and intestinal membrane anemic sembrane is first treated with anticondensation, e.g. heparin, wafarin, etc., which requires timely surgery to remove the dead intestine if there are signs of intestinal death. Chronic intestinal ischaemic amniotics are mainly carried out through vascular reconstruction operations, such as dysenteral animation and intestinal arthropod implantation, to improve intestinal blood supply and to mitigate symptoms. The treatment of ischaemic colonitis begins with fasting, gastrointestinal decompression, rehydration and nutritional support, while medicines to improve micro-circumcycling in the intestinal tract, such as the frontal ones, are available for gradual recovery after conservative treatment for most patients, but still requires surgical treatment for serious complications such as intestine perforation and peritonealitis.
Prevention of ischaemic intestinal diseases. For groups with high-risk factors, such as cardiovascular diseases, primary diseases should be actively treated, blood pressure, sugar, blood resin should be controlled, and haemorrhages should be prevented; during abdominal surgery, doctors should take care of operating norms to reduce damage to intestinal membrane vessels; and in cases of long-term bed rest, infirmary and infirmary, care should be taken to prevent low blood pressure, shock, etc., and to avoid low infusion in the intestinal tract.
In general, ischaemic enteropathy is a serious intestinal disease, which raises awareness and values it, helps to reduce its morbidity and mortality, and guarantees its intestinal health.
