It’s all a mess.
Junior A is 20 years old, he’s a little fat, he likes spicy. It’s his birthday and he invited a few good friends to a hotpot to celebrate. They ordered a lot of meat, and they drank a couple of beers, very “hot skin.”
After cooking the hot pot, Little A feels a little swollen, but doesn’t care. Upon return to the house at night, Little A suddenly felt severe pain in the abdomen, along with discomfort in the back and gradually symptoms of nausea, vomiting and fever. He thought he might have eaten too much or been on fire, so he took some digestives and accelerants.
The next morning, Xiao A ‘ s symptoms did not improve, but became more serious. His family quickly took him to the hospital. After the examination, the doctor told Xiao A that he was suffering from HTTGP, a life-threatening disease requiring immediate hospitalization.
Little A wonders why he had pancreas?
I. What’s acute pancreas?
Pancreas can be distributed with a variety of digestive enzymes, but these digestive enzymes are inert in pancreas. In cases of acute pancreasitis, the digestive enzymes in the pancreas are activated, causing internal digestion, oedema, haemorrhage and even death of the pancreas tissue, with severe organ dysfunctions that endanger life.
ii. Why eating a pancreas leads to acute pancreasitis
The high amount of fat in foods such as hot pots can lead to increased levels of glycerine in blood, especially when the triester >11.3 mmol/L induces an increased risk of acute pancreas. High glycerine triester haematosis has some toxic effect on pancreas and causes microcircumcirculosis, which eventually leads to pancreasitis.
III. What symptoms to consider are acute pancreas
Abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal: most major symptoms. (b) Severe pain in the abdomen, which is sustained, is frequently ejected to the back and often occurs after eating or drinking;
2 Disgusting: vomiting is food, gravy, pain in the abdomen after vomiting.
3 heat: mostly moderately heated, with body temperature between 38 and 39°C;
Other manifestations: Severely ill persons can experience shock, such as hypervelocity, low blood pressure, and less urine.
IV. Reasons for acute pancreasitis
There are many reasons for acute pancreas. In our country, the most common are cholesterol diseases, such as gallstones, inflammation, etc., followed by hyperglycerinemia and excessive alcohol consumption. Other causes include medicine, endoscope reverse insulin cholesterol (ERCP) after surgery, calcium haemorrhage, infections, genetics, autoimmune diseases and trauma.
High glycerine tri-ester haemoemia and alcohol acute pancreas are common among young male patients, with older patients being more timid.
V. Unusual results in acute pancreas
1. Unusual test results
(1) Haemorrhagic insulin is elevated: serosol, fat enzyme, urine insulin, etc. are commonly used as an insulin test.
(2) White cell increase: White cell numbers reflect the extent of inflammatory response. White cell counts are generally above 10 x 109/L and 20 x 109/L for serious cases.
(3) Declination of insulin: resulted in an increase in blood sugar and even in ketone acid poisoning.
(4) Glycerine triester rises: Glycerine triester (TG) pancreasitis tends to exceed 11.3mmol/L.
2. Unusual findings
(1) B Super: B supervised pancreas growth, uneven echoes, blurred edges, etc. However, B is highly susceptible to intestine interference, resulting in unclear observations.
(2) Enhancement of CT: is the detection of acute pancreas. It is visible that the pancreas are swollen or pervading, with an increase in non-homogeneity, blurred or disordered edges of the pancreas, immersed fat around the pancreas, pancreas weeks and frontal plaque of the kidneys. Combining haemorrhagic necrosis, CT can see low density areas within or around pancreas.
(3) MRI: Similar to the CT, pancreas volume increases, low signals on T1WI, high signals on T2WI, clearly uneven signals, clearly blurred edges of pancreas, visible strips or disproportionately high signals on T2WI increas. The dynamically enhanced scans can see an uneven increase in the substance of pancreas.
V. Treatment of pancreasitis
The treatment of acute pancreas is based on the principles of fasting, gastrointestinal decompression, acidism, enzyme and rehydration. The treatment also includes measures such as rational use of antibacterial drugs, surgical treatment of local and general complications, and application of painkillers, depending on the severity of the condition, the causes of the disease and the complications.
As a result of timely treatment, pancreasitis in Little A was brought under control through infusion, pressure relief, pain relief and anti-infection, and levels of serogly glycerine triester were gradually reduced to normal levels, and abdominal enhancement of the CT was completed, no glucose deaths or the formation of pseudocysts were detected and discharged after a week. After the hospital, Little A. would never dare to drink so much.
Acute pancreasitis
