It’s a common manifestation of congenital aortic arctic deformities.

The aortic diabolical deformity is a common congenital cardiac valor deformation, characterized by the fact that the aortic valor is made up of two, not the normal three. This deformity may result in incomplete aortic valve functions, including narrow and/or incomplete aortic valves, as well as complications such as aortic expansion and aortic piping. Different clinical manifestations depending on the stage of the disease;In the early non-symptomatic stages, many infertile aerobic second-leave malformations may not be evident during childhood or even adolescence, and the heart function is largely normal, with no significant impact on daily life, learning and growth and development, often at regular check-ups where abnormal signs such as heart noises are detected, or incidentally through heart ultrasounds.Symptoms of progress. Respiration difficulties:• Labour respiratory difficulties: As age increases and the effects of malformations on blood flow mechanics of the heart gradually become apparent, when the aortic valves are narrow or shut down, the patient, with increased activity, such as running, rapid climbing, long walking, etc., starts to feel agitated, breathing and effort, requiring a break to alleviate. This is due to the impact on the blood function of the heart pump, which has resulted in increased pulmonary siltation and limited pulmonary gas exchange.• Sit-in breathing: even in a state of rest, when the condition is further aggravated, it can be difficult to breathe, and even a serious patient needs to take a seat at the end (sitting by the bed, leaning under his legs and leaning in front of his body) to feel a little smoother. This is due to the increased levels of lung silt and respiratory difficulties, which are further exacerbated by the increase in cardiac respiration during flat sleep.• Heartbreak:• The combined effect of changes in the function of the heart, such as abnormal heart output; on the other hand, the increase in the incidence of heart disorder (e.g., room tremors) as the disease evolves and the increase in heart capacity or stress loads, which often leads to an abnormal heart rate and a feeling of anxiety. This heart attack is often more pronounced after the activity, which further increases the burden on the heart and makes the abnormal heartbeat more visible.• chest pain:• Some patients may feel pain behind their chests or in front of their heart, which is varied in nature and can be sore, swelling or stinging. The reasons for this are mainly related to myocardial ischaemic blood (e.g., the narrow aortic valves can lead to underfilling of coronary artery), and the ability of the heart to reach the surrounding tissue. Such pain is also usually exacerbated by further increase in the heart load when active or emotional.• Irritation, fatigue:• Due to the loss of the blood function of the heart pump, the blood and oxygen delivered to the body are not sufficient to meet the normal needs of the body, and the patient is gradually suffering from the symptoms of physical inactivity and fatigue and a marked decrease in activity resistance. In the past, for example, routine activities that can be easily accomplished, such as long walking distances, simple housework, etc., can feel exhausted when they become ill.• Stifler:• In situations where the aortic valves are narrower and more severe, especially when the heart load is suddenly increased by the patient’s intense exercise, emotional agitation, etc., the heart output is reduced sharply, which in turn leads to a lack of blood in the brain, which can lead to fainting, in the form of a sudden loss of consciousness and a brief collapse of the patient, although in most cases the awareness is restored more quickly. But stunning is a sign of a more serious condition and requires high priority.Oedema:• At the advanced stages of the disease, there may be signs of right heart failure as the condition progresss, with lower leg edema being more common, first dentible edema in ankles, calves, etc., and then the extent of the edema may gradually spread upward to the thigh, or even to the whole body edema, and possibly to the chest and abdominal fluids. This is due to the circulation of blood after heart failure, which causes the liquid to seep into the interlocking gap.It should be noted that the specific symptoms of each patient may vary according to the specific structural characteristics of the arctic diflex (e.g. size of the valve, degree of cross-border integration, etc.), the severity of the condition, and individual differences, and that, in the event of the above-mentioned symptoms, a detailed medical examination and corresponding treatment should be carried out in a timely manner.